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64 Cards in this Set
- Front
- Back
Contents of breast milk: (5)
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Carb:Lactose
Protein:Whey & Casein IgA, IgG enzymes |
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Discharge Criteria: (8)
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Feeding: q2-4h
Elimination: meconium,void Circumcision:void Color:pink,no juandice Cord: drying Newborn Screen. Com.:docu. Stable VS: esp. temp Activity: all 4 ex., carseat!! |
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Caput Succedaneum-
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collection of blood and serum underneath the scalp
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Subgleal hemorrhage-
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collection of blood- boggy skull
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Cephalhematoma-
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compartmentalized- ruptured capillaries
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Erythema toxicum-
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flea bite dematitis- very common rash, small lesions first presenting on face then moves down. Not seen on palms or soles of feet. Lasts 5-7 days.
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Bullous Impetigo-
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impetigo neonatorum- seen on babys trunk first. Caused by staph. aureas. Isolation required. Tx: oral/top. antibiotic
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Hyperbilirubinemia-
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increased con./uncon. bilirubin in blood. S/S: jaundice.
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Physioloic Jaundice:
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Most common. Causes: RBC hemolysis, immature liver fn. Tx: nothing or phototherapy.
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Breast Milk Jaundice:Early
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Causes: Lack of breast milk. Tx: frequent feedings, phototherapy. Onset:24h
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Breast Milk Jaundice: Late
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Onset:5-7d. Caused by contents of breast milk. Mon. closely.
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Normal bilirubin?
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0.2-1.4 mg/dl unconjugated
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Jaudice will appeard when bilirbuin exceeds?
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5mg
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Rh incompatibility:
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Rh- mom and Rh+ baby: mother developes antibodies and second Rh+ baby will be affected
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Hydrops fetalis:
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Most severe: very swollen, severely anemic, common to have still births,death
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ABO incompatibility: Occurs:
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When mother has different blood type than infant, usually mom= O and baby=A/B. Does not occur if mom is AB b/c no antibodies
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What happens when ABO incompatibility occurs?
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antibodies cross placenta and attach to fetal RBCx causing hemolysis
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S/S of ABO in.:
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Jaundice in first 24h, anemia from RBC hemolysis, hepatoplenomegaly, hydrops
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Tx. of Rh sensitivity?
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Rhogam w/i 72h
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Hypoglycemia:
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Decreased serum glucose. Exact level to cause hypo. depents on gest. age, birth wt., metabolic needs and illness state.
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Patho. of hypoglycemia:
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Baby has increase in insulin.
Sepsis- b/c baby does not have glycogen stores to keep up. Mom has DM-insulin |
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S/S of hypoglycemia:
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Cerebral: Jittery, high-pitched cry, seizures, tremors, low muscle tone
Others: apnea, sweating |
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Diag. Eval. of hypoglycemia:
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chemstrip less than 40mg/dl. Two should be checked
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Hyperglycemia:
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bl. glucose above 125mg/dl in full term infant. Bl. Glucose greater than 150mg/dl in preterm infant
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Hypocalcemia: Early
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Appears in 24-48h. S/S: Jittery, apnea, cyanosis, high-pitched cry. Resovles in 1-3 days.
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Hypocalcemia: Late (patho)
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Appears in 3-4d. Seen in infants given cow milk-> intestinal malabsorbtion-> hypomagnesemia -> hyperinsulinemia
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S/S of Hypocalcemia: Late
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Tremors, seizures, tetany
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Normal calcium?
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7-8.5mg/dl
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Tx. of hypocalcemia:
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-Early feed
-IV 10% ca. gluc. over 30min |
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Nsg. Care of hypocalcemia:
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Be aware of s/s of hyperca.- vomiting, bradycardia.
Max. rest |
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ABO incompatibility: Occurs:
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When mother has different blood type than infant, usually mom= O and baby=A/B. Does not occur if mom is AB b/c no antibodies
|
|
What happens when ABO incompatibility occurs?
|
antibodies cross placenta and attach to fetal RBCx causing hemolysis
|
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S/S of ABO in.:
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Jaundice in first 24h, anemia from RBC hemolysis, hepatoplenomegaly, hydrops
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Tx. of Rh sensitivity?
