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232 Cards in this Set
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Respiratory Disruptions
I. Laryngeal Polyps A. Etiology and Pathophysiology--more common in men, ----------, talkers/yellers, ----------, and ----------- |
Respiratory Disruptions
I. Laryngeal Polyps A. Etiology and Pathophysiology--more common in men, singers, talkers/yellers, smokers, and GERD |
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Respiratory Disruptions
I. Laryngeal Polyps B. Clinical Manifestations 1. _______--not going to go away 2. Continuously ________ (breathy, harsh, and low in quality) |
Respiratory Disruptions
I. Laryngeal Polyps B. Clinical Manifestations 1. Cough--not going to go away 2. Continuously change voice (breathy, harsh, and low in quality) |
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Respiratory Disruptions
I. Laryngeal Polyps C. Medical Management 1. Rest vocal cords and maybe a course of --------- 2. If long enough ------------ (rare) and surgically removed (could become malignant) |
Respiratory Disruptions
I. Laryngeal Polyps C. Medical Management 1. Rest vocal cords and maybe a course of steroids 2. If long enough biopsied (rare) and surgically removed (could become malignant) |
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Respiratory Disruptions
I. Laryngeal Polyps C. Medical Management 1. Rest vocal cords and maybe a course of steroids 2. If long enough biopsied (rare) and surgically removed (could become malignant) i. Potential for voice quality to ---------- after surgery |
Respiratory Disruptions
I. Laryngeal Polyps C. Medical Management 1. Rest vocal cords and maybe a course of steroids 2. If long enough biopsied (rare) and surgically removed (could become malignant) i. Potential for voice quality to not be same after surgery |
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Respiratory Disruptions
II. Laryngeal Cancer A. Etiology and Pathophysiology--squamous cell ----------- |
Respiratory Disruptions
II. Laryngeal Cancer A. Etiology and Pathophysiology--squamous cell carcinomas |
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Respiratory Disruptions
II. Laryngeal Cancer A. Etiology and Pathophysiology--squamous cell carcinomas 1. Found in smokers and ------------ users (can be prevented) 2. Prevalence--_____% of head and neck cancers in pts >_______yrs |
Respiratory Disruptions
II. Laryngeal Cancer A. Etiology and Pathophysiology--squamous cell carcinomas 1. Found in smokers and ETOH users (can be prevented) 2. Prevalence--90% of head and neck cancers in pts >50yrs |
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Respiratory Disruptions
II. Laryngeal Cancer B. Clinical Manifestations--hoarseness, persistent -----------, change in --------- quality |
Respiratory Disruptions
II. Laryngeal Cancer B. Clinical Manifestations--hoarseness, persistent sore throat, change in voice quality |
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Respiratory Disruptions
II. Laryngeal Cancer B. Clinical Manifestations--hoarseness, persistent sore throat, change in voice quality 1. Eventually pain and difficulty ---------------- (late presentation) |
Respiratory Disruptions
II. Laryngeal Cancer B. Clinical Manifestations--hoarseness, persistent sore throat, change in voice quality 1. Eventually pain and difficulty swallowing (late presentation) |
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Respiratory Disruptions
II. Laryngeal Cancer C. Diagnostic Studies--visualize -----------, biopsies, ------------- (determine spread) |
Respiratory Disruptions
II. Laryngeal Cancer C. Diagnostic Studies--visualize cancer, biopsies, CT/MRI (determine spread) |
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Respiratory Disruptions
II. Laryngeal Cancer C. Diagnostic Studies--visualize cancer, biopsies, CT/MRI (determine spread) 1. If diagnosed early--good cure rate (after partial/total -------------, laser treatment/---------) |
Respiratory Disruptions
II. Laryngeal Cancer C. Diagnostic Studies--visualize cancer, biopsies, CT/MRI (determine spread) 1. If diagnosed early--good cure rate (after partial/total laryngectomy, laser treatment/chemo) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. _____________--partial/complete surgical removal of larynx usually from cancer (disfiguring) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) a. Often try other ----------- first b. Partial when ---------- limited to 1 spot (may be able to speak, but quality will be different) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) a. Often try other methods first b. Partial when CA limited to 1 spot (may be able to speak, but quality will be different) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) c. Total--radical surgery creating permanent airway through---------- (no speech) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) c. Total--radical surgery creating permanent airway through trachea (no speech) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-___________ |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy • Assessment of ---------- • Pre-op --------- |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy • Assessment of knowledge • Pre-op teaching |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy • Expected goals for ------- |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy • Expected goals for pts |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy • Post-op expectations (J-Ps/---------/suction/pain/---------/---------) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management i. Pre-Laryngectomy • Post-op expectations (J-Ps/ventilation/suction/pain/catheters/feeding tube) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Maintain patient airway (administer ------------, clean --------- tubes TID) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Maintain patient airway (administer humidified air, clean trach tubes TID) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Prevention of infection (dressing changes q---------h, ------- hygiene) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Prevention of infection (dressing changes q8h, oral hygiene) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • ---------- control • Maintain adequate nutritional support --NG tube for -------- days |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Pain control • Maintain adequate nutritional support --NG tube for 7 days |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Effective comm.--__________ speech to make sounds/artificial voice box |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management ii. Post-Laryngectomy • Effective comm.--esophageal speech to make sounds/artificial voice box |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management iii. Home Care--teaching so that they can manage themselves at home • S---------- • Daily cleaning of ----------- |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management iii. Home Care--teaching so that they can manage themselves at home • Suctioning • Daily cleaning of trach |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management iii. Home Care--teaching so that they can manage themselves at home • ________________--steam filled shower, moistened cover over stoma (prevent tracheal bronchitis) • Use of _________ covers--to protect during shower, etc. |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management iii. Home Care--teaching so that they can manage themselves at home • Humidification--steam filled shower, moistened cover over stoma (prevent tracheal bronchitis) • Use of stoma covers--to protect during shower, etc. |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management iii. Home Care--teaching so that they can manage themselves at home • Changing --------- ties or Velcro-type holders • ------------: should have ID band stating that they are neck breathers |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 1. Laryngectomy--partial/complete surgical removal of larynx usually from cancer (disfiguring) d. Nursing Management iii. Home Care--teaching so that they can manage themselves at home • Changing twill ties or Velcro-type holders • Resuscitation--should have ID band stating that they are neck breathers |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of ---------- spread |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread a. Removal of all --------- nodes and --------- channels |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread a. Removal of all lymph nodes and lymphatic channels |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread b. May involve -------- muscle, internal jugular vein, ------------ gland, part of thyroid/parathyroid, --------- accessory nerve (controls speech/swallowing) removal |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread b. May involve sternocleidomastoid muscle, internal jugular vein, submxillary gland, part of thyroid/parathyroid, spinal accessory nerve (controls speech/swallowing) removal |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread c. Usually involves ------- side of neck, but can have -------- (very disfiguring and long recovery) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread c. Usually involves 1 side of neck, but can have 2 (very disfiguring and long recovery) |
