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18 Cards in this Set

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  • Back
What is Osteochondrosis? What specifically is happening to the cartilage? What is the symptoms of this change?
Defect of endochondral ossification

Increase thickness of cartilage; mineralization and ossification of cartilage fails

Asymptomatic lesion
Explain how Osteochondrosis becomes OCD.
Thicken cartilage loses vascular supply --> necrosis of deep cartilage (articular cartilage gets nutrients from joint. chondrocytes die = cleft formation) --> dissection of cartilage flap (once fissure communicated with surface of joint = acute lameness) --> detritic synovitis causes CS
What is the etiology of Osteochondrosis? (3)
Nutrition - excess Ca++; high caloric intake
Genetics
Mechanical Factors
Where are large breed/deep chested K9s most likely to develop osteochondrosis?
Shoulder - Caudal Aspect of Glenoid
What are the most common Osteochondrosis sites? Which has the best/worst Px?
Shoulder - best > Elbow > Stifle > Hock - worst
What is your typical signalment and hx for shoulder OCD? (3 for each)
Large breed
4-8m
uni-/bi-lateral

lame with excersice
pain on shoulder extension (d/t driving glenoid into Cd humoral head - ie where the lesion is), palpation of Cd humoral head
Shoulder m. atropy (fyi - supraspinatus m)
What would you look for on radiograph of possible shoulder OCD case? (2)
remodeling of Cd glenoid
subchondral bone defect mineralization of flap d/t chronic ocd
3 radiographic studies that can be used to id shoulder ocd
Radiograph - CdLCrMO
Positive contrast arthrography
arthroscopy
What is a joint mouse?
Are they dead?
Are they a big deal?
A rodent that typically can be found behind doors where the door meets the wall - sometimes a druggie rodent cousin whom can be seen smoking the ganga w/his homies on saturday nite

OR

a displaced fragment of cartilage from an OCD lesion, found in the joint capsule

no they get nutrients from joint capsule

it depends - they can become irritants depending on location/size
What is the classic lesion from stifle ocd? What tendon sheath can it effect?
lateral femoral condyle

long dig extensor
What is elbow dysplasia (at least according to what i wrote down from Dr. D)?

What are 4 types of elbow dysplasia?

Which is most important?
Dev dz of elbow jt inconguity

OCD of medial portion of humeral condyle
UAP
FMCP** most important
Ununited medial epicondyle
What part of the medial coronoid process fragments? Can it be seen on radiograph? What would make u suspect its presence?
The lateral part of MCP

No - ulna contour appears normal

2nd changes - remodeling
You suspect chronic OA d/t compromised range of motion of that joint. If bone spurs are present, should you remove them asap? What are your tx options? (2)
No - not very helpful

Elbow replacement
Medically tx
A 4m old GSD arrives at your clinic with severe lameness. You radiograph the elbow and notice an UAP. Can this be normal? What about a 14m old dog?
Yes - that growth plate does not close until 5m

Not normal >5m; can cause severe lameness
Where is hock OCD located specifically? Describe the joint space radiographically? Any specfic PE findings?
prox border of med ridge of trochlea
widen - though i though we didn't evaluate that on non-weight bearing animals, whatever, dont shoot the messenger :-D

soft ts swelling
Why is the hock the worst place to get ocd in the K9?
mod-severe OA + lameness
What the hell is this retained cartilaginous core exactly? WTF does it look like radiographicly? What are its clincial symptoms? What deformity does it typically result in? How the fuck do you fix it?
Defect in the endochondral ossification of the metaphyseal growth plate

Candle flame (normal is a V shape)

Clinically asymptomatic

Carpal valgas def - lower leg deviate3 from midline

Give approp diet + mild carpal valgas deform = spontan correction
OCD Tx
medical mang
arthrotomy vs arthroscopy - debride to subcond bone --> granulation --> fibrous ts --> fibrocartilage in 3-4wks