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Rhogam w/i 72h
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Hypoglycemia:
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Decreased serum glucose. Exact level to cause hypo. depents on gest. age, birth wt., metabolic needs and illness state.
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Patho. of hypoglycemia:
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Baby has increase in insulin.
Sepsis- b/c baby does not have glycogen stores to keep up. Mom has DM-insulin |
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S/S of hypoglycemia:
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Cerebral: Jittery, high-pitched cry, seizures, tremors, low muscle tone
Others: apnea, sweating |
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Diag. Eval. of hypoglycemia:
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chemstrip less than 40mg/dl. Two should be checked
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Hyperglycemia:
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bl. glucose above 125mg/dl in full term infant. Bl. Glucose greater than 150mg/dl in preterm infant
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Hypocalcemia: Early
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Appears in 24-48h. S/S: Jittery, apnea, cyanosis, high-pitched cry. Resovles in 1-3 days.
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Hypocalcemia: Late (patho)
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Appears in 3-4d. Seen in infants given cow milk-> intestinal malabsorbtion-> hypomagnesemia -> hyperinsulinemia
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S/S of Hypocalcemia: Late
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Tremors, seizures, tetany
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Normal calcium?
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7-8.5mg/dl
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ABO incompatibility: Occurs:
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When mother has different blood type than infant, usually mom= O and baby=A/B. Does not occur if mom is AB b/c no antibodies
|
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Tx. of hypocalcemia:
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-Early feed
-IV 10% ca. gluc. over 30min |
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What happens when ABO incompatibility occurs?
|
antibodies cross placenta and attach to fetal RBCx causing hemolysis
|
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Nsg. Care of hypocalcemia:
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Be aware of s/s of hyperca.- vomiting, bradycardia.
Max. rest |
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S/S of ABO in.:
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Jaundice in first 24h, anemia from RBC hemolysis, hepatoplenomegaly, hydrops
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Tx. of Rh sensitivity?
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Rhogam w/i 72h
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Hypoglycemia:
|
Decreased serum glucose. Exact level to cause hypo. depents on gest. age, birth wt., metabolic needs and illness state.
|
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Patho. of hypoglycemia:
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Baby has increase in insulin.
Sepsis- b/c baby does not have glycogen stores to keep up. Mom has DM-insulin |
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S/S of hypoglycemia:
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Cerebral: Jittery, high-pitched cry, seizures, tremors, low muscle tone
Others: apnea, sweating |
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Diag. Eval. of hypoglycemia:
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chemstrip less than 40mg/dl. Two should be checked
|
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Hyperglycemia:
|
bl. glucose above 125mg/dl in full term infant. Bl. Glucose greater than 150mg/dl in preterm infant
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Hypocalcemia: Early
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Appears in 24-48h. S/S: Jittery, apnea, cyanosis, high-pitched cry. Resovles in 1-3 days.
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Hypocalcemia: Late (patho)
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Appears in 3-4d. Seen in infants given cow milk-> intestinal malabsorbtion-> hypomagnesemia -> hyperinsulinemia
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S/S of Hypocalcemia: Late
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Tremors, seizures, tetany
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Normal calcium?
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7-8.5mg/dl
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Tx. of hypocalcemia:
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-Early feed
-IV 10% ca. gluc. over 30min |
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Nsg. Care of hypocalcemia:
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Be aware of s/s of hyperca.- vomiting, bradycardia.
Max. rest |
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What is hemorrhagic disease caused by?
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Vit. K def.
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S/S of Hemorrahgic Disease:
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bruising, bloddy stools, bleeding from stick site
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Classig Hem. Dis.:
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1-7d
prolonged pT and pTT normal platelets and normal fibrinogen |
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Late onset Hem. Dis.:
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2-12 wks.
Usually breast fed infants who did not recieve Vit. k. S/S: intracranial hem, deep bruising, GI bleed |