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Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread d. Operation should not be preformed if it has spread further (surgery won’t ----------) |
Respiratory Disruptions
II. Laryngeal Cancer D. Medical Management 2. Radial Neck Dissection--remove as much cancer as possible and ↓risk of lymphatic spread d. Operation should not be preformed if it has spread further (surgery won’t contain) |
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Respiratory Disruptions
III. Lung Cancer A. Epidemiology--leading cause of ------------- related death in men and women (survival rate --------%) |
Respiratory Disruptions
III. Lung Cancer A. Epidemiology--leading cause of CA related death in men and women (survival rate 15%) |
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Respiratory Disruptions
III. Lung Cancer B. Etiology-->________yrs w/ long smoking hx (also 2nd hand) is most sig. risk factor (80-______% of lung CA) |
Respiratory Disruptions
III. Lung Cancer B. Etiology-->50yrs w/ long smoking hx (also 2nd hand) is most sig. risk factor (80-90% of lung CA) |
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Respiratory Disruptions
III. Lung Cancer B. Etiology-->50yrs w/ long smoking hx (also 2nd hand) is most sig. risk factor (80-90% of lung CA) 1. Total exposure to ------------- (asbestos, --------, ---------, radiation) |
Respiratory Disruptions
III. Lung Cancer B. Etiology-->50yrs w/ long smoking hx (also 2nd hand) is most sig. risk factor (80-90% of lung CA) 1. Total exposure to carcinogens (asbestos, Ni, Fe, radiation) 2. If stop smoking for 10yrs decrease risk by 30-50% |
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Respiratory Disruptions
III. Lung Cancer B. Etiology-->50yrs w/ long smoking hx (also 2nd hand) is most sig. risk factor (80-90% of lung CA) 2. If stop smoking for _________yrs decrease risk by 30-_____% |
Respiratory Disruptions
III. Lung Cancer B. Etiology-->50yrs w/ long smoking hx (also 2nd hand) is most sig. risk factor (80-90% of lung CA) 2. If stop smoking for 10yrs decrease risk by 30-50% |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 1. Occurs primarily in ------------- or beyond and have preference for upper lobes |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 1. Occurs primarily in segmental bronchi or beyond and have preference for upper lobes |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 1. Occurs primarily in segmental bronchi or beyond and have preference for upper lobes a. ---------% originate from ---------- and are very slow growing (8-10yrs to show on XR) |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 1. Occurs primarily in segmental bronchi or beyond and have preference for upper lobes a. 90% originate from epithelium and are very slow growing (8-10yrs to show on XR) |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. ____________ (20%)--caused by smoking (most malignant w/ poor prognosis-_______m) |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers b. ____________ (80%)--Staged (TNM--_______, node, metastasis) |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) i. _____________--most common (more common in women) |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) i. Adnocarcinoma--most common (more common in women) |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) i. Adnocarcinoma--most common (more common in women) • Not related to ---------- • Nonclinical manifestations until it ----------- |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) i. Adnocarcinoma--most common (more common in women) • Not related to smoking • Nonclinical manifestations until it metastasizes |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) i. Adnocarcinoma--most common (more common in women) • Not related to smoking • Nonclinical manifestations until it metastasizes • Does not respond well to ----------- |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) i. Adnocarcinoma--most common (more common in women) • Not related to smoking • Nonclinical manifestations until it metastasizes • Does not respond well to chemo/treatment |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) ii. ________ cell--30-_____% of CAs (associated with smoking) |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) ii. Squamous cell--30-35% of CAs (associated with smoking) |
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Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) iii. Large cell--correlated with --------- |
Respiratory Disruptions
III. Lung Cancer C. Pathophysiology 2. Primary Lung Cancers a. Small cell CA (20%)--caused by smoking (most malignant w/ poor prognosis-16m) iii. Large cell--correlated with smoking |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with ----------- 1. -------------- (most common-74% of pts)--can cause chest pain from sore muscles 2. --------------: not always |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 1. Persistent cough (most common-74% of pts)--can cause chest pain from sore muscles 2. Hemoptysis--not always |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 3. D---------- 4. H----------- |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 3. Dyspnea 4. Hoarsness, |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 5. -------------- or stridor and recurrent pneumonia or ------------ |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 5. Wheezing or stridor and recurrent pneumonia or bronchitis 6. Difficulty swallowing anorexia/weight loss/fatigue (late manifestation) |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 6. Difficulty ------------, -------------/weight loss/fatigue (late manifestation) |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 6. Difficulty swallowing anorexia/weight loss/fatigue (late manifestation) |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 7. Pleural ---------, pericardial ------------- (if mediasternum), cardiac ----------- |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 7. Pleural effusion, pericardial effusion (if mediasternum), cardiac tamponade |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 8. Superior ------------- syndrome--swelling in ---------, neck, and face |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 8. Superior vena cava syndrome--swelling in arms, neck, and face |
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Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 9. Swollen lymph nodes--swell with ----------- |
Respiratory Disruptions
III. Lung Cancer D. Clinical Manifestations--late in disease, especially with adnocarcinoma 9. Swollen lymph nodes--swell with metastasis |
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Respiratory Disruptions
III. Lung Cancer E. Diagnosis 1. History and --------- 2. CXR--detect -------, pleural effusions, and -------- (1-2sonometers) |
Respiratory Disruptions
III. Lung Cancer E. Diagnosis 1. History and physical 2. CXR--detect metastasis, pleural effusions, and tumors (1-2sonometers) |
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Respiratory Disruptions
III. Lung Cancer E. Diagnosis 2. CXR--detect metastasis, pleural effusions, and tumors (1-2sonometers) a. Routing CXRs often lead to --------- of lung ---------- |
Respiratory Disruptions
III. Lung Cancer E. Diagnosis 2. CXR--detect metastasis, pleural effusions, and tumors (1-2sonometers) a. Routing CXRs often lead to Dx of lung CA |
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Respiratory Disruptions
III. Lung Cancer E. Diagnosis 3. ________--single most effective noninvasive test to determine CA and metastasis |
Respiratory Disruptions
III. Lung Cancer E. Diagnosis 3. CT--single most effective noninvasive test to determine CA and metastasis |
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Respiratory Disruptions
III. Lung Cancer E. Diagnosis 4. Biopsy--definitive --------- (must have ----------- cells) |
Respiratory Disruptions
III. Lung Cancer E. Diagnosis 4. Biopsy--definitive Dx (must have malignant cells) |
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Respiratory Disruptions
III. Lung Cancer E. Diagnosis 4. Biopsy--definitive Dx (must have malignant cells) a. From morning ---------- sample, bronchoscopy, needle ----------, tap pleural --------- |
Respiratory Disruptions
III. Lung Cancer E. Diagnosis 4. Biopsy--definitive Dx (must have malignant cells) a. From morning sputum sample, bronchoscopy, needle biopsy, tap pleural effusion |
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Respiratory Disruptions
III. Lung Cancer E. Diagnosis 4. Biopsy--definitive Dx (must have malignant cells) b. Brain and ---------- most common metastasis sites |
Respiratory Disruptions
III. Lung Cancer E. Diagnosis 4. Biopsy--definitive Dx (must have malignant cells) b. Brain and bone most common metastasis sites |
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Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early ----------- is not helpful) 1. Surgery--only hope (------------ more likely to be resected) |
Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 1. Surgery--only hope (squamous cells more likely to be resected) |
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Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 1. Surgery--only hope (squamous cells more likely to be resected) a. ----------- (part of lung) vs. ------------ (whole lung) |
Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 1. Surgery--only hope (squamous cells more likely to be resected) a. Lobectomy (part of lung) vs. pneumonectomy (whole lung) |
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Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 1. Surgery--only hope (squamous cells more likely to be resected) b. Many times not possible if already ----------- |
Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 1. Surgery--only hope (squamous cells more likely to be resected) b. Many times not possible if already metastasized |
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Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 2. Radiation--when used with surgery and -------- (not beneficial for --------- cell) a. Sometimes just for --------- measures |
Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 2. Radiation--when used with surgery and chemo (not beneficial for small cell) a. Sometimes just for palliative measures |
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Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 3. Chemotherapy--standard treatment for advanced ---------- CA |
Respiratory Disruptions
III. Lung Cancer F. Treatment--based on staging (early detection is not helpful) 3. Chemotherapy--standard treatment for advanced non-small cell CA |
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Respiratory Disruptions
III. Lung Cancer G. Nursing Management 1. Assess level of ----------- related to disease 2. Provide pre-op/post-op procedure and ------------- teaching 3. Assess --------- system |
Respiratory Disruptions
III. Lung Cancer G. Nursing Management 1. Assess level of knowledge related to disease 2. Provide pre-op/post-op procedure and intervention teaching 3. Assess support system |
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Respiratory Disruptions
III. Lung Cancer G. Nursing Management 4. Assess pain and provide --------- measures 5. Assess ------------ status (pts easily lose wt) 6. Be able to provide------------ in the community |
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Respiratory Disruptions III. Lung Cancer G. Nursing Management 1. Assess level of knowledge related to disease 2. Provide pre-op/post-op procedure and intervention teaching 3. Assess support system |
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Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax A. Blunt--body is struck by a blunt object (ex. MVAs chest wall in contact with steering wheel) 1. --------------- trauma--impact of parts of the body against other objects |
Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax A. Blunt--body is struck by a blunt object (ex. MVAs chest wall in contact with steering wheel) 1. Countrecoup trauma--impact of parts of the body against other objects |
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Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax A. Blunt--body is struck by a blunt object (ex. MVAs chest wall in contact with steering wheel) 2. Pulmonary ---------, vessel ------------- (great vessel tears), cardiac tamponade, crush ---------- |
Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax A. Blunt--body is struck by a blunt object (ex. MVAs chest wall in contact with steering wheel) 2. Pulmonary contusions, vessel ruptures (great vessel tears), cardiac tamponade, crush injuries |
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Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax B. ___________--foreign body impales or passes through body tissues C. _____________--air/fluid in pleural space (have a tendency to reoccur) |
Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax B. Penetrating--foreign body impales or passes through body tissues C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) |
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Respiratory Disruptions
IV. Traumatic Injuries of the Chest and Thorax C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) • If >_____% of lung resp. distress, if <________% they can still function (may need chest tube) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) • If >40% of lung resp. distress, if <25% they can still function (may need chest tube) |
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IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by -------- rupture on lung a. Characteristics--no associated open ---------, no underlying disease |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease |
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IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease i. -------------- pneumothorax--no clear cause (tall, thin, 20-_____yr men who smoke) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease i. Sponaneous pneumothorax--no clear cause (tall, thin, 20-40yr men who smoke) |
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IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease ii. Injury from ----------- ventilation (PEEP) iii. Injury from broken -------- |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease ii. Injury from mechanical ventilation (PEEP) iii. Injury from broken ribs |
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IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease iv. S/P ------------- insertion--always follow w/ ----------- to check for pneumo |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 1. Closed--caused by bleb rupture on lung a. Characteristics--no associated open wound, no underlying disease iv. S/P subclavian catheter insertion--always follow w/ CXR to check for pneumo |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) a. Causes--stab/--------- wound; s/p ---------- |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) a. Causes--stab/gunshot wound; s/p thoracotomy |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) b. Manifestations--SOB, ----------------/hyperresonance on affected side, see ------------ |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) b. Manifestations--SOB, shallow breaths/hyperresonance on affected side, see bubbling |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) c. Management--cover w/ ---------- dressing and taped on ------------ sides (creates 1 way valve) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) c. Management--cover w/ vented dressing and taped on 3 sides (creates 1 way valve) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) c. Management--cover w/ vented dressing and taped on 3 sides (creates 1 way valve) i. If taped on all ----------- sides pneumo gets bigger tension ----------- |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) c. Management--cover w/ vented dressing and taped on 3 sides (creates 1 way valve) i. If taped on all 4 sides pneumo gets bigger tension pneumo |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) c. Management--cover w/ vented dressing and taped on 3 sides (creates 1 way valve) ii. Give ----------- until ---------- placement |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 2. Open--air enters pleural space from opening in chest wall and gets trapped (“sucking wound”) c. Management--cover w/ vented dressing and taped on 3 sides (creates 1 way valve) ii. Give oxygen until chest tube placement |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid ------------- of air in pleural space causing severely high ---------------- pressures w/ resultant tension on heart and great vessels (life threatening) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or ----------- object) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) i. Triad of symptoms--resp ----------, look ------------ (↓BP), no breath sounds |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) i. Triad of symptoms--resp distress, look shocky (↓BP), no breath sounds |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) ii. Interferes with ----------- shock ↓BP hypoxic ↓---------, etc. |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) ii. Interferes with venous return shock ↓BP hypoxic ↓CO, etc. |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) iii. Complete collapse -------------- shift (tracheal -----------) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) a. Clinical Manifestations (can occur due to blunt or penetrating object) iii. Complete collapse mediastinal shift (tracheal deviation) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) b. Diagnosis--on clinical grounds (do NOT need ----------) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) b. Diagnosis--on clinical grounds (do NOT need CXR) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) b. Diagnosis--on clinical grounds (do NOT need CXR) c. Treatment--large gauge needle --------------- (if air comes out pt will need chest tube) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 3. Tension--pneumothorax w/ rapid accumulation of air in pleural space causing severely high intrapleural pressures w/ resultant tension on heart and great vessels (life threatening) b. Diagnosis--on clinical grounds (do NOT need CXR) c. Treatment--large gauge needle decompression (if air comes out pt will need chest tube) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. __________--blood accumulates in intrapleural space (blood and air-hemopneumothroax) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) a. Causes--massive bleeding from major chest ---------- or ----------- vessel rupture |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) a. Causes--massive bleeding from major chest vessel or intercostal vessel rupture |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) a. Causes--massive bleeding from major chest vessel or intercostal vessel rupture i. Can accumulate up to -----------L of blood from blunt or ------------ trauma |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) a. Causes--massive bleeding from major chest vessel or intercostal vessel rupture i. Can accumulate up to 1L of blood from blunt or penetrating trauma |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) a. Causes--massive bleeding from major chest vessel or intercostal vessel rupture ii. Pulmonary ------------, -------------- therapy, TB, etc. |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax) a. Causes--massive bleeding from major chest vessel or intercostal vessel rupture ii. Pulmonary embolus, coagulation therapy, TB, etc. |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax). b. Clinical Manifestations--dull ------------, shock (from ↓---------) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax). b. Clinical Manifestations--dull percussion, shock (from ↓blood) |
