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98 Cards in this Set

  • Front
  • Back
tidal volume VT or TV
-volume of air inhaled and exhaled w each breath
tidal volume VT or TV normal values
500ml or 5-10ml/kg
tidal volume VT or TV significance
TV may not vary, even w severe disease
Inspiratory reserve volume IRV
-maximum volume of air that can be inhaled after a normal inhalation
Inspiratory reserve volume IRV normal values
3000ml
Expiratory reserve volume ERV
the maximum volume of air that can be exhaled forcibly after a normal exhalation
Expiratory reserve volume ERV normal values
1100ml
Expiratory reserve volume ERV significance
ERV is decreased with restrictive conditions such as obesity, ascities, pregnancy
Residual volume RV
the volume of air remaining in the lungs after a maximum exhalation
Residual volume RV normal values
1200ml
Residual volume RV significance
RV may be increased with obstructive disease
Vital capacity VC
the maximum volume of air exhaled from the point of maximum inspiration
VC= TV + IRV + ERV
Vital capacity VC normal values
4600ml
Vital capacity VC significance
a decrease in VC may be found in neuromuscular disease, generalized fatigue, atelectasis, pulmonary edema, COPD, and obesity
Inspiratory capacity IC
the maximum volume of air inhaled after normal expiration
IC= TV + IRV
Inspiratory capacity IC normal values
3500ml
Inspiratory capacity IC significance
a decrease in IC may indicate restrictive disease. may also be decreased w obesity
Functional residual capacity FRC
the volume of air remaining in the lungs after a normal expiration
FRC= ERV + RV
Functional residual capacity FRC normal values
2300ml
Functional residual capacity FRC significance
FRC may be increased w COPD and decreased w ARDS and obesity
total lung capacity TLC
the volume of air in the lungs after a maximum inspiration
TLC= TV + IRV + ERV + RV
total lung capacity TLC normal values
5800ml
total lung capacity TLC significance
TLC may be decreased w restrictive disease (atelectasis, pneumonia) and increased in COPD
Eupnea
normal breathing at 12-18 breaths/min
Bradypnea
slower than normal rate (<10 breaths/min), w normal depth & regular rhythm
-associated w increased intracranial pressure, brain injury, and drug OD
Tachypnea
rapid, shallow breathing > 24 breaths/min
-associated w pneumonia, pulm edema, metabolic acidosis, septicemia, severe pain, or rib fracture
Hypoventilation
shallow, irregular breathing
Hyperpnea
increase depth of respirations
hyperventilation
increased rate and depth of breathing that results in decreased PaCO2 level
-inspiration and expiration are nearly = in duration
-called kussmaul's respiration if associated w diabetic ketoacidosis or renal origin
apnea
period of cessation of breathing; time duration varies; life threatening if sustained
Cheyne-stokes
-regular cycle where the rate & depth of breathing increase, then decrease until apnea (usually about 20sec) occurs
-duration of apnea may vary and progressively lengthen; therefore is timed & recorded
-associated w heart failure & damage to the respiratory center (drug induced, tumor, or trauma)
Biot's respiration
-periods of normal breathing (3-4breaths) followed by varying period of apnea (usually 10-60sec)
-aka clustered breathing
-associated w some nervous system disorders
flatness percussion ex.
intensity-soft
pitch-high
duration-short
location-thigh
ex. large pleural effusion
dullness percussion ex.
intensity-medium
pitch-medium
duration-medium
location-liver
ex. lobar pneumonia
resonance percussion ex.
intensity-loud
pitch-low
duration-long
location-normal lung
ex. simple chronic bronchitis
hyperresonance percussion ex.
intensity-very loud
pitch-lower
duration-longer
location-none normally
ex. emphysema, pneumothorax
tympany percussion ex.
