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92 Cards in this Set

  • Front
  • Back
Mechanisms of abdominal pain include?
1-obstruction
2-peritoneal irritation
3-vascular insufficiency
4-mucosal alteration
5-altered motility of bowel
6-capsular distention
7-metabolic disorders
8-nerve injuries
9-abdominal wall injuries
10-or pain referred from extra-abdominal sites
Evaluator needs to identify?
emergent from non-emergent causes in an expedient and cost-effective manner.
Evaluation begins with?
a thorough history
PE of abdomen includes?
inspection, auscultation of BS, vascular sounds, percussion, palpation for masses and/or tenderness.
What other examination should be done for the abdomen?
for groin abnormalities
DRE
check peripheral pulses and other sources of referred pain (i.e.lungs, CV system, head and neck, musculoskeletal, R/O lymphadenopathy)
Work-up includes for abdomen?
-CBC -UA, C & S
-CXR, EKG, prn-further testing usually includes abdominal U/S, CT with contrast
KUB, prn
WBC > 12 usually indicates?
inflammation or infectious condition. When WBCs are well over 12, usually, see increase in bands and segs with left shift (increase in immature forms)
Obstruction?
distention and increased bowel wall tension induce a response by pain receptors in the bowel, biliary tree, and uterus
Small bowel obstruction induces?
severe pain with a jejunal obstruction
Proximal obstruction often induces?
vomiting
Initally, with proximal bowel obstruction, stool passed may range from?
small volume to diarrhea, then with complete obstruction, stool passage ceases.
Distal obstruction leads to?
distention
Large bowel obstruction is often preceded by?
constipation or a change in bowel habit
With large bowel obstruciton, what diagnostic test is usually helpful?
KUB is helpful to demonstrate loops of distended bowel (“slinkys”).
Biliary colic?
= cystic duct obstruction by a stone
What are common S/S with common duct obstruction?
jaundice, vomiting, increased alk phos and bilirubin.
Urinary tract obstruction?
may be due to a stone blockage of a ureter. Occurs with sudden onset crampy flank pain with radiation to the groin and lower abdomen. With bladder obstruction, lower abdominal distention and suprapubic pain are usual.
Peritoneal irritation produces?
severe pain: sharp, burning, aching. Pt is still rather than restless
Familial Mediterranean fever causes recurrent?
acute abdominal pain with peritoneal irritation, pleuritis and synovitis, abdominal distention, Increased ESR. Usually treated with colchicine.
Vascular disease?
acute arterial insufficiency secondary to thrombus or atherosclerosis
Aortic dissection produces what kind of pain?
very severe pain that radiates to the back or genitalia.
Mucosal injury, PUD induces what kind of pain?
“gnawing” pain, burning.
Inflammation due to IBS, acute gastroenteritis usually causes what kind of pain?
diffuse pain.
Altered motility, includes that caused by irritable bowel syndrome, is characterized by?
crampy, lower abdominal pain
Tenesmus?
term used to describe feeling or urge to defecate but without needing to pass stool.
Functional dyspepsia?
“irritable gut syndrome” characterized by early satiety, bloating and nausea.
Acute ileus secondary to?
trauma, infection, peritonitis, abdominal surgery, narcotics or anticholinergics
Capsular distention, is secondary to?
edema of liver or spleen.
Splenic distention is noted for what kind of pain?
pain that radiates to the ipsalateral shoulder
Metabolic disorders?
ketoacidosis
porphyria?
fever, increased white cells, soft abdomen
Nerve injury?
from zoster, or secondary to pancreatic CA.
Abdominal wall pathology secondary to?
muscular injury to the abdominal wall.
Referred pain?
especially from lower lobe pneumonia, pulmonary infarct. Acute inferior wall MI may present with upper abdominal pain, nausea and vomiting.
Most emergent causes of referred pain include problems resulting in?
obstruction, peritoneal insufficiency or vascular insufficiency.
For irritable bowel syndrome, the Manning criteria are often helpful in diagnosis
in presence of all 6 criteria, the predictive value is 88-90%
Manning criteria are?
1 -visible abdominal distention
2-pain relief with defecation
3-with onset pain, increased number of stools
4-with onset pain, looser stools
5-rectal mucus passage
6-feeling of incomplete evacuation
The Rome criteria, which are refined Manning criteria, require?
that symptoms continue for at least 3 months, on a continuous or recurrent basis.
For the Rome criteria, there needs to be evidence of abdominal pain or discomfort associated with any or all of the following?
1-relief with defecation
2-change in stool consistency
3-change in stool frequency
+ PLUS, two or more of the following....
For the Rome criteria, there needs to be evidence of abdominal pain or discomfort associated with any or all of the following? + PLUS, two or more of the following?
1-altered stool frequency (> 3 BM qD or < 3 per week)
2-altered stool form (lumpy, hard, or loose/watery)
3-altered stool passage (urgency, incomplete evacuation, straining)
4-mucus passage
5-bloating or feeling of abdominal distention
Rome criteria for functional dyspepsia?
“ulcerlike variant” of predominantly abdominal pain
Constipation tx is often?
is self-treated, is also a frequent reason for an office visit.
Definition of constipation?
diverse interpretation usually involves les frequent passage of stool than normal, or passage of hard stool
Constipation is a common disorder secondary to?
low fiber diet consumed by most Americans.
