Neuro 2

Front 1

using touch and motor function to make a complex judgment about a familiar object.

Back 1

Stereognosis.

Front 2

using touch to identify what is being written on your hand (Fairly isolated to the hand). Lost with higher level lesions of the somatosensory system.

Back 2

Graphesthesia.

Front 3

Vibration is sensed by the:

Back 3

Dorsal column-medial lemniscus system.

Front 4

Discriminative touch is sensed by the:

Back 4

Dorsal column-medial lemniscus system.

Front 5

The ALS is tested using:

Back 5

Pin prick.

Front 6

Itch and tickle are sensed through the:

Back 6

ALS.

Front 7

Sexual sensations are sensed through the:

Back 7

ALS.

Front 8

The sensory ganglia of V and the mesencephalic nucleus of V are homologous to:

Back 8

The dorsal root ganglion (DRG).

Front 9

The chief sensory nucleus of V is homologous to:

Back 9

The dorsal column nuclei.

Front 10

The spinal nucleus is homologous to:

Back 10

The dorsal part of the dorsal horn.

Front 11

The spinal trigeminal tract is homologous to:

Back 11

The dorsolateral fasciculus.

Front 12

This modality is represented bilaterally for the face:

Back 12

Discriminative touch.

Front 13

Most information processing occurs at the:

Back 13

Axon hillock.

Front 14

Link between physical stimuli and input to the nervous system:

Back 14

Transduction.

Front 15

Link between nervous system and perception:

Back 15

Representation.

Front 16

A receptor potential is a type of:

Back 16

Generator potential.

Front 17

Graded potential generated at the sensory ending of axons:

Back 17

Receptor potential.

Front 18

The amplitude of a receptor potential is modified by:

Back 18

The stimulus.

Front 19

The 1st node of Ranvier in myelinated axons:

Back 19

The trigger zone.

Front 20

Action potential frequency is determined by:

Back 20

Receptor potential amplitude.

Front 21

Where does AM-FM conversion occur?

Back 21

At the trigger zone.

Front 22

Rapidly adapting receptors only respond during the time that:

Back 22

The membrane potential is changing.

Front 23

Slowly adapting receptors are also known as:

Back 23

Tonic.

Front 24

Rapidly adapting receptors are also known as:

Back 24

Phasic.

Front 25

Specific combinations of primary sensations:

Back 25

Derivative sensations.

Front 26

These receptors have Aa and AB neurons:

Back 26

Touch.

Front 27

These receptors have Ad and C neurons:

Back 27

Temperature, nociception.

Front 28

Fast pain is carried by:

Back 28

Ad fibers.

Front 29

Slow pain is carried by:

Back 29

C fibers.

Front 30

The periaqueductal gray matter and locus ceruleus are examples of:

Back 30

Anti-nociceptive systems.

Front 31

Rubbing your shin after you bump it is an example of:

Back 31

Gate control.

Front 32

This descending motor tract starts in the superior colliculus and travels down to innervate at cervical levels, coordinating head position and eye input:

Back 32

Tectospinal tract.

Front 33

This descending motor tract carries out the reflexive control of posture:

Back 33

The reticulospinal tract.

Front 34

A drooping lower face is indicative of:

Back 34

Damage to the corticobulbar tract on the contralateral side.

Front 35

Polio and ALS are examples of:

Back 35

Lower motor neuron disease.

Front 36

Hyperactivity of a denervated muscle:

Back 36

Fasciculaitons.

Front 37

This area of the spinal cord is underperfused and often referred to as the vulnerable zone:

Back 37

T4-T8.

Front 38

The largest neurons in the nervous system; make up about 3% of the corticospinal system:

Back 38

Betz cells.

Front 39

Axons of the lateral corticospinal system cross the midline here:

Back 39

The pyramidal decussation.

Front 40

The internal capsule is perfused by:

Back 40

Medial and lateral striates, anterior choroidal.

Front 41

Rapidly-adapting receptors sensitive to vibration:

Back 41

Pascinian corpuscles.

Front 42

A complex, rapidly-adapting receptor sensitive to light touch:

Back 42

Meissner's corpuscles.

Front 43

Medial medullary syndrome results from occlusion of branches of the:

Back 43

Anterior spinal artery.

Front 44

Lateral medullary syndrome results from occlusion of the:

Back 44

Posterior inferior cerebellar artery.

Front 45

Medial pontine syndrome results from occulsion of:

Back 45

Paramedian branches of the basilar artery.

Front 46

Lateral pontine syndrome results from occusion of:

Back 46

Long circumferential branches of the basilar artery.

Front 47

Counteracts head movements, keeping the head stable:

Back 47

Vestibulocollic reflex.

Front 48

Contracts limb mucles to counteract sway of body; also prepares you for a fall:

Back 48

Vestibulospinal reflex.

Front 49

Contracts neck muscles that are stretched:

Back 49

Cervicocollic reflex.

Front 50

Bending the neck forward elicits flexion of the upper extremeties; opposite the vestibulospinal reflex:

Back 50

Cervicospinal reflex.

Front 51

Used when developing an appropriate strategy for movement (planning):

Back 51

Premotor cortex.

Front 52

Used for programming motor sequences and in the coordination of bilateral movements:

Back 52

Supplementary motor area.

Front 53

This part of the basal ganglia receives inputs from the frontal lobes and the limbic area; underlies the emotional component of basal ganglia disorders:

Back 53

The head of the caudate nucleus.

Front 54

This part of the basal ganglia receives a lot of information from the motor cortex:

Back 54

The putamen.

Front 55

These two parts of the basal ganglia receive dopaminergic input from the substantia nigra pars compacta:

Back 55

The caudate and putamen.

Front 56

The two parts of the thalamic fasciculus:

Back 56

Ansa lenticularis and lenticular fasciculus.

Front 57

rhythmic, oscillatory, involuntary movements:

Back 57

Tremors.

Front 58

Slow, writhing motion of fingers, hands, and toes:

Back 58

Athetosis.

Front 59

Abrupt movements of limbs, facial muscles

Back 59

Chorea.

Front 60

Violent flailing of arms (due to subthalamic nucleus lesion):

Back 60

Hemiballismus.

Front 61

Persistent distorted position:

Back 61

Dystonia.

Front 62

Difficulties in initiating movements:

Back 62

Akinesia.

Front 63

Slowness in executing movements:

Back 63

Bradykinesia.

Front 64

This portion of the ALS mediates location and intensity of the stimulus:

Back 64

Spinothalamic tract.

Front 65

This portion of the ALS mediates the emotional and arousal aspects of pain:

Back 65

Spinoreticular tract.

Front 66

This portion of the ALS projects to the PAG for pain modulaiton:

Back 66

Spinomesencephalic tract.

Front 67

The ALS and DC-ML system have the same vascular supply in the:

Back 67

Midbrain.

Front 68

Controls automatic posture and gait-related movements:

Back 68

Reticulospinal tract.

Front 69

This provides the link between the idea of movement and th emotor expression of that idea:

Back 69

The basal ganglia.

Front 70

Lesions here cause disturbances in the initiation or cessation of a motor event:

Back 70

The basal ganglia.

Front 71

Trunk tremor while standing/sitting:

Back 71

Titubation.

Front 72

Errors in range and force of movement:

Back 72

Dysmetria.