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63 Cards in this Set

  • Front
  • Back
What are the 2 major motor control systems?
extrapyramidal
pyramidal
FUNCTION pyramidal system
initiated, voluntary skeletal mm activity
FUNCTION extrapyramidal system
involuntary skeletal mm activity
DEFINE supplemental motor area
association area in frontal lobe
FUNCTION supplemental motor area
finessed movement:
*initiation of movement
*involved w/ orientation of the eyes/head
*planning sequential and bi-manual movements
DEFINE pre-motor area
association area ant to motor cortex
FUNCTION pre motor area
controls trunk, pelvic and pectoral girdle musculature (change posture)
DEFINE broca's area
association area in frontal lobe
FUNCTION broca's area
instigate speech
What percentage of the coricospinal tracts decussate in the pyramids of the medulla?
90%
FUNCTION corticofugal tract
inn voluntary skeletal mm activity from the extremities and trunk
FUNCTION corticobulbar tract
inn voluntary skeletal mm activity of the head and neck, mm of facial expression, extrinsic eye mm, tongue, mastication, neck, pharynx, larynx, and scalp (includes ALL Cranial NN w/ motor fx)
FUNCTION modulatroy descending motor tract
modify the effects of the CORTICOSPINAL tract in that they refine and finesse the activity of lower motor neurons, which receive input from the UMNs of the corticospinal tract
What are the modulatory descending motor tracts?
rubrospinal tract
tectospinal tract
vestibulospinal tract
reticulospinal tract
FUNCTION rubrospinal tract
influence is biased towards excitation for flexor mm and inhibition for extensor mm
FUNCTION tectospinal tract
involved w/ reflex postural movements of head, neck, and UE in response to visual stimuli
FUNCTION vestibulospinal tract
righting reflexes in response to activation of the vestibular system
FUNCTION reticulospinal tract
excitatory to extensor mm activity and inhibits flexor mm activity
Which cranial nn have a unilateral projection pattern (ie-do not decussate)?
CN VII and XII
FUNCTION oculomotor n
inn extrinsic eye mm (superior rectus, medial rectus, inferior rectus, and inf oblique)
inn iris and ciliaty body
FUNCTION trochlear n
inn sup oblique eye mm
FUNCTION trigeminal n
inn mm of mastication
FUNCTION abducens n
inn lateral rectus eye mm
FUNCTION facial n
inn the muscles of facial expression
inn submandibular, sublingual, and lacrimal glands
FUNCTION glossopharyngeal n
inn the stylopharyngeus m of the pharynx
inn parotid gland
FUNCTION vagus n
inn mm of the larynx, pharynx and soft palate
Inn viscera of the thorax, abdomen, and pelvis
FUNCTION spinal accessory n
inn SCM and trapezius mm
inn the intrinsic laryngeal mm
FUNCTION hypoglossal n
inn extrinsic and intrisic mm of tongue via genioglossus m
What are classical signs of UMN damage?
paresis
paralysis
exaggerated DTR
clonus
spastic paralysis
hypertonia
DEFINE paresis
weakness b/c skeletal mm are receiving less input
DEFINE paralysis
loss of movement; large range of less bc of less input
DEFINE exaggerated DTR
hyperreflexia (simple reflex arc more active
DEFINE clonus
spasms w/ alterations of contractions and relaxation in rapid succession of antagonistic and agonistic mm
DEFINE spastic paralysis
characterized by involuntary contraction of 1 or more mm w/ loss of fx
DEFINE hypertonia
incr mm tone
When do contralateral affects occur with a damaged UMN?
when damage occurs BEFORE decussation
Do you still have a reflex arc when there is UMN damage? why?
yes; bc LMN is not damaged therfore reflex arc still occurs to stimuli (there is just no voluntary action that requires input from UMN and cerebral cortex
DEFINE babinski test for UMN lesion
run an object up the lateral side of the foot
normal: toes will plantarflex
positive: toes will dorsiflex and the great toe fans
How do LMN get damaged?
can damage the ventral horn itself
can damage a peripheral n
What are classical signs of LMN damage?
paresis
flaccid paralysis
hypotonia
decreased or absent DTR
atrophy
fibrillations/fasciculations
DEFINE flaccid paralysis
total loss of mm tone w/ resultant loss of fx
DEFINE hypotonia
decrease mm tone
DEFINE decreased or absent DTR
wiped out LMN part of reflex arc, therfore no reflex
DEFINE atrophy
reduction in size of skeletal mm as a result of decreased tone
DEFINE fibrillations/fasciculations
spontaneous activity of skeletal mm
On which side will symptom appear in relation to the damaged LMN?
ipsilateral
What are collective responses of LMN?
spinal reflexes
rhythmic patterned movements
central pattern generators (CPG)
What are some spinal reflex responses?
DTR
noxious stimulation reflexes
DEFINE rhythmic patterned movements
predictable involuntary movements involved w/ specific motor activities (some hardwired since birth, some learned)
DEFINE central pattern generators
used to delineate rhythmic patterns
Where are central pattern generators set up and remembered in?;
basal ganglia
SC
brainstem
Cerebellum
What are characteristics of SC reflexes?
segmental in nature
may involve propriospinal loops
can be modulated by supraspinal influences
Which tracts can modulate SC reflexes?
rubrospinal tract (bias toward flex)
reticulospinal tract (bias toward extensors)
What are the 4 fundamental anatomical parts to a SC reflex?
*receptor organ on distal end
*afferent sensory neuron
*efferent motor neuron
*an effector organ
DEFINE flexor reflexes
(aka protective reflexes) activated by type II and IV fibers
DEFINE DTR/stretch reflex
contraction of agonistic and synergistic mm following the stretching of agonistic mm ( activates type 1a fibers)
FUNCTION DTR reflex
maintain upright posture and mm tone
DEFINE receptor organ
Muscle spindles (type 1a fibers)
What fibers are inside muscle spindles?
intrafusal fibers
What are the 3 types of muscles spindles?
dynamic nuclear bag (Ia)
static nuclear bag (II)
nuclear chain fiber (II)
FUNCTION intrafusal fibers
differentiate between static and dynamic change in length and rate of change in length of skeletal mm
DEFINE extrafusal fibers
muscle fibers
DEFINE noncontractile portion of intrafusal fibers
middle of intrafusal fiber that does no contract