|
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax). c. Treatment--CXR, ↑------------, chest tube, ----------------- devices (diverts blood back into pt) |
IV. Traumatic Injuries of the Chest and Thorax
C. Pneumothorax--air/fluid in pleural space (have a tendency to reoccur) 4. Hemothorax--blood accumulates in intrapleural space (blood and air-hemopneumothroax). c. Treatment--CXR, ↑HR, chest tube, autotransfusion devices (diverts blood back into pt) |
|
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 1 Clinical Manifestations--pain, --------------, bruising, tenderness, some ------------ bleeding, hurts to take --------------- (increased risk for atalectasis), splinting ----------- side |
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 1 Clinical Manifestations--pain, swelling, bruising, tenderness, some internal bleeding, hurts to take deep breath (increased risk for atalectasis), splinting affected side |
|
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 2. Diagnosis--________ 3. Treatment--↓pain (more apt to take deep breaths) w/ ASA, _________, _______ |
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 2. Diagnosis--CXR 3. Treatment--↓pain (more apt to take deep breaths) w/ ASA, NSAIDS, narcotics |
|
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 2. Diagnosis--CXR 3. Treatment--↓pain (more apt to take deep breaths) w/ ASA, NSAIDS, narcotics a. -------------- if in resp distress (possible pneumothorax) |
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 2. Diagnosis--CXR 3. Treatment--↓pain (more apt to take deep breaths) w/ ASA, NSAIDS, narcotics a. Intubation if in resp distress (possible pneumothorax) |
|
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 2. Diagnosis--CXR 3. Treatment--↓pain (more apt to take deep breaths) w/ ASA, NSAIDS, narcotics b. -------------- chest tubes if fractures and intubation |
IV. Traumatic Injuries of the Chest and Thorax
D. Fractured Ribs--blunt/penetrating injuries (most common from trauma) 2. Diagnosis--CXR 3. Treatment--↓pain (more apt to take deep breaths) w/ ASA, NSAIDS, narcotics b. Prophylactic chest tubes if fractures and intubation |
|
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple -------------- fractures causing instability of chest wall |
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, paradoxical chest movement, ↑RR, shallow breaths, ↑HR |
|
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, ---------- chest movement, ↑---------, shallow breaths, ↑--------- |
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, paradoxical chest movement, ↑RR, shallow breaths, ↑HR |
|
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, paradoxical chest movement, ↑RR, shallow breaths, ↑HR a. Breathe in ------------- pressure sucks chest wall in (opposite when breathing out) |
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, paradoxical chest movement, ↑RR, shallow breaths, ↑HR a. Breathe in negative pressure sucks chest wall in (opposite when breathing out) |
|
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, paradoxical chest movement, ↑RR, shallow breaths, ↑HR b. Often have ------------- contusion and/or ------------- |
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 1. Clinical Manifestations--crepitis, paradoxical chest movement, ↑RR, shallow breaths, ↑HR b. Often have pulmonary contusion and/or pneumothorax |
|
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 2. Diagnosis--fractures on -------- (visual diagnosis) |
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 2. Diagnosis--fractures on CXR (visual diagnosis) |
|
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 3. Treatment--stabilize-----------, ----------- (put on ventilator for flail chest) |
IV. Traumatic Injuries of the Chest and Thorax
E. Flail Chest--from multiple rib fractures causing instability of chest wall 3. Treatment--stabilize chest wall, oxygenate (put on ventilator for flail chest) |
|
V. Pleural Drainage System
A. Purpose--evacuate ----------/air/pus/fluid from ------------- and reestablish ----------- pressure in intrapleural space so lungs can reexpand |
V. Pleural Drainage System
A. Purpose--evacuate blood/air/pus/fluid from thoracic cavity and reestablish negative pressure in intrapleural space so lungs can reexpand |
|
V. Pleural Drainage System
B. Chambers 1. Collection chamber--collects ---------- that drains into chest tub through -------ft connecting tube |
V. Pleural Drainage System
B. Chambers 1. Collection chamber--collects fluid that drains into chest tub through 6ft connecting tube |
|
V. Pleural Drainage System
B. Chambers 1. Collection chamber--collects fluid that drains into chest tub through 6ft connecting tube a. Holds up to ------------mL |
V. Pleural Drainage System
B. Chambers 1. Collection chamber--collects fluid that drains into chest tub through 6ft connecting tube a. Holds up to 2000mL |
|
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--______cm water act as 1-way valve (air drains from______, but not back to pt) |
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) |
|
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) a. B---------- b. T---------: fluctuations on inspiration and expiration (when pt is off suction) |
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) a. Bubbling b. Tidaling--fluctuations on inspiration and expiration (when pt is off suction) |
|
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) b. Tidaling--fluctuations on inspiration and expiration (when pt is off suction) i. Deep breath in with -------------- breathing water moves up |
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) b. Tidaling--fluctuations on inspiration and expiration (when pt is off suction) i. Deep breath in with normal breathing water moves up |
|
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) b. Tidaling--fluctuations on inspiration and expiration (when pt is off suction) ii. Deep breath on -------------- water moves down iii. No ----------: full lung expansion |
V. Pleural Drainage System
B. Chambers 2. Water-Seal Chamber--2cm water act as 1-way valve (air drains from chest, but not back to pt) b. Tidaling--fluctuations on inspiration and expiration (when pt is off suction) ii. Deep breath on vent water moves down iii. No tidaling--full lung expansion |
|
V. Pleural Drainage System
B. Chambers 3. ------------- Chamber--applies controlled suction to chest drainage system |
V. Pleural Drainage System
B. Chambers 3. Suction Control Chamber--applies controlled suction to chest drainage system |
|
V. Pleural Drainage System
B. Chambers 3. Suction Control Chamber--applies controlled suction to chest drainage system a. To regulate suction, connect ------------- line tubing to wall suction and set at ordered level |
V. Pleural Drainage System
B. Chambers 3. Suction Control Chamber--applies controlled suction to chest drainage system a. To regulate suction, connect vacuum line tubing to wall suction and set at ordered level |
|
V. Pleural Drainage System
B. Chambers 3. Suction Control Chamber--applies controlled suction to chest drainage system b. Suction order must be written by ---------- (usually ----------sonometers in suction chamber) |
V. Pleural Drainage System
B. Chambers 3. Suction Control Chamber--applies controlled suction to chest drainage system b. Suction order must be written by physician (usually 20sonometers in suction chamber) |
|
V. Pleural Drainage System
C. Nursing Management 1. Keep tubing coiled loosely below ------------ level and do not let pt lie on it 2. Check ------------ and tape them |
V. Pleural Drainage System
C. Nursing Management 1. Keep tubing coiled loosely below chest level and do not let pt lie on it 2. Check connections and tape them |
|
V. Pleural Drainage System
C. Nursing Management 3. Mark -------------- levels--time depends on drainage (check q5-_______min post-surgery) |
V. Pleural Drainage System
C. Nursing Management 3. Mark drainage levels--time depends on drainage (check q5-10min post-surgery) |
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V. Pleural Drainage System