intensity-loud
pitch-high
location ex. gastric air bubble or puffed out cheek
vesicular
-inspiratory sounds longer than expiratory
soft, low
-entire lung field except over the upper sternum & between the scapulae
bronchiovesicular
-inspiratory = expiratory (about)
intermediate intensity & pitch
-often in the 1st & 2nd interspaces anteriorly and between the scapulae (over the main bronchus)
bronchial
-expiratory sounds longer than inspiratory
loud, high pitch
-over manubrium, if heard at all
tracheal
-inspiratory & expiratory sounds about =
loud, high pitched
-over trachea in the neck
crackles in general
soft, high pitched, discontinuous popping sounds
-may or may not be cleared by coughing
crackles in general-etiology
secondary to fluid in the airways or alveoli or to delayed opening of collapsed alveoli
-associated with heart failure & pulmonary fibrosis
coarse crackles
discontinuous popping sounds heard in early inspiration; harsh, moist sound originating in the large bronchi
coarse crackles-etiology
associated w obstructive pulmonary disease
fine crackles
discontinuous popping sounds heard in late inspiration
-sounds like hair rubbing together; originates in alveoli
fine crackles-etiology
associated w intersitial pneumonia, restrictive pulm disease (ex. fibrosis)
-in early inspiration are associated w bronchitis or pneumonia
wheezes in general
usually heard on expiration-but can be on inspiration
wheezes in general-etiology
associated w bronchial wall oscillation & changes in airway diameter
-associated w chronic bronchitis or bronchiectasis
sonorous wheezes (rhonchi)
deep, low pitched rumbling sounds heard primarily during expiration
-caused by air moving through narrowed tracheobronchial passages
sonorous wheezes (rhonchi)-etiology
associated w secretions or tumor
sibilant wheezes
continuous, musical, high pitch, whistle like sounds heard during insp & exp
-caused by air passing through narrowed or partially obstructed airways
-may clear w coughing
sibilant wheezes-etiology
associated w bronchospasm, asthma, & build up of secretions
pleural friction rub
harsh crackling sound (imitated by rubbing thumb & finger tog near ear)
-may subside pt holds breath-coughing will not clear
-best heard over lower lateral anterior surface of thorax
-sound enhanced by applying pressure to the chest wall w diaphragm of stethoscope
pleural friction rub-etiology
secondary to inflammation & loss of lubricating pleural fluid
consolidation (ex. pneumonia)
tf increased
percussion dull
bronchial breath sounds, crackles, bronchophony, egophony, whispered pectoriloquy
bronchitis
tf normal
percussion resonant
normal to decreased breath sounds, wheezes
emphysema
tf decreased
percussion hyperresonant
decreased intensity of breath sounds, usually w prolonged expiration
asthma (severe attack)
tf normal to decreased
percussion resonant to hyperresonant
wheezes
pulmonary edema
tf normal
percussion resonant
crackles at lung base, possibly wheezes
pleural effusion
tf absent
percussion dull to flat
decreased to absent breath sounds, bronchial breath sounds & bronchophony, egophony, & whispered pectoriloquy above the effusion over the area of compressed lung
pneumothorax
tf decreased
percussion hyperresonant
absent breath sounds
atelectasis
tf absent
percussion flat
decreased to absent breath sounds
forced vital capacity FVC
VC performed w a maximally forced expiratory effort
forced vital capacity FVC remarks
often reduced in COPD bc of air trapping
forced expiratory volume FEVt (usually FEV1)
volume of air exhaled in the specific time during the performance of forced vital capacity
FEV1 is volume exhaled in 1sec
forced expiratory volume FEVT (usually FEV1) remarks
a valuable clue to the severity of the expiratory airway obstruction
ratio of timed forced expiratory volume to forced vital capacity
FEVt/FVC% (usually FEV1/FVC%)
FEVt expressed as a % of the FVC
ratio of timed forced expiratory volume to forced vital capacity
FEVt/FVC% (usually FEV1/FVC%) remarks
another way of expressing the presence or absence or airway obstruction
forced expiratory flow
FEF200-1200
mean forced expiratory flow between 200 and 1200ml of the FVC
forced expiratory flow
FEF200-1200 remarks
an indicator of large airway obstruction
forced midexpiratory flow FEF25-75%
mean forced expiratory flow during the middle half of the FVC
forced midexpiratory flow FEF25-75% remarks
slowed in small airway obstruction
forced end expiratory FEF75-85%
mean forced expiratory flow during the terminal portion of the FVC
forced end expiratory FEF75-85% remarks
slowed in obstruction of smallest airways
maximal voluntary ventilation MVV
volume of air expired in a specific period (12sec) during repetitive maximal effort
maximal voluntary ventilation MVV remarks
an important factor in exercise tolerance
common types of bronchodilator meds for COPD
.
salbutamol, albuterol (Proventil, Ventolin)
Beta2-Adrenergic Agonist Agents
fenoterol (Alupent, Isuprel)
Beta2-Adrenergic Agonist Agents
terbutaline (Brethine)
Beta2-Adrenergic Agonist Agents
formoterol (Foradil)
Beta2-Adrenergic Agonist Agents
salmeterol (Serevent, Diskus)
Beta2-Adrenergic Agonist Agents
salbutamol, albuterol (Proventil, Ventolin)
routes & duration
inhaler, nebulizer, oral
short duration of action
fenoterol (Alupent, Isuprel)
routes & duration
inhaler, nebulizer, oral
short duration of action
terbutaline (Brethine)
routes & duration
inhaler
short duration of action
formoterol (Foradil)
routes & duration
inhaler
long duration of action
salmeterol (Serevent, Diskus)
routes & duration
inhaler
long duration of action
Ipratropium bromide (Atrovent)
Anticholinergic Agents
Ipratropium bromide (Atrovent)
routes & duration
inhaler, nebulizer
short duration of action
fenoterol/ipratropium (Duovent)
Combination Short-Acting Beta-2 Adrenergic Agonist and Anticholinergic Agents
salbutamol/ipratropium (Combivent)
Combination Short-Acting Beta-2 Adrenergic Agonist and Anticholinergic Agents
fenoterol/ipratropium (Duovent)
routes & duration
inhaler, nebulizer
short
salbutamol/ipratropium (Combivent)
routes & duration
inhaler, nebulizer
short
aminophylline (Phyllocontin, Truphylline)
Methylxanthines
theophylline (Theo-Dur, Slo-Bid)
Methylxanthines
aminophylline (Phyllocontin, Truphylline)
routes & duration
oral
variable duration
theophylline (Theo-Dur, Slo-Bid)
routes & duration
oral
variable duration