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14 Cards in this Set
- Front
- Back
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Chlamydia pneumoniae may be implicated in |
Arteriosclerosis |
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Lymphatics seep through capillaries to become |
interstitial fluidsurrounding tissue cells, which it bathes, feeds |
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Acute and subacute bacterial endocarditis differ in |
their suddenness and severity of onset |
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Subacute Bacterial Endocarditis (SBE) Causative agent |
Usually normal flora of mouth or skin such as viridansstreptococci and Staphylococcus epidermidis |
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Subacute Bacterial Endocarditis (SBE) Pathogenesis |
Bacteria enter blood during dental procedures, tooth brushing, or trauma – Previously damaged heart valve immune complexes that may lodge in skin, eyes, otherstructures; trigger inflammatory response – Glomerulonephritis in kidneys |
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tumor necrosis factor cytokine released frommacrophages |
seems to play role in endotoxic shock |
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Tularemia (Rabbit Fever) signs and symptoms |
Regional lymph nodes enlarge fever, chills, aches Usually clear in 1–4 weeks, sometimes last months |
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Plague (Black Death) Signs and Symptoms |
Following flea bite, symptoms appear within 2–6 days Enlarged and tender lymph nodes called buboes (bubonic plague) High fever, shock, delirium, patchy bleeding under skin |
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Yersinia pestis |
like other enterobacteria, facultativelyanaerobic, Gram-negative rod |
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Plague (Black Death) Pathogenisis |
Transmitted from flea bites Major factors are carried on 3 separate plasmids |
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Plague (Black Death) – Endotoxins cause |
septicemic plague Gram-negative – fatal septicemias |
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Infectious Mononucleosis (Mono or KissingDisease) Causative Agent |
Epstein-Barr virus (EBV) |
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Yellow Fever Causative Agent |
Transmitted by bite of infected Aedes aegypti mosquitoes |
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Malaria Causative Agent |
Protozoa of genus Plasmodium |
