• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/103

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

103 Cards in this Set

  • Front
  • Back

Neuroscience

The study of the nervous system and its role in behavior

Monism

The mind and brain are the same thing

Dualism

The mind and the brain are two separate things

Golgi Stain

Randomly stains about 5% of neurons allowing them to jump out from other neurons

Myelin Stain

Taken up by the fatty myelin on the axons to identify neural pathways

Nissl Stain

Opposite of the myelin stain. identifies the cell bodies of neurons.

Tracer

Trace pathways to determine origin or destination

Autoradiography

make active neurons stand out. can be correlated with behavior

Immunochemistry

Identifies receptors, neurotransmitters, or enzymes. Uses fluorescent dye and custom designed antibodies to target particular cellular components

In Situ Hybridization

Constructs complementary strand of radioactive DNA to pair with active mRNA to make it light up

CT Scan

Produces a series of 2D Xrays from different angles that computers combine to make a 3D image. Skull fractures.

MRI

Measures the radio frequency waves emitted by the nucleus of hydrogen atoms when they are subjected to strong magnetic field

fMRI

measures the brain activation by detecting the increase in oxygen levels in active neural structures

PET Scan

injecting radioactive dye into the blood stream

Neurons

10% of the brain. 100 billion in number. Communication cells

Glial cells

Glue that holds the neurons together. make up the myelin sheath which protects axon and makes communication faster. more populous than neurons

Dendrites

receive information at receptor sites from presynaptic cell

Cell Membrane

Maintains cell/ controls cell environment. important in communication because electrical gradient signals travel around it. made up of lipid proteins

Cell body

Produces DNA. Cell body stuff

Axon Hillock

Location where neuron determines if it will fire or not. connects cell body/membrane to axon

Axon

transports message length of neuron from one cell to the next

Myelin Sheath

Protects axon and allows message to travel faster. Prevents cross communication

Nodes of Ranvier

Allows message to maintain full strength by allowing an action potential to occur

Terminals

release NT into synapse

Vesicles

Hold NT in presynaptic cell

Receptor Sites

Where NT binds on dendrites of cell

Motor neurons

Multipolar. Muscles, organs and other neurons. responsible for movement

Sensory Neurons

Unipolar or Bipolar. From senses to central nervous system

Interneurons

Multipolar. between neurons in the same area. most numerous.

All-or-none Law

Neuron fires completely or not at all.

Rate Law

neurons fire faster in response to stronger stimuli

IPSP

Inhibitory Post Synaptic Potential

EPSP

Excitatory Post Synaptic Potential

Reuptake

Neurotransmitters back into presynaptic cell into vesicles to be stored

Deactivation

Enzymes tear apart specific NT's so they cannot bind on the postsynaptic cell

Autoreceptors

Outside of synapse on the presynaptic cell and tell the cell to stop releasing NT's into the synapse

Glial Cells (Synaptic Modulation)

Take neurotransmitters from synapse so they cannot bind to postsynaptic cell

Gyri

bumps on the brain

Sulci

indentations on the brain

Fissures

big Sulci

Convoluted

allows brain to store more information in a small area

Grey matter

Cell bodies

White matter

Cell axons

Meninges layers

Top: Dura Mater


Middle: arachnoid


Inner: Pia mater

Frontal Lobe

planning, organization, impulse control, emotion, behavior, primary motor cortex. humans only animal with large PFC--> inhibit inappropriate behaviors

Parietal Lobe

body sensations, primary somatosensory cortex. processes skin senses and position and movement

Occipital Lobe

Vision

Temporal Lobe

Hearing. Advanced visual processing

Thalamus

Central Relay. directs information

Hypothalamus

Four F's : Fight, flight, feeding and fornicating. Life sustaining behaviors

Pituitary gland

Hormones

Pineal Gland

Melatonin. sleep/wake cycles

Corpus Callosum

bridge between hemispheres that allows cross communication

Superior colliculi

vision

Inferior colliculi

hearing

substantia nigra

involved in movement

ventral tegmental area

reward.

medulla

heart rate and respiration

pons

sleep paralysis, arousal, REM dreams

Cerebellum

balance, motor movement

ventricles

contain cerebralspinal fluid. nutrient/waste control of the brain

amygdala

emotions

hippocampus

memory consolidation

drug

a non-required substance that changes body function

psychoactive drug

drug that has psychological effects

agonist

mimics or enhances NT

antagonist

blocks or decreases NT

addiction

preoccupation with obtaining/using drug

withdrawal

Negative reactions. opposite effects

tolerance

decreased response with exposure to drug

dispositional tolerance

an increase in drug metabolism. drug won't reach the same level or last as long

functional tolerance

central nervous system becomes less sensitive to noticeable drug effects. "New Normal"

Behavioral (Learned) Tolerance

learn ways to compensate for for effects. (ABC's backwards when drunk)

Cross tolerance

extension of tolerance to similar drugs. work at same NT so they have to built up tolerance. (Addiction to Xanax = tolerance to alcohol)

Mixed tolerance

tolerance to only some of the drugs effects (May not slur words but stumble when walking)

Reverse Tolerance

Sensitization --> doesn't take as much to be sensitive again (THC binds to fat cells and stays in body so don't have to smoke as much to get high again)

Sensory Memory

Milliseconds to seconds. High capacity. Not consciously aware. Loss through decay

Short-term memory

seconds to minutes. limited capacity to 7 +- 2 items. consciously aware. loss through decay.

long-term memory

days to years. high capacity. both consciously and unconsciously aware. lost through interference and decay

modal model of memory

memory is gained in discrese stages with different characteristics


sensory inputs -> sensory register+attention -> short-term memory + rehearsal -> long-term memory

Working memory model

short term and long term memory work together in consolidation to manipulate information. Visuospatial sketch pad and phonological loop are coordinated by central executive to manipulate information

Declarative Memory

can be explained in words

Episodic Memory

Events

Semantic

Facts

Nondeclarative Memory

Hard to explain in words. more behaviors

Procedural Memory

skills. motor and cognitive

Perceptual Representation System Memory

Exposure to stimuli activates a network of information. Priming

Classical conditioning Memory

Conditioned response between two stimuli

Nonassociative Memory

Habituation: decreased response to a stimulus over time


Sensitization: increased attention to a stimulus over time

Retrograde Amnesia

Loss of long term memory. cannot remember past

Anterograde amnesia

Loss of short term memory. cannot form new memories

CA1 Region of Hippocampus

80% of memory success. consolidation

Learning

a change in behavior, or the potential for that change, that indicates a permanent knowledge

neuroplasticity

The ability of the brain to change and form new/stronger connections

LTP

strengthening of long term memory: cells that fire together wire together

LTD

weakening in strength of connections of neurons no longer activate each other. clears hippocampus for new stuff

ALTP

Two stimuli occur at the same time. One elicits strong response. second elicits weaker response

ALTD

Loss of second weaker stimulus eliciting response because of loss of the first stimulus so through association, the second is no longer paired with the first so no response is elicited

How Depression Treatments work

increase receptor sensitivity and number. increase neural plasticity. trigger release of BDNF which is necessary for neurogenesis. increase cell death -> out with the old, in with the new.

factors related to surfacing of Schizophrenia

divorce, seeing something traumatic, poor nutrition, various birthing factors, genetics

MAOI's

non nicotine cigarette ingredients

Triclyclics

block reuptake

Second generation antidepressants

effect a single NT