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23 Cards in this Set

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__________ is A motor speech disorder resulting from damage to the ______ system and its pathways.
B. Ataxic Dysarthria/Cerebellar
A. apraxia of speech/basal ganglia
b. Ataxic dysarthria/cerebellar
c. apraxia of speech/cerebellar
d. hypokinetic dysarthria/substantia nigra
Ataxic Dysarthria is characterized by (6)
A. characterized by loss of the ability to execute or carry out learned (familiar) movements, despite having the desire and the physical ability to perform the movements. It is a disorder of motor planning.

B. characterized by errors in timing, stress, and articulation, harsh vocal quality, and monotonous pitch and volume.
B. errors in timing, stress, and articulation, harsh vocal quality, and monotonous pitch and volume.
Frequent causes of ataxic dyarthria are ________
A. degenerative diseases.
B. Lesions of the SMA
C. multiple strokes
D. traumatic head injuries
A. degenerative diseases.
A terminal disease, this disease is inherited and manifests in middle age.
A. Friedreich’s ataxia
B. Olivopontocerebellar degeneration
C. Cerebellar ataxia
D. Autosomal dominant cerebellar ataxia of late onset
D. Autosomal dominant cerebellar ataxia of late onset
Autosomal dominant cerebellar ataxia of late onset is characterized by?
A. balance and visual deficits, and ataxic dysarthria.
B.Muscle rigidity and limited range of motion
C.dysarthria, and visual disorders
D.progressive cerebellar ataxia, retinal degeneration, muscle rigidity, sensorineural deafness, balance deficits, and dementia
D. progressive cerebellar ataxia, retinal degeneration, muscle rigidity, sensorineural deafness, balance deficits, and dementia
Friedreich’s ataxia Symptoms appear in individuals in their _____ & cause ____ Ataxia , _____, _____ disorders.
A.limb ataxia, balance and visual deficits, and ataxic dysarthria.
B.Sensorineural, limited range of motion, rigidity.
C.cerebellar, dysarthria, and visual .
C. 20s and cause cerebellar ataxia (gait and manual dexterity), dysarthria, and visual disorders.
A progressive disorder that tends to run in families.
A.Olivopontocerebellar degeneration
B.Friedreich’s ataxia
C. Idiopathic sporadic late-onset cerebellar ataxia
A. Olivopontocerebellar degeneration
A rare, progressive, hereditary disease that attacks the spinal cord and cerebellum.
A. Olivopontocerebellar degeneration
B. Friedreich’s ataxia
C. Idiopathic sporadic late-onset cerebellar ataxia
B. Friedreich’s ataxia
______is similar to the autosomal diseases, except that is has fewer neurological signs
A. Olivopontocerebellar degeneration
B. Friedreich’s ataxia
C. Autosomal dominant cerebellar ataxia of late onset
D. Idiopathic sporadic late-onset cerebellar ataxia
D. Idiopathic sporadic late-onset cerebellar ataxia
Cerebellar strokes have distinctive signs: (4)
A.Sensorineural, limited range of motion, rigidity, ataxic dysathria
B.retinal degeneration, muscle rigidity, sensorineural deafness, balance deficits, and dementia
C.balance and visual deficits, Muscle rigidity and ataxic dysarthria.
D.limb ataxia, balance and visual deficits, and ataxic dysarthria.
D. limb ataxia, balance and visual deficits, and ataxic dysarthria.
Toxic conditions associated with cerebellar dysfunction include: (4)
Lead and mercury poisoning
Chronic and acute alcohol consumption
Phenytoin (Dilantin: anti-seizure drug)
Exposure to cyanide and acrylamide
L & M
Chronic and Acute...
Anti....drug
Exposure to c...
METABOLIC CONDITIONS associated with cerebellar dysfunction include: (3)
Prolonged vitamin E or B12 deficiency
Severe cases of hypothyroidism
Wilson’s disease (Wilson's disease, also called hepatolenticular degeneration, is a rare autosomal recessive inherited disease caused by an accumulation of copper in tissues all over the body, mainly in the liver, brain, kidneys and cornea).
Vitamin _ & _ deficiency
Severe hypo...
Accumulation of ___ = Wilson's Dz.
Cerebellar damage may occur in those whose ____ has been ___and ____by the force ____ ___.
A. Brain, damaged and hurt, car accident
B. cerebellum, distorted and stretched, head trauma
C. Basal ganglia, distorted and stretched, head trauma
B. cerebellum, distorted and stretched, head trauma
primary cancerous tumor seeds a secondary tumor =
Metastatic tumors
Metastatic tumors
Low-grade astrocytomas
Hemangioblastomas
Three primary characteristics of ataxic dysarthria are:
A. Imprecise consonants, Excess and equal stress, Irregular articulatory breakdown
B. Prosodic disturbances, Silent pauses, Placement errors
A. Imprecise consonants, Excess and equal stress, Irregular articulatory breakdown
True or False Prosodic errors in ataxic dysarthria include
Prolonged phonemes
Prolonged intervals between phonemes
Monopitch
Monoloudness
Slow rate
True
The most prominent phonatory deficit in Ataxic Dysarthria is?
A.Intermittent hyponasality
B.Voice tremor
C.Harsh voice qualiy
C. harsh voice quality
______ _____ ___ may be caused by decreased muscle tone in the laryngeal and respiratory structures.
harsh voice quality
HVQ
Resonance errors: Intermittent hyponasality may be evidenced by some individuals because of
A. Muscle Strength
B. Timing Errors
C. Reduced Rate
D. Reduced Vital capacity
B. timing errors between the muscles of the velum and other muscles of articulation.
There are two key evaluative tasks that are appropriate to assess ataxic dysarthria: ____ and ____ ....
Speech AMRs

Reading, conversational speech, and repeating sentences containing numerous multisyllabic words
: Patients with this dysarthria will be unable to sustain a steady rhythm as they repeat the target sound

to evaluate irregular articulatory breakdowns.
Tx for Prosody:
Reciting syllables to a metronome using familiar passages;
Finger or hand tapping while reciting a familiar passage;
Cued reading material using visual cues for reading sentences or paragraphs.
RC = R/F/C
Tx for Stress and Intonation:
Contrastive stress drills using clinician questions and patient answers emphasizing appropriate stress on target words;
Pitch range exercises using singing and printed materials with up and down arrows;
CSD/PRE
Tx for Articulation Errors: (4)
Intelligibility drills (Previously discussed)
Phonetic placement in which the clinician instructs the patient about correct placement of the articulators for a target phoneme;
Exaggerated consonants (previously discussed)
Minimal contrast drills (previously discussed).
ID/PP/ExC/MCD