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62 Cards in this Set

  • Front
  • Back
Akinesia (Dyskinesia)
impaired ability to initiate voluntary and spnotaneous motor responses such as the interruption of performance in movement when attention is distracted
Fasciculations
Rapid flickering twitching movements of a part of a muscle occurring irregularly in time and location
Chorea
abrupt irregular movements of short duration involving the hands fingers arms face tongue and head
Huntingtons chorea
degenerative disease of the basal ganglia in the brain. It is characterized by abnormalities in postural reactions, trunk rotation, distribution of tone, and extraneious movements
Tardive Dyskinesia
Side effect of neuroleptic drugs; involuntary movement disorder most often characterized by puckering of the lips tongue and or writhing of the arms and legs
Athetosis
nonrythmic. sloww writhing,sinous movements, predominantly in distal muscles; alternating postures of the proximal limbs often blend continuously to produce a flowing stream of movement. Athetosis often occurs with chorea as choreoasthetosis caused by dopaminergic activity in the basal ganglia
Dystonias
are sustained involuntary muscle contractions often distorting body posture
Dystonic
dostorted body positioning of the limbs neck trunk that is held for a few seconds and then releases
Hemiballismus
nonrythmic rapid nonsuppressable movement characterized usuually by unilateral, violent, flinging movements of the proximal arm. caused by a lesion usually an infarct around teh subthalamic nuclei
Generalized dystonia
rare, progressive and characterized by movements that result in sustained often bizarre postures often hereditary
focal dystonia
affect a single body part ; can be periodic random or progressive
Meiges Syndrom
involuntary blinking jaw grinding and grimacing
Dystonic writers cramp
occupational dystonia, focal dystonic spasms initiated by performing skilled acts such as writing or typing
Spasmodic Torticollis
begins with a pulling sensation followed by sustained torsion and deviation of the head and neck
Hemifacial spasm
unilateral painless irregular contractions of the facial muscles due to impairment of the 7th CN
o
o
o
t
t
a
f
v
g
v
a
h
olfactory
optic
occulomotor
trochlear
trigeminal
abducens
facial
vestibulocochlear
glossopharangyl
vagus
accessory
hypoglossal
paresis

paralysis
partial loss of motor movement (UMN)

total loss of motor movement (LMN)
Scale used to measure muscle tone
modified ashworth scale
fibrillations
brief contractions of single muslce fibers not visible
4 focal dystonias
meiges disease
spasmidoc torticollis
hemifacial spasm
dystonic writres cramp
dysdiadochokinesia
inability to perform rapid alternating movements
dysmetria
inability to control range of movement; patient often overshoots target
Anterior Cord Syndrome
loss of movement pain adn temp

still able to feel position vibration and touch
central cord syndrome
loss of movement and sensation
incomplete loss
Brown sequard syndrome
loss of movemnt on the SAME side as the cord damage

Loss of pain and temp and sensation on the OPPOSITE
C1-C3
head and neck sensation
some neck control
respirator dependant
C4
Good head and neck sensation and motor control
Scapular elevation
diaphragmatic movement
C5
full head and neck control and sensation
some shoulder strength
shoulder external rotation
shoulder abduction to 90
elbow flexion
supination
C6
forearm pronation
wrist extension
tenodesis
C7
elbow extension
wrist flexion
finger extension
allodynia
condition in which nonpainful stimuli produce painful sensations
hyperesthesia
increases sensitivity to somatosensory stimuli
hypoesthesia
decreased sensitivity to somatosensory stimuli
parathesia
feeling numb or tingling
dysesthesia
unpleasant sense to touch often pain
thermesthesia
sensitivity to head or cold
analgesia
inability to feel pain
hypalgesia
decreased sensitivity to pain
hyperalgesia
exaggerated sense of pain
charcot marie tooth disease
hereditary motor and sensory neuropathy resulting in paresis of the muslces distal to the knee
Radial nerve compression results in?
wrist drop
Sat night palsy
tennis elbow
ulnar nerve compression results in
clawhand
wallerian degeneration
is a process that results when a nerve fiber is cut or crushed, in which the part of the axon separated from the neuron's cell body degenerates.
Why do PNS regenerate and CNS does not?
Scwann cells with nerve growth factor instead of oligodendrites
anhedonia
lack of emotion
Ach
In CNS regulates ANS

In PNS facilitates actions at neuromuscluar junction
Too much ACH=

Too little ACH=
dyskinesia (involuntary muscle contractions)

paralysis
GABA
Inhibitory- without it neurons would fire uncontrollable producing seizures
too much GABA=

Too little GABA=
memory loss and inability for new learning

anxiety disorders insomnia and epilepsy
Glutamate

Too much=
Excitatory
Implicated with CVA TBI as major cause of cell damage after initail injury

seizures learning and memory
Dopamine

Too little=

Too much=
Affects motor system cognition and motivation

Parkinsons

Schizophrenia and addiction
Seratonin 5-HT

Too little=
associated with sleep emotional control equanamity pain regulation and carbohydrate binging

depression and suicidal ideation
Norepenephrine

Too much=
associated with attention to sensory information in environement

fear and panic
Substance P
peptide
mediates sensation of pain
Endorphins
pain modulators
Adenosine
facilitates sleep
depression
seratonin
NE
schizophrenia
dopamine
wakefulness and conscious levels
NE, Dopamine, ACH, Histamine, glutamate, seratonin
cognitive processes
Gaba and glutamate
motor activity
ACH
dopamine
seizure disorders
Gaba
glutamate