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41 Cards in this Set

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alcoholic liver disease
disease resulting form excessive alcohol ingestion, characterized by fatty liver, hepatitis, cirrhosis
aromatic amino acids
aa phenylalanine, tryptophan, tyrosine
ascites
accumulation of fluid, serum protein, electrolytes within the peritoneal cavity caused by increased pressure from portal hypertension and decreased production of albumin
bile
thick, viscous fluid secreted from liver, stored in the gallbladder, and released into the duodenum when fatty foods enter the duodenum; emulsified fats in intestine and forms cmpds w FA to facilitate their absorption
BCAA
a valine, isoleucine, leucine
alcoholic liver disease
disease resulting form excessive alcohol ingestion, characterized by fatty liver, hepatitis, cirrhosis
aromatic amino acids
aa phenylalanine, tryptophan, tyrosine
ascites
accumulation of fluid, serum protein, electrolytes within the peritoneal cavity caused by increased pressure from portal hypertension and decreased production of albumin
bile
thick, viscous fluid secreted from liver, stored in the gallbladder, and released into the duodenum when fatty foods enter the duodenum; emulsified fats in intestine and forms cmpds w FA to facilitate their absorption
BCAA
aa valine, isoleucine, leucine
cholangitis
inflammation in bile ducts; may be acute or sclerosing
cholecystectomy
removal of gall bladder
cholecystitis
inflammation of gall bladder
choledocholithiasis
presence of gallstones in common bile duct
cholelithiasis
presence or formation of gallstones
cholestasis
suppression of biliary flow
cirrhosis
chronic liver disease caused by diffuse necrosis and regeneration, leading to an increase in fibrous tissue formation disrupting the normal liver structure
fasting hypoglycemia
low blood glucose caused by decreased availability of glucose from glycogen as result of depressed liver function
fatty liver
condition (hepatic steatosis) characterized by the accumulation of excess fat in liver commonly caused by alcohol abuse but also associated with obesity, starvation, intestinal bypass, parenteral alimentation, insulin resistance
fulminant liver disease
absence of preexhisting liver disease and development of liver disease with hepatic encephalopthy within 2 mos of onset of illness
hepatic encephalopathy
clinical syndrome characterized by impaired mentation, neuromuscular disturbances, and altered consciousness; four stages for progression
hepatic failure
conditionin which liver fxn is dimished to 25% or less
hepatic osteodystrophy
complication of chronic liver disease in which bone mass declines
hepatic steatosis
fatty liver
hepatitis
widespread inflammation of liver; usually viral in origin
hepatorenal syndrome
functional renal failure w/o anatomic or histopathologic renal changes; assoc w cirrhosis and ascites or with obstructive jaundice
jaundice
syndrome characterized by hyperbilirubinemia and deposition of bile pigment, resulting in yellowing of skin, mucous membranes and sclera
kayser-fleischer ring
greenish yellow pigmented ring encircling the cornea just within the corneoscleral margin; formed by copper deposits in Descement's membrane of the cornea; occurs in patients with Wilson's disease
kuipffer cells
fixed phagocytes in sinusoids of liver
nonalcoholic steatohepatitis
intermediate stage in fatty liver disease characterized by accumulation of fat droplets in the hepatocytes and presence of fibrous tissue and acute and chronic inflammatory cells
pancreatoduodenectomy
(whipple procedure)
excision of head of pancreas along with encircling loop of duodenum; may include partial gastrectomy
pancreatitis
inflammation of pancreas caused by autodigestion of pancreatic tissue by its own enzymes
paracentesis
procedure during which fluid from the abdomen is removed through needle
portal hypertension
normally increased blood pressure in portal venous system due to the obstruction of blood flow thorugh liver
portal systemic encephalopathy
hepatic encephalopathy
primary biliary cirrhosis
immune mediated chronic cirrhosis of liver caused by obstruction or infection of small and intermediate-size intrahepatic bile ducts; the extrahepatic biliary tree and larger intrahepatic ducts are normal; 90% of patients are women
secondary biliary cirrhosis
liver damage that results from bile backup as a result of gall bladder disease
steatorrhea
presence of excess fat in stool
varicies
low pressure veins that become distended from increased pressure; most commonly developed in the lower esophagus and upper stomach
Wernickle's encephalopathy
conditiion of damage to the CNS from thiamin deficiency; common in alcoholism
Wilson's Disease
autosomal-recessive disorder of copper metabolism in which excessive accumulation of copper occurs in liver, CNS and kidney