C. Nursing Management 4. Assess ---------- level in water seal chamber q-------h (suction regulated by water amount in chamber) |
V. Pleural Drainage System
C. Nursing Management 4. Assess water level in water seal chamber q8h (suction regulated by water amount in chamber) |
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V. Pleural Drainage System
C. Nursing Management 5. Observe for ----------- bubbling in water seal chamber and ------------- (tidaling) |
V. Pleural Drainage System
C. Nursing Management 5. Observe for air bubbling in water seal chamber and fluctuations (tidaling) 6. Never elevate drainage system to level of pts chest |
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V. Pleural Drainage System
C. Nursing Management 6. Never ------------ drainage system to level of pts chest 7. Never ---------- tubes--except for changing drainage system |
V. Pleural Drainage System
C. Nursing Management 6. Never elevate drainage system to level of pts chest 7. Never clamp tubes--except for changing drainage system |
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V. Pleural Drainage System
C. Nursing Management 8. If continuous ---------- in water seal chamber, assess for air leak (assess appropriateness) a. Use ------------ to find leak’s location |
V. Pleural Drainage System
C. Nursing Management 8. If continuous bubbling in water seal chamber, assess for air leak (assess appropriateness) a. Use clamp to find leak’s location |
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V. Pleural Drainage System
C. Nursing Management 9. Daily ---------- to determine if totally reinflated (before taking out try ----------- suction and assess) |
V. Pleural Drainage System
C. Nursing Management 9. Daily CXR to determine if totally reinflated (before taking out try gravity suction and assess) |
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V. Pleural Drainage System
C. Nursing Management 10. Premedicate w/ chest tube ----------- (morphine)--take deep breath in and ------------, pull |
V. Pleural Drainage System
C. Nursing Management 10. Premedicate w/ chest tube removal (morphine)--take deep breath in and blow out pull |
|
VI. Acute Respiratory Failure
A. Clinical Manifestations 1. Change in ------------ status (early sign) 2. Dyspnea,-----------, mild --------- |
VI. Acute Respiratory Failure
A. Clinical Manifestations 1. Change in mental status (early sign) 2. Dyspnea, tachypnea, mild HTN |
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VI. Acute Respiratory Failure
A. Clinical Manifestations 2. Dyspnea, tachypnea, mild HTN a. ----------- breathing slowed w/ no -------------, unable to ----------- (bad) intubate |
VI. Acute Respiratory Failure
A. Clinical Manifestations 2. Dyspnea, tachypnea, mild HTN a. Fast breathing slowed w/ no intervention unable to compensate (bad) intubate |
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VI. Acute Respiratory Failure
A. Clinical Manifestations 3. Skin cool, clammy, and ---------- 4. ---------: if PaO2 <45 (late sign) |
VI. Acute Respiratory Failure
A. Clinical Manifestations 3. Skin cool, clammy, and diaphoretic 4. Cyanosis--if PaO2 <45 (late sign) |
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VI. Acute Respiratory Failure
A. Clinical Manifestations 4. Cyanosis--if PaO2 <---------- (late sign) |
VI. Acute Respiratory Failure
A. Clinical Manifestations 4. Cyanosis--if PaO2 <45 (late sign) |
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VI. Acute Respiratory Failure
A. Clinical Manifestations 5. Assess pt for comfort position breathing (ab breathing, ---------- lip, --------- muscles, high --------) |
VI. Acute Respiratory Failure
A. Clinical Manifestations 5. Assess pt for comfort position breathing (ab breathing, pursed lip, IC muscles, high fowlers) |
|
VI. Acute Respiratory Failure
A. Clinical Manifestations 6. --------- breath sounds and ------------- upon percussion |
VI. Acute Respiratory Failure
A. Clinical Manifestations 6. Adventitious breath sounds and hyperresonance upon percussion |
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VI. Acute Respiratory Failure
A. Clinical Manifestations 7. Prolonged expiration (I:E ratio)--1:-------, 1:--------- |
VI. Acute Respiratory Failure
A. Clinical Manifestations 7. Prolonged expiration (I:E ratio)--1:3, 1:4 |
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VI. Acute Respiratory Failure
B. Diagnosis 1. ------------- assessment 2. Lab values--ABGs (checks both --------- and -----------) |
VI. Acute Respiratory Failure
B. Diagnosis 1. Physical assessment 2. Lab values--ABGs (checks both oxygenation and ventilation) |
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VI. Acute Respiratory Failure
B. Diagnosis 3. -------: diagnosis and see changes 4. Mixed venous blood gases--amount of -------- delivered to tissues |
VI. Acute Respiratory Failure
B. Diagnosis 3. CXR--diagnosis and see changes 4. Mixed venous blood gases--amount of O2 delivered to tissues |
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VI. Acute Respiratory Failure
B. Diagnosis 4. Mixed venous blood gases--amount of O2 delivered to tissues a. ----------- (venous 38-42) and ---------- (venous 60-80%) from pulmonary artery catheter |
VI. Acute Respiratory Failure
B. Diagnosis 4. Mixed venous blood gases--amount of O2 delivered to tissues a. PVO2 (venous 38-42) and SVO2 (venous 60-80%) from pulmonary artery catheter |
|
VI. Acute Respiratory Failure
B. Diagnosis 4. Mixed venous blood gases--amount of O2 delivered to tissues a. PVO2 (venous _____-42) and SVO2 (venous ____-80%) from pulmonary artery catheter |
VI. Acute Respiratory Failure
B. Diagnosis 4. Mixed venous blood gases--amount of O2 delivered to tissues a. PVO2 (venous 38-42) and SVO2 (venous 60-80%) from pulmonary artery catheter |
|
VI. Acute Respiratory Failure
B. Diagnosis 4. Mixed venous blood gases--amount of O2 delivered to tissues b. ------------ measure tissue consumption |
VI. Acute Respiratory Failure
B. Diagnosis 4. Mixed venous blood gases--amount of O2 delivered to tissues b. SWANs measure tissue consumption |
|
VI. Acute Respiratory Failure
B. Diagnosis 5. Pulse oximetry (---------2)--if poor (<---------%) get ABG **want >---------% |
VI. Acute Respiratory Failure
B. Diagnosis 5. Pulse oximetry (SpO2)--if poor (<95%) get ABG **want >90% |
|
VI. Acute Respiratory Failure
B. Diagnosis 6. V/Q lung scan--acute ------------- embolus |
VI. Acute Respiratory Failure
B. Diagnosis 6. V/Q lung scan--acute pulmonary embolus |
|
VI. Acute Respiratory Failure
B. Diagnosis 7. Pulmonary ---------: rule out pulmonary embolism |
VI. Acute Respiratory Failure
B. Diagnosis 7. Pulmonary angiography--rule out pulmonary embolism |
|
VI. Acute Respiratory Failure
B. Diagnosis 8. Possible insertion of ---------- to monitor 9. ---------- status ↑ or ↓ |
VI. Acute Respiratory Failure
B. Diagnosis 8. Possible insertion of PA catheter to monitor 9. Fluid status ↑ or ↓ |
|
VI. Acute Respiratory Failure
C. Categories **------------- and -----------: physiologic mechanism for hypoxemia (1-4) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure--alteration in ---------- transport between alveoli and-------------- capillary bed (gas exchange) resulting in ----------2 <60mmHg w/ FiO2 >60% due to problems with lungs (ex. pneumonia, pulmonary edema, pulmonary emboli, CHF, shock) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure--alteration in O2 transport between alveoli and pulmonary capillary bed (gas exchange) resulting in PaO2 <60mmHg w/ FiO2 >60% due to problems with lungs (ex. pneumonia, pulmonary edema, pulmonary emboli, CHF, shock) |
|
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure--alteration in O2 transport between alveoli and pulmonary capillary bed (gas exchange) resulting in PaO2 <-----------mmHg w/ --------------2 >60% due to problems with lungs (ex. pneumonia, pulmonary edema, pulmonary emboli, CHF, shock) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure--alteration in O2 transport between alveoli and pulmonary capillary bed (gas exchange) resulting in PaO2 <60mmHg w/ FiO2 >60% due to problems with lungs (ex. pneumonia, pulmonary edema, pulmonary emboli, CHF, shock) |
|