Results from either difficulty?
filling rectum by colonic transport or with reflex stool defecation.
Aids to prevent constipation are?
1-increase decaf. Fluid intake
2- increase fiber, increase exercise to promote bowel motility
Incidence of constipaton increases with?
co-existing hypokalemia (may occur secondary to laxative abuse or increased diuretic use)
Constipation occurs in >50% of pts with colon CA secondary to?
1-obstruction from tumor
2-common presentation of Crohn’s disease or irritable bowel
3-with drug use, especially opiates (including codeine)
4-calcium carbonate
Work-up for constipation includes?
A careful H & P
1 include wt
2 skin
3 abdomen
4 rectal with guiac testing
5 neuro exam
For constipation, try ?
Therapeutic measures (first, D/C all OTC meds)
1-bowel training
2-increase decaf fluids
3-increase exercise increase fiber in diet (bran, fruits, green veggies, whole grain breads and cereals) to 15 gm per day.
Increasing fiber may also increase flatulence or bloating for up to one month after increase, then fades.
Nondigestible fiber sources include?
psyllium (Metamucil), methylcellulose (Citrucel): these must be used with increased fluids
Laxatives are NOT preferred?
as first line therapy.
For the elderly client who doesn’t respond to the above measure: consider use of?
lactulose, sorbitol for long term use.
Avoid stimulant or irritant laxatives, like?
e.g. Pericolace, senna, cascara, or protokinetic agents, e.g. cisapride, since they may induce ventricular arrhythmias (including TdP, v fib, v tach, QT prolongation). Enemas are okay, only for fecal impaction.
Work up labs for constipation?
1 potassium
2 calcium
3 TSH
4 glucose
5 HgA1C
&amp; Colonoscopy
Appendicitis' general presentation?
classically pain occurs first, then nausea, vomiting, anorexia. Onset periumbilical pain, gradually migrates over hours to RLQ
For appenicitis, on PE, check?
VS
Cholecystitis?
stones may block duct inducing acute biliary colic, or be chronic
RF for cholecystitis?
women are affected more often than men (female, forty, fat, fertile), increases with increased age.
Presentation of cholecystitis?
Abrupt onset, severe 10/10 pain, epigastric, RUQ with radiation around to back, straight through to back or to right shoulder. Accompanied by nausea, often with vomiting, often awakens pt 1-3 AM, lasts several hours (8-12), unrelated to food intake.
How to relieve pain of cholecystitis can be relieved by?
Narcotics relieve pain, if not: surgical consult., or if pt presents with fever and increased WBCs.
Versus cardiac pain, what is different with pt who has cholecystitis?
Pt is usually restless in an attempt to relieve pain (vs. cardiac pt is quiet)

With PE & labs, Appendicitis may present?
+ Murphy’s.
Tx for cholecystitis?
IV meperidine for pain, NPO.
If pt refuses surgery, drug therapy with?
ursodiol (Actigal) may be tried for cholesterol stone dissolution (not indicated for bile stones). Dose is 8-10 mg/kg/day in BID, TID dosing: available as 300 mg capsules.
Actigal's contraindicated for which kind of stones?
calcified stones.
Indications for Actigal, are for?
pt who refuse surgery and for those in whom prevention on gallstone development during rapid weight loss is problematic.
Safety over 24 months of use not established
many interactions have been noted, especially with OCPs, aluminum based antacids, cholestyramine, estrogen.
Recurrence is common: 50% of pts + within ?
5 years.
SE of Ursodiol?
GI side effects with ursodiol.
Diverticula?
are out-pouchings from the large intestine
Diverticula occurs most commonly in which part of the intestines?
the sigmoid colon
Diverticual presents usually ?
LLQ pain, sometimes affects the RLQ or suprapubic area Pain is severe and steady, often associated with urinary frequency Pain increases with ambulation, coughing, or moving.
On PE, diverticulosis presents?
normal BS, extreme LLQ tenderness with +/- palpable LLQ mass.
What do you do for pt's with diverticulosis that look toxic?
Hospitalize the toxic for IV antibiotics (the febrile)
What tests are contraindicated to do for diverticulitis? Why?
Recommendation for pta with diverticulitis?
High fiber, low fat diet is recommended, gradually increasing amount fiber over time to avoid distention or flatulence..
How is fiber administered to pts with diverticulitis?
in form of dietary sources or hydrophillic colloids (Metamucil, one tablespoon qD or BID).
Acute pancreatitis: high association with?
chronic alcoholism or gallstones (80%)
Pancreatitis also occurs as?
idipathic, or secondary to perforated peptic ulcer disease, trauma, neoplasm, hypercalcemia, hyperlipidemia, drug use, viral infections.
Some 75-85% of pts with mild disease resolve. Mortality in mild disease <1%, but mortality in severe disease approaches ?
30%.
The pancreas is a unique structure since it is the only?
endocrine and exocrine gland in the body
Insulin and glucagon are released secondary to which function of the pancreas?
endocrine function
Enzymes of the pancreas?
These enzymes are lipase, amylase, and trypsin.
Trypsin?
digests proteins
Amylase and lipase digest?
carbs and fats.
The pancreas is composed primarily of?
protein.
The manufactured enzymes are stored as?
an inactive form so the pancreas doesn’t digest itself.
Pancreatitis occurs when?
trypsin is activated. The mechanism is uncertain.
Pancreatitis usually affects?
those between 40-60 years of age