Study your flashcards anywhere!

Download the official Cram app for free >

  • Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off

How to study your flashcards.

Right/Left arrow keys: Navigate between flashcards.right arrow keyleft arrow key

Up/Down arrow keys: Flip the card between the front and back.down keyup key

H key: Show hint (3rd side).h key

A key: Read text to speech.a key


Play button


Play button




Click to flip

27 Cards in this Set

  • Front
  • Back
filling pressure of the right atria
filling pressure of the left heart
3 Antiarrhythmic Agent theraputic goals
maintain electricial impulses at SA node
supress irregular firing outside the pathway

control ventricular rate and cardiac output
main MOA for Anti arrhythmics
alter conduction thru atria and ventricles by affection depolorization and the level of cell excitablity and alter conduction by depressing conduction
no cardiac electrical activity, hence no contractions of the myocardium and no cardiac output or blood flow
Class I: Na Channel Blockers
MOA: decreases excitability of myocardium. lowers the inward Na flow in heart cells to depress their automaticity. And to increase their resting potential.
Na Channel Blockers are indicated for (4)
Premature Ventricular contractions and Ventricular Tachycardia
Quinidine (quinaglute)
Class I Na channel blocker

usused for atrial fib and flutter PAT’ (premature atrial tach), PVC’s and V-tach
Lidocaine (Xylocaine)
mexileten (Mexitil)
tocainide (tonocard)
Class I Na channel blocker

used to control PVC’s or tachycardia

doses sb adj in pt with CHF and liver dx
Procainamide (Pronestyl)
Class I Na channel blocker

used to treat ventricular ectopics(missed beats), V-tach, & Atrial arrhythmias

negative inotropic effect
& increases vascular resistance.
USED supra ventricular tach, V-tach,PAT’s, atrial flutter and fib (used for everything)
Moricizine (Ethmozine)
Flecainide (Tambocor)
propafenone (rythmol)
Class I Na channel blocker

act alittle like class II
slows conduction velocity more than the oger agents.

can cause slow contractility
Class II Drugs: Beta Blocker
used to control hypertension.
extends AV conduction reducing HR & contractile force.
propanolol (inderal)
esmolo (brevibloc)
Metaprolol (Lopressor)
Class II Drugs: Beta Blocker

treat supraventricular tach
Class III Drugs: K Channel Blocker MOA
prolonging the duration of repolarization, (the Refractory Period)
Class III Drugs: K Channel Blocker indications
Indicated in V-fib, or V-tach.
Aminodarone (cordarone)
Class III Drugs: K Channel Blocker antiarrhytmic

inhibits Alpha and beta
reduces contractile velocity, increases the refactory period.

used for v fib,v tach, & atrial fib
bretylium (bretylol)
Class III Drugs: K Channel Blocker antiarrhytmic

prevent and treat ventricular fibrillation.

Also inhibits norepinephrine release, causing a sympathetic blockade
sotalol (betaspace)
Class III Drugs: K Channel Blocker antiarrhytmic

slows HR,decreases AV conductions,increases refractory period

Treat A fib
Class IV Drugs: Ca Channel Blocker MOA
prevents influx of Calcium in pacemaker cells & in myocardial cells.lowers the automaticity of atrial tissue and pacemaker cells and by slows conduction velocity through the AV node.
Verapamil (Calan)
Class IV Drugs: Ca Channel Blocker

used for supraventricular tachycardia also used for hypertension and angina
Digoxin MOA:
inhibits Na/K exchange in the heart which leads to increased Na Ca in the cell which leads to increased heart contraction.
Calcium Chloride
Other Anti-Arrhythmics

provides calcium necessary for cardiac contractility. Used to reverse electromechanical dissociation & increase cardiac output.
Other Anti-Arrhythmics

slows AV conduction, The treatment for Paroxysmal supra V-tach.
Digoxin as an Antiarrhythmic
Other Anti-Arrhythmics

Digitalis works by inhibiting Na/K exchange which increases intracellular calcium. The increased intracellular Ca gives a positive inotropic effect. It also has a vagal effect on the parasympathetic nervous system, used: to slow the ventricular rate during atrial fib. The dependence on the vagal effect means it is not effective when a pt has a high sympathetic nervous system drive, which is the case with acutely ill persons, and also during exercise.
what is proarrhythmia???
arrhythmia caused by drugs
t or f
arrhythmias can be caused by antiarrhythmic drug toxicity