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure--alteration in O2 transport between alveoli and pulmonary capillary bed (gas exchange) resulting in PaO2 <60mmHg w/ FiO2 >60% due to problems with lungs (ex. ------------, pulmonary -----------, pulmonary ------------, CHF, ---------) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure--alteration in O2 transport between alveoli and pulmonary capillary bed (gas exchange) resulting in PaO2 <60mmHg w/ FiO2 >60% due to problems with lungs (ex. pneumonia, pulmonary edema, pulmonary emboli, CHF, shock) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. -------------- Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-________L/min blood to alveoli and 4/_______L of gas into lungs 1:____) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) i. ↑secretions in --------------- (COPD, --------------, asthma)-↓ventilation w/ same blood |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) i. ↑secretions in airway (COPD, pneumonia, asthma)-↓ventilation w/ same blood |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) ii. Conditions resulting in ---------------- collapse (---------: not taking in enough air) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) ii. Conditions resulting in alveolar collapse (atelectasis--not taking in enough air) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) iii. ↓-------------- flow (pulmonary ----------: blood can’t get to area ↓perfusion) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure a. Ventilation-Perfusion Mismatch--alteration in ratio of ventilation to perfusion (should have 4-5L/min blood to alveoli and 4/5L of gas into lungs 1:1) iii. ↓blood flow (pulmonary embolism--blood can’t get to area ↓perfusion) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. _________-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme --------- mismatch) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) i. ----------: bypasses lungs through ventricular septal defect (↓oxygenation) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) i. Anatomic--bypasses lungs through ventricular septal defect (↓oxygenation) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) ii. --------------: flow through pulmonary capillaries w/out gas exchange |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) i. Anatomic--bypasses lungs through ventricular septal defect (↓oxygenation) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) i. Anatomic--bypasses lungs through ventricular septal defect (↓oxygenation) • ARDS, ------------, pulmonary ----------- |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure b. Shunt-blood exits heart w/o gas exchange (severe hypoxia extreme VQ mismatch) i. Anatomic--bypasses lungs through ventricular septal defect (↓oxygenation) • ARDS, pneumonia, pulmonary edema |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure c. ---------------: gas exchange across alveolar-capillary membrane is compromised by process that thickens/destroys membranes (not permeable) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure c. Diffusion Impairment--gas exchange across alveolar-capillary membrane is compromised by process that thickens/destroys membranes (not permeable) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure c. Diffusion Impairment-- i. Takes longer for ---------- to exchange ii. Pts are ok when at rest, but have no --------- tolerance |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure c. Diffusion Impairment-- i. Takes longer for gas to exchange ii. Pts are ok when at rest, but have no exercise tolerance |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure c. Diffusion Impairment-- iii. _________--thickening of membrane with fibrosis and scarring (main problem) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure c. Diffusion Impairment-- iii. ARDS--thickening of membrane with fibrosis and scarring (main problem) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. ----------------: generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑-----------2 and ↓-----------2 (not enough air into lungs) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) i. Can cause --------- |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) i. Can cause hypoxia |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) ii. Restrictive ------------ disease (asthma), chest wall ------------, neuromuscular disease (------------ brae) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 1. Hypoxemic Respiratory Failure d. Alveolar Hypoventilation--generalized ↓ ventilation resulting in ↑PaCO2 and ↓PaO2 (not enough air into lungs) ii. Restrictive lung disease (asthma), chest wall dysfunction, neuromuscular disease (Gyron brae) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. ---------------: CO2 transport alteration from ventilatory factor (good lungs, but air can’t get in/out hypercapnia main problem, but will have hypoxia too) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure--CO2 transport alteration from ventilatory factor (good lungs, but air can’t get in/out hypercapnia main problem, but will have hypoxia too) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure--CO2 transport alteration from ventilatory factor (good lungs, but air can’t get in/out, ---------- main problem, but will have ---------- too) |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure--CO2 transport alteration from ventilatory factor (good lungs, but air can’t get in/out hypercapnia main problem, but will have hypoxia too) |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- a. Abnormalities of the---------- and --------: CF, asthma, emphazema |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- a. Abnormalities of the airways and alveoli--CF, asthma, emphazema |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- b. Abnormalities of the ------: narcotic overdose, brain stem infarct |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- b. Abnormalities of the CNS--narcotic overdose, brain stem infarct |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- c. Abnormalities of the ---------: flail chest, morbidly obese, rib fractures |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- c. Abnormalities of the chest wall--flail chest, morbidly obese, rib fractures |
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VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- d. Neuromuscular Conditions--MS, -------------, ----------- |
VI. Acute Respiratory Failure
C. Categories **etiology and pathophysiology--physiologic mechanism for hypoxemia (1-4) 2. Hypercapnic Respiratory Failure-- d. Neuromuscular Conditions--MS, muscular dystrophy, dyron-brae |
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VI. Acute Respiratory Failure
D. Tissue ------------ Needs--major threat of underlying resp. failure (------------ or -----------) is inability to meet oxygen demands of tissues |
VI. Acute Respiratory Failure
D. Tissue Oxygen Needs--major threat of underlying resp. failure (hypoxic or hypercapnia) is inability to meet oxygen demands of tissues |
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VI. Acute Respiratory Failure
D. Tissue Oxygen Needs- 1. Inadequate tissue ------------ delivery--valvular disease, -------------, CHF (pump problem) |
VI. Acute Respiratory Failure
D. Tissue Oxygen Needs- 1. Inadequate tissue O2 delivery--valvular disease, anemia, CHF (pump problem) |
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VI. Acute Respiratory Failure
D. Tissue Oxygen Needs- 2. Inadequate usage, ---------- shock (tissues don’t know what to do w/ it or not using appropriately) |
VI. Acute Respiratory Failure
D. Tissue Oxygen Needs- 2. Inadequate usage-septic shock (tissues don’t know what to do w/ it or not using appropriately) |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal a. pH: 7.35-_______ b. PaO2: 80-_______mmHg |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal a. pH: 7.35-7.45 b. PaO2: 80-100mmHg |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal c. PaCO2: 35-______mmHg d. SaO2: 95-______% |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal c. PaCO2: 35-45mmHg d. SaO2: 95-100% |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal e. HCO3: 22-_______mEq/L f. Base Excess/Deficit: +/______ |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal e. HCO3: 22-26mEq/L f. Base Excess/Deficit: +/-2 |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal f. Base Excess/Deficit: +/-2 i. Amount of buffering ---------- in blood (from all buffer systems: --------- greatest) |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal f. Base Excess/Deficit: +/-2 i. Amount of buffering anions in blood (from all buffer systems-bicarb greatest) |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal f. Base Excess/Deficit: +/-2 ii. Describes amount of --------- needed to bring blood back to normal pH iii. Excess--metabolic ------------ or compensation |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 1. Normal f. Base Excess/Deficit: +/-2 ii. Describes amount of acid/base needed to bring blood back to normal pH iii. Excess--metabolic alkalosis or compensation |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 2. Analysis a. What is primary disturbance--acidosis/-----------? b. What is primary cause--respiratory/----------? |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 2. Analysis a. What is primary disturbance--acidosis/alkalosis? b. What is primary cause--respiratory/metabolic? |
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VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 2. Analysis c. Is there -------------? |
VI. Acute Respiratory Failure
E. Arterial Blood Gases (ABGs) 2. Analysis c. Is there compensation? |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy--goal PaO2 >--------- and SaO2 >---------- |
VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy--goal PaO2 >60 and SaO2 >90 |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy a. Risk of oxygen ------------ (strive for oxygenation w/ minimum ---------2) |
VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy a. Risk of oxygen toxicity (strive for oxygenation w/ minimum FiO2) |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy b. ---------- mismatch may respond well to ↑O2 (2-4L via nasal cannula) |
VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy b. VQ mismatch may respond well to ↑O2 (2-4L via nasal cannula) |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy b. VQ mismatch may respond well to ↑O2 (2-_______L via nasal cannula) |
VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy b. VQ mismatch may respond well to ↑O2 (2-4L via nasal cannula) |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy c. Shunt will likely need--------- or ----------- |
VI. Acute Respiratory Failure
F. Respiratory Therapy 1. Oxygen therapy c. Shunt will likely need facemask or intubation |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 2. Mobilization of secretions a. Coughing and positioning--good lung ----------, postural ----------- |
VI. Acute Respiratory Failure
F. Respiratory Therapy 2. Mobilization of secretions a. Coughing and positioning--good lung down, postural drainage b. Hydration and humidification--fluids help thin secretions out |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 2. Mobilization of secretions b. ------------ and ------------: fluids help thin secretions out |
VI. Acute Respiratory Failure
F. Respiratory Therapy 2. Mobilization of secretions b. Hydration and humidification--fluids help thin secretions out |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 2. Mobilization of secretions c. Chest ---------- therapy d. Airway ----------- |
VI. Acute Respiratory Failure
F. Respiratory Therapy 2. Mobilization of secretions c. Chest physical therapy d. Airway suctioning |
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VI. Acute Respiratory Failure
F. Respiratory Therapy 3. -------------- ventilation |
VI. Acute Respiratory Failure
F. Respiratory Therapy 3. Positive pressure ventilation |
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VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 1. Relief of bronchospasm: ------------ or -------- (severe) |
VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 1. Relief of bronchospasm--Albuterol or Anophelin (severe) |
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VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 2. Airway inflammation reduction (-----------, asthma)--: ------------ (quick acting, IV if acute) |
VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 2. Airway inflammation reduction (COPD, asthma)--Corticosteroids (quick acting, IV if acute) |
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VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 3. Reduction of pulmonary congestions--: ----------- (Lasix), ------------ (↑contractility for A-fib) |
VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 3. Reduction of pulmonary congestions--diuretics (Lasix), Digoxin (↑contractility for A-fib) |
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VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 4. Treatment of pulmonary infection--IV -----------, frequent ----------- samples |
VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 4. Treatment of pulmonary infection--IV antibiotics, frequent sputum samples |
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VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 5. Reduction of severe anxiety--↑--------- and ---------2 consumption (Propofol, Atavan, ----------) |
VI. Acute Respiratory Failure
G. Pharmacologic Therapy--Goals 5. Reduction of severe anxiety--↑RR and O2 consumption (Propofol, Atavan, Versed) |
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VI. Acute Respiratory Failure
H. Supportive Care Interventions 1. Treat ----------- cause (need ---------- at cellular level)--primary goal |
VI. Acute Respiratory Failure
H. Supportive Care Interventions 1. Treat underlying cause (need O2 at cellular level)--primary goal |
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VI. Acute Respiratory Failure
H. Supportive Care Interventions 2. Maintain adequate cardiac output--BP indicates cardiac function (check MAP >---------- and ---------2) |
VI. Acute Respiratory Failure
H. Supportive Care Interventions 2. Maintain adequate cardiac output--BP indicates cardiac function (check MAP >60 and SVO2) |
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VI. Acute Respiratory Failure
H. Supportive Care Interventions 3. Maintain adequate----------- concentration (ex. if anemic give blood) |
VI. Acute Respiratory Failure
H. Supportive Care Interventions 3. Maintain adequate hemoglobin concentration (ex. if anemic give blood) |
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VI. Acute Respiratory Failure
H. Supportive Care Interventions 4. Nutritional Therapy--avoid ↑------------ (metabolizes into CO2); --------------- (↑protein ↓CHO) |
VI. Acute Respiratory Failure
H. Supportive Care Interventions 4. Nutritional Therapy--avoid ↑CHO (metabolizes into CO2); pulmonary diet (↑protein ↓CHO) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)--sudden and progressive form of ------------ where alveolar capillary membrane becomes ----------- and more permeable to ------------- fluid |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)--sudden and progressive form of ARF where alveolar capillary membrane becomes damaged and more permeable to intravascular fluid |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 1. ARF can become---------- (----------% mortality) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 1. ARF can become ARDs (40% mortality) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) a. Aspiration of -------- contents b. Viral/bacterial --------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) a. Aspiration of gastric contents b. Viral/bacterial pneumonia |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) c.------------ (esp. gram (-) infection)--most common cause w/ greatest mortality (70-90%) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) c. Sepsis (esp. gram (-) infection)--most common cause w/ greatest mortality (70-90%) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) c. Sepsis (esp. gram (-) infection)--most common cause w/ greatest mortality (70-_____%) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) c. Sepsis (esp. gram (-) infection)--most common cause w/ greatest mortality (70-90%) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) d. Severe massive ---------- e. Multiple ---------- transfusions f. ----------------- bypass |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) d. Severe massive trauma e. Multiple blood transfusions f. Cardiopulmonary bypass |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) g. Consequence of multiple ------------- dysfunction syndrome (MODS) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 2. Etiology--predisposing factors stimulate inflammatory/immune response (cause unknown) g. Consequence of multiple organ dysfunction syndrome (MODS) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)--1-_______days after insult/injury (usually within ______hrs) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)--1-7days after insult/injury (usually within 24hrs) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- i. Injury inflammatory mediators ↑ cap ------------, interstitial edema, ----------- edema, ---------------- shunting (fluid filled alveoli exchange gas) V/Q mismatch ↓gas exchange refractory hypoxemia |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- i. Injury inflammatory mediators ↑ cap permeability interstitial edema alveolar edema intrapulmonary shunting (fluid filled alveoli exchange gas) V/Q mismatch ↓gas exchange refractory hypoxemia |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- i. Injury inflammatory mediators ↑ cap permeability interstitial edema alveolar edema intrapulmonary shunting (fluid filled alveoli exchange gas) ------------- mismatch, ↓----------- exchange, refractory ------------ |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- i. Injury inflammatory mediators ↑ cap permeability interstitial edema alveolar edema intrapulmonary shunting (fluid filled alveoli exchange gas) V/Q mismatch ↓gas exchange refractory hypoxemia |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- ii. ------------- 1 2 (produce surfactant) cell damage, ↓------------, ↓----------- compliance and recall widespread atelectasis ↓lung compliance |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- ii. Alveolar 1 2 (produce surfactant) cell damage ↓surfactant ↓alveolar compliance and recall widespread atelectasis ↓lung compliance |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- ii. Alveolar 1 2 (produce surfactant) cell damage ↓surfactant ↓alveolar compliance and recall, widespread -------------, ↓lung ------------ |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- ii. Alveolar 1 2 (produce surfactant) cell damage ↓surfactant ↓alveolar compliance and recall widespread atelectasis ↓lung compliance |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- iii. ----------- membrane lines alveoli fibrosis, ↓--------- exchange ↓ --------------- (stiff lung-need high pressure to get air into) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology a. Injury (Exudative Phase)- iii. Hyaline membrane lines alveoli fibrosis ↓gas exchange ↓ compliance (stiff lung-need high pressure to get air into) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology b. ---------------(Proliferative Phase)--1-2weeks after initial injury |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology b. Reparative (Proliferative Phase)--1-2weeks after initial injury |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology b. Reparative (Proliferative Phase)--1-2weeks after initial injury i. Inflammatory response dense, ---------- tissue ↑pulmonary ------------ resistance, pulmonary ------------, ↓lung compliance hypoxia |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology b. Reparative (Proliferative Phase)--1-2weeks after initial injury i. Inflammatory response dense, fibrous tissue ↑pulmonary vascular resistance, pulmonary HTN, ↓lung compliance hypoxia |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology b. Reparative (Proliferative Phase)--1-2weeks after initial injury ii. If phase ends ------------ resolve iii. If phase persists widespread ----------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology b. Reparative (Proliferative Phase)--1-2weeks after initial injury ii. If phase ends lesions resolve iii. If phase persists widespread fibrosis |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. ----------- (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) i. Lung remodeled by sparsely ------------ and ------------ tissues (diffuse scarring and fibrosis) ↓lung compliance (stiff lung) and ↓ gas exchange surface area hypoxia and pulmonary HTN |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) i. Lung remodeled by sparsely collagenous and fibrous tissues (diffuse scarring and fibrosis) ↓lung compliance (stiff lung) and ↓ gas exchange surface area hypoxia and pulmonary HTN |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) i. Lung remodeled by sparsely collagenous and fibrous tissues (diffuse scarring and fibrosis) ↓---------- (stiff lung) and ↓ --------- exchange surface area hypoxia and pulmonary --------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) i. Lung remodeled by sparsely collagenous and fibrous tissues (diffuse scarring and fibrosis) ↓lung compliance (stiff lung) and ↓ gas exchange surface area hypoxia and pulmonary HTN |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) ii. Survival rate poor and will need long term------------- ventilation |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 3. Pathophysiology c. Fibrotic (Chronic/Late Phase)--2-3 weeks after initial injury (irreversible changes) ii. Survival rate poor and will need long term mechanical ventilation |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations a. ↑---------- (work of breathing), tachypnea b. T----------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations a. ↑WOB (work of breathing), tachypnea b. Tachycardia |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations c. ----------, pallor, ------------ d. ↓---------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations c. Cyanosis, pallor, diaphoresis d. ↓Mentation |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations e. Diffuse ----------- and rhonchi f. CXR--“------------” |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations e. Diffuse crackles and rhonchi f. CXR--“white out” |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations g. ABGs--resp -------------- (initially) decompensates to combined met and resp ------------ |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations g. ABGs--resp alkalosis (initially) decompensates to combined met and resp acidosis |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations h. Elevated---------- with normal ---------: --key finding |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations h. Elevated PAP with normal PAWP--key finding |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations h. Elevated PAP with normal PAWP--key finding i. Can’t ----------- w/ ↑------------ b/c could be something else (heart prob fluid overload) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations h. Elevated PAP with normal PAWP--key finding i. Can’t Dx w/ ↑PAWP b/c could be something else (heart prob fluid overload) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations h. Elevated PAP with normal PAWP--key finding ii. If fluid overloaded ↑-------------- (backing up of fluids causing too much preload) give diuretics, ---------2 improves not ARDS |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 4. Clinical Manifestations h. Elevated PAP with normal PAWP--key finding i. Can’t Dx w/ ↑PAWP b/c could be something else (heart prob fluid overload) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 5. Diagnosis Criteria a. --------------: --do not respond to ↑FiO2 |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 5. Diagnosis Criteria a. Refractory hypoxia--do not respond to ↑FiO2 |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 5. Diagnosis Criteria b. CXR with new bilateral -----------/--------------- infiltrates (“white out”) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 5. Diagnosis Criteria b. CXR with new bilateral interstitial/alveolar infiltrates (“white out”) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 5. Diagnosis Criteria c. PAWP of --------------mmHg or less and no evidence of heart failure d. Must have predisposing condition for ARDS within -----------hrs (need trigger) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 5. Diagnosis Criteria c. PAWP of 18mmHg or less and no evidence of heart failure d. Must have predisposing condition for ARDS within 48hrs (need trigger) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment a. Prevention of further ---------- b. Maintain adequate -----------: --mechanical ventilation (5-6mL/kg w/ lowest FiO2) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment a. Prevention of further injury b. Maintain adequate oxygenation--mechanical ventilation (5-6mL/kg w/ lowest FiO2) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment b. Maintain adequate oxygenation--mechanical ventilation (5-_________mL/kg w/ lowest ______2) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment b. Maintain adequate oxygenation--mechanical ventilation (5-6mL/kg w/ lowest FiO2) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment b. Maintain adequate oxygenation- i.-------------- Ventilatory--high frequency (100-_________breath/min) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment b. Maintain adequate oxygenation- i. Jet Ventilatory--high frequency (100-300breath/min) |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment b. Maintain adequate oxygenation- ii.---------------- ratio--sedate and paralyze iii. --------------- strategies |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment b. Maintain adequate oxygenation- ii. Inverse ratio--sedate and paralyze iii. Positioning strategies |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment c. Optimize oxygen delivery--keep PaO2 >----------- and SaO2 >----------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 6. Treatment c. Optimize oxygen delivery--keep PaO2 >60 and SaO2 >90 |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management a. Mechanical ----------- b. -------------- strategies |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management a. Mechanical ventilation b. Positioning strategies |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management b. Positioning strategies i. Lateral ------------ ii. ------------ position (-----------)--lungs down |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management b. Positioning strategies i. Lateral decubitus ii. Prone position (proning)--lungs down |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management b. Positioning strategies ii. Prone position (proning)--lungs down • ↓----------------, improves perfusion and ventilation, reduces lung constriction |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management b. Positioning strategies ii. Prone position (proning)--lungs down • ↓atelectasis, improves perfusion and ventilation, reduces lung constriction |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management c. Drug Therapy--no good drug i. _________--antifungal ii. ________--controversial iii. ________ oxide--dilate pulmonary vasculature |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 7. Management c. Drug Therapy--no good drug i. Ketoconadol--antifungal ii. Steroids--controversial iii. Nitrous oxide--dilate pulmonary vasculature |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications a. Noscomial --------- b. --------: --can cause and also lead to c. Pulmonary ------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications a. Noscomial pneumonia b. Sepsis--can cause and also lead to c. Pulmonary emboli |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications d. Pulmonary --------- f. Stress ------------ and hemorrhage g. Acute --------- failure |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications d. Pulmonary fibrosis f. Stress ulceration and hemorrhage g. Acute renal failure |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications h. A---------- i. Decreased ------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications h. Arrhythmias i. Decreased CO |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications j. DIC (disseminated------------ coagulation-often in sepsis)/T-------------- |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications j. DIC (disseminated intravascular coagulation-often in sepsis)/Thrombocytopenia |
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VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications k. MODS (multiple ------------ dysfunction syndrome) |
VI. Acute Respiratory Failure
I. Acute Respiratory Distress Syndrome (ARDS)-- 8. Complications k. MODS (multiple organ dysfunction syndrome) |