Mnt Final Htn Medications

Front 1

Diuretics

Back 1

promote excretion of salt and water through the kidneys to decrease volume in bloodstream

Front 2

B blockers

Back 2

decrease the vigor of hearts contractions by blocking catecholamines

Front 3

Aldosterone receptor blocker

Back 3

prevents the retention of Na and H2O

Front 4

ACE inhibitor

Back 4

inhibit conversion of Angiotensin 1to all....
& less Na/H20 is reabsorbed which prevents constriction of arteries

Front 5

Angiotensin II antagonist

Back 5

blocks the action of all

Front 6

Renin Inhibitor

Back 6

Inhibits renin production thereby reducing AI and all production

Front 7

Ca-channel blockers

Back 7

Blocks Ca from myocardial muscle which decreases contractility and dilates arteries

Front 8

Centrally Acting (antiandrenergics)

Back 8

lower heart rate (IV during crisis)

Front 9

Direct vasodilators

Back 9

work right away
nitroglycerine

Front 10

3 new DM drugs

Back 10

Byette
Januvia
Symlin

Front 11

sulfonylureas
meglitinides

Back 11

stimulate insulin secretion by pancreas

Front 12

biguanides

Back 12

inhibit hepatic glucose production

Front 13

thiazides

Back 13

increase insulin sensitivity

Front 14

alpha-glucosidase inhibitors

Back 14

delay glucose starch absorption

Front 15

DM fasting
normal
pre-dm
dm

Back 15

<100
100-125
≥126

Front 16

OGTT 2º

Back 16

<140
140-199
>200

Front 17

chylomicrons

Back 17

exogeneous TG transport

Front 18

VLDL

Back 18

endogenous TG transport

Front 19

IDL

Back 19

precursor to LDL

Front 20

LDL

Back 20

cholesterol transport

Front 21

HDL

Back 21

reverse transport

Front 22

TG optimal #

Back 22

<150

Front 23

total cholesterol

Back 23

<200

Front 24

HDL optimal #

Back 24

≥60

Front 25

LDL optimal #

Back 25

<100

Front 26

HTN normal

Back 26

<120, <80

Front 27

pre HTN

Back 27

120-139
80-89

Front 28

stage 1 HTN

Back 28

140-159
90-99

Front 29

HTN stage 2

Back 29

≥160
≥100

Front 30

3 compensatory mechanisms

Back 30

SNS
Renin angiotensin system
kidneys

Front 31

nicotenic acid

Back 31

affects the production of blood fats in the liver

Front 32

bile acid sequestrans
(questran - interact w fat sol vit)

Back 32

promote increased disposal of cholesterol in the intestines

Front 33

statins
(lipitor)

Back 33

iinterupt the formation of cholesterol from the circulating blood

Front 34

fibrates

Back 34

lower TG and increase HDL levels

Front 35

byetta

Back 35

stimulates insulin production, inhibit glucagon. lower post and fasting BG w weight loss side effect

Front 36

januvia

Back 36

destroys glucagon

Front 37

Symlin

Back 37

maintain normal blood glucose levels

Front 38

sibutramine

Back 38

appetite suppressant. causes tachycardia and high BP

Front 39

orlistat

Back 39

inhibits pancreatic lipase which causes reduced absorption of fat. (side effects: fatty stools, fecal urgency, malabsorption of fat soluble vits)

Front 40

orlistat

Back 40

inhibits pancreatic lipase which causes reduced absorption of fat. (side effects: fatty stools, fecal urgency, malabsorption of fat soluble vits)

Front 41

orlistat

Back 41

inhibits pancreatic lipase which causes reduced absorption of fat. (side effects: fatty stools, fecal urgency, malabsorption of fat soluble vits)

Front 42

rapid acting
humalog & novolog

Back 42

starts acting with in 15 min. peak at 30 min

Front 43

short acting

Back 43

starts acting with in 30 min. peaks at 2 hrs

Front 44

intermediate acting

Back 44

starts with in 1-3 hrs. Peaks at 18 hrs

Front 45

long acting
(Lantus)

Back 45

starts with in 2-4 hrs. steady effects, lasts 24hrs