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33 Cards in this Set

  • Front
  • Back
Name for Classifications of antiarrhythmics?
vaughan-williams
vaughan-williams class for quinidine?
Ia
vaughan-williams class for procainamide
Ia
vaughan-williams class for disopyramide
Ia
vaughan-williams class for lidocaine
Ib
vaughan-williams class for mexiletine
ib
vaughan-williams class for flecainide
Ic
vaughan-williams class for propafenone
Ic
vaughan-williams class for beta blockers
II
vaughan-williams class for amiodarone
III
vaughan-williams class for dofetilide
III
vaughan-williams class for sotalol
III
vaughan-williams class for verapamil
IV
vaughan-williams class for diltiazem
IV
moa of vaughan-williams class ia drugs?
block NA channels, decreasing conduction velocity, increasing refractory period, decreasing automaticity
se of quinidine?
diarrhea, stomach cramping, cinchonism (tinnitus, blurred vision, HA, N, hearing loss)
what is the metabolite of procainamide?
n-acetyl procainamide (NAPA); renally eliminated; active metabolite
black box on procainamide?
blood dyscarasias (agranulocytosis) and drug induced lupus
moa of vaughan-williams class ib?
block NA channels, little effect on conduction velocity, decrease refractory period, decrease automaticity
moa of vaughan-williams class ic?
block na channels, significantly decrease conduction velocity, little effect on refractory period, decrease automaticity
moa vaughan-williams class II?
block beta recceptors and indirectly block calcium channels
moa vaughan-williams class III?
block K channels, no change on conduction velocity, significant increase in refractory period, no change in automaticity
amiodarone infusions longer than 2 h must be admin how?
in non-polyvinyl chloride container such as glass
SEs of amiodarone?
hypotension (IV), bradycardia, hypothyroidism/hyperthyroidism (more hypo), increase in LFTs, corneal microdeposits, optic neuritis, pulmonary fibrosis, ataxia, dizziness, tremor, photosensitivity, slate blue skin discoloration, insomnia
preg cat amiodarone?
D
SEs of dronedarone?
hepatic failure, renal failure, diarrhea, n, hypokalemia, hypomagnesemia, incr scr
Betapace sub for betapace af?
no
moa of vaughan-williams class iv?
block calcium channels
moa of adenosine?
decreases conduction through av node restoring normal sinus rhythm
moa of digoxin?
causes direct av node suppression, increasing refractory period and decreasing conduction velocity. has positive inotropic effect, enhance vagal tone, and derease ventircular rate to fast atrial arrhythmias
indication for adenosine?
paroxysmal supraventricular tachycardia (not for converting afib/aflutter or ventricular tachycardia)
therapeutic range for digoxin for afib?
0.8-2.0ng/ml
s/sx of digoxin toxicity?
n/v, loss of appetite, bradycardia, blurred vision, yellow vision, ab pain, confusion , delirium, prolonged pr interval, accelerated juntional rhythm, bidirectional ventricular tachycardia