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23 Cards in this Set

  • Front
  • Back

Heartburn

after eating acidic foods (spicy, fatty)


sphincter opens upward and acidic food enters into esophagus (no mucus layer)


symptoms: burning in esophagus, chronic cough, ear/nose/throat ailments

Gastroesophageal Reflux

aggravated by: reclining after eating, stress, increased intra-abdominal pressure (cough,strain,bend,obesity,preg,trauma)


increase acid secretion

GERD Treatment

reduce intra-abdominal pressure and gastric acid production


avoid certain foods


patient identify food triggers

Vomiting

reverse perstalsis (going opposite direction)


get rid of things in stomach (bac, virus, eat too much)


loss of fluids and electrolytes


can cause dehydration

Constipation

defecation with straining to pass hard, dry stools


slow movement of feces through colon


waste in colon too long- keeps absorbing


fiber enhances peristalsis


physical activity increases blood flow

Diarrhea

passing of loose, watery bowel movement


can't absorb electrolytes


bac/viral infection, lac intol, spoiled foods, stress


dehydration risk- infants and older adults

Dysphagia

can't chew/swallow adequately


diet must meet nutrition needs


prevent aspiration (protect airwave)


strokes (CVA), blood supply cut off


Dysphagia Nutrition Therapy

individualized diet based on swallowing ability and food pref


solid foods and liquids eval and modified- watch where the food goes (texture,cohesiveness,density,viscosity,consistency,temp,taste)

Patient Positioning

safest eating is upright


gravity can help passage of food along esophagus (prevent choking/aspiration)


can't sit unassigned- raise head of bed or use pillows/wedges

Peptic Ulcer Disease (PUD)

break/ulceration (impairment of tissue) in protective mucosal lining of lower esophagus, stomach, duodenum


exposed to acid= digest stomach wall


cut in GI wall- small intestine bleeding


meds can cause GI bleeding


chronic- can be slow/small (anemia)


deep ulcers penetrate mucosa and damage blood vessels (hemorrhage)


causes for PUD

infection with H Pylori (80% of ulcers)


weakens protective mucosal layer of stomach and duodenum (gastric acid damage epith tissue)


treat with antibiotics


reduce stomach acidity, relieve symptoms, heal ulcer, prevent recurrence, avoid complications

nutrition therapy for PUD

bland diet (no evidence it improves symptoms or ulcer healing)


individualized diet- avoid foods that are associated with symptoms


patient tells triggers


eat healthy diet

Celiac Disease

small intestine disease


gluten-sensitive enteropathy


body attacks itself


mucosa of small intestine (duodenum/prox jejunum)


damage villi- toxic reaction


people with Type I diabetes

damaged Villi in Celiac Disease

shrinking, inflamed villi


variety of symptoms


use "differential"


no energy, feel sick


do not eat gluten

symptoms of Celiac Disease

diarrhea


abdominal distention


fat malabsorption


weight loss


many have no gastrointestinal symptoms and are asymptomatic

Celiac Disease nutrition therapy

gluten removed


no BROW: barley, rye, oats, wheats

Inflammatory Bowel Disease (IBD)

2 idiopathic (unknown cause disease) inflammatory conditions of intestine:


CUC- inflame disease of colon


Chrons- infect anywhere in GI (most small int)


both cause diarrhea (profuse/bloody)


may have no symptoms for some time


IBD: chronic pain, bloating, bowel habits


from time to time

IBD symptoms

abdominal pain


clinical signs: intestinal bleeding, protein loss, fever


nutritional depletion

IBD causes of nutritional depletion

decreased intake


malabsorption


increased nutrient loss


increased nutrient use


drug-nutrient interactions


certain drugs help prevent

IBD nutrition therapy

replace nutrients lost from inflammatory process


correct deficits


maintain energy, nitrogen, fluid, and electrolyte balance


attention given to intestinal resection

Diverticular Disease

pockets in large intestine due to lack of fiber


difficult passage in colon

Diverticulitis

itis-inflammation


acute stages- nutrition therapy individualized to tolerance


low fiber, soft diet


nothing per oral


diverticule don't go away but inflammation stops

Diverticulosis

existence of pockets


high fiber (nuts, corn, seeds)


added sources of soluble/insoluble fiber, supplements, fiber supplemented food and water


any added fiber is good