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32 Cards in this Set

  • Front
  • Back
what functions are laterized on
r side
non verbal - visual material [ faces]
auditory - music
geograhpical info [r temporal r occipital
l side
verbal information , major speech centers
anterior posterior gradient
posterior - sensation and perception [auditory, visual, somatosensory]
anterior - effector systems
motor strip
ant to rolandic sulcus
what constitutes temporal lobes
mesotemporal region
hippocampal complex
entorhinal, perirhinal cortices, hippocampus,
temporal lobes
mesotemporal region
ant portion parahippocampal gyrus
hippocampal complex fuunction
declaritive memory
acquisition of new factual knowledge - anterograde memory imparied in injury
L side for verbal
R side for non-verbal
hippocampal complex inputs
assoc cortices of temporal lobes which receive feedback connections from hipocampus
amygdyla function
potentiates memory traces of emot stimuli = emot signif in social situations
role in autonomic conditioning
loss ability reconize faces
temporal poles [anterior lateral] and inferior temporalregions
L side injury
loss naming = lexical retreival
naming from different conceptual categories
injury L temporal pole
retreval of proper nouns
injury L IT
retreival of common nouns
injury L occipitotemporal junction
severe prosopagnoisia
occipital lobe = OL
visual association cortices -lateral - brodman 18 19

primary visual cortices - mesial
occipital lobe anat
clcerine fissure divides dorsal and ventral
dorsal component occiptial lobes
primary visual cortex superior calcerine fissue, asssociation cortex [brodman 18-19]
loss form vision in inferior visual field
injury to dorsal component plus posterior part parietal lobe [brodman 17]
balint's syndrome
injury sssoc cortex and adjacent pareitla lobes
1- visual disorientation
2-ocular apraxia
3- optic ataxia
visual disorientation
inibility to atend to more than small part of visual field - tends to e unstable and attention shifts without warning
ocular ataxia
loss of ability to execute purposeful movement using vision
ocucular apraxia
loss ability to scan and direct vision twoardsa and object [usually in periph vision]
ventral component
primary visual cortex below calcerien fissure and increiorportions of brodman 18-19
injury to ventral component OL
along with injury to ociptior temporal junction

loss form vision superior visual field -contralterally or bilaterall
types of visual disturbance
loss form vision - blindess
visual disorientation = simultanagnosia
ocular apraxia = psychic gaze paralysis
otpic ataxia
acquired central achromatopsia
inferior association cortex and adjacent white matter
loss color vision but not form vision
can be quadrant, hemifield, bilat
acquired [pure] alexia
severs l and r visual association cortex from dominent temperoparietal cortices - interupts fibers from corpus collusum and visual assoc cortex
color agnosia
inbility recognize colors though can match it
lision in dominant OC incl splenium and corups callosum
color anomia
inability name a color though able point to it
central achromatopsia
complete inability to preceive colot
anton's syndrome
failure to acknowledge blindness
bilat OL lesions caused by
stroke, anoxia, herniation, metabolic encephalopathy
grestmann's syndrome
dominant parietal lobe
acuculalia, agraphia, L-R disrimiation, finger agnosia