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50 Cards in this Set

  • Front
  • Back
what compartment does ringers go to?
intravascular (plasma), which is 1/12 of TBW
when can you not use ringers
when hyperkalemia is a concern (it contains potassium)
how to calculate maintenance fluids
100 ml/kg for first 10 kg
50 for 2nd 10
20 for each other 1 kg
tx for hypo, iso and hyper volemic hypernatremia
hypo: isotonic NaCl, and then replace free water deficit

iso: vasopressin plus oral fluids or D5W

hyper: furossemide
formula for ionied Ca
total Ca - (serum Albumin x 0.8)
effect of PTH
increase Ca
decrease PO4
effect of calcitonin
decrease Ca and PO4
effect of vitamin D
increased Ca and PO4
numbness, tingling
hypoCa
effect of hypomagnesemia
decreased PTH
Chvostek's
decreased Ca
TPH in pseudohypoPTH
very high
tx for hypocalcemia
IV calcium gluconate, and replace
tx for PTH deficiency
vitamin D
thiazides to lower urinary Ca and prevent stones
Calcium in MM
increased due to lysis and release of osteoclast activating facot
mechanism for hypercalcemia in sarcoid
increased renal reabsorption
increased urinary cAMP
primary hyperparathyroidism
tx for hyper Ca due to vit D abnormalities, MM
glucocorticoids
tx for hyper calcemia
IV fluids
diuretics
effect of alkalosis on K
alKaLOsis (low K+)
chronic volume depletion secondary to an autosomal-recessive defect in salt reabsorption in TAL
Bartters
EKG effects of hypokalemia
T wave flattening/inversion
U wave
effect of rapid administration of beta blocker on K levels
hyper K
level of K for EKG changes
?6.0
effects of hyper K on EKG
peak T
prolonged PR
wide QRS
Vfib, leading to MI
tx for severe hyperK
- IV Ca (careful of digoxin)
- glucose, insulin and Na bicarb to send K into cells
- kayexalate
effect of low Mg
makes low Ca hard to treat
relationship between Mg and K
they go down together

M and Kecilia
increased Mg
usually renal failure
effect of Mg on EKG
decreased Mg prolongs QT
flattens T
torsades
anti Mg tx
calciumgluconate
anti K tx
IV calcium
effect of hyperphosphatemia
soft tissue calcifications when [Ca]x[Ph]>70
soft tissue calcifications
hyperphosphatemia
effect of acidosis on CNS
depresses
effect of alkalosis on CNS
decreases blood flow
nl anion gap
8-15
kussmaul breathing
metabolic acidosis
winter's formula
expected PaCo2 = 1.5[HCO2]+8 +-2

this tells us the expected respiratory compensation to metabolic acidosis
causes of proximal tubular acidosis
MM
cystinosis
Wilson's disease

(all feature decreased bicarb reabsorb)
causes of distal tubular acidosis
SLE
Sjogrens
Amphotericin B

(can't make bicarb)
causes of normal AG acidosis
renal or Gi losses of bicarb
- PTA
- DTA
- Acetazolamide
- Diarrhea
- Pancreatic fistulas
- small bowel fistulas
- ureterosigmoidostomy
causes of metabolic alkalosis
with ECF contraction:
- vomit, NG suction, diuretics, villous adenoma

with ECF expansion, HTN
- hyperaldo, Cushings, Severe hypoK, Barrters, Diuretic abuse
tx for volume contraction alkalosis
Na and K
tx for volume expansion alkalosis
spironolactone
how many days for renal compensation in respiratory acidosis
5 days
headache
confusion
papilledema
acute CO2 retention
in acute respiratory acidosis how much does bicarb increase with PaCO2
1 for every 10
in chronic respiratory acidosis how much does bicarb increase with PaCO2
4 for every ten
in acute respiratory alkalosis how much does bicarb decrease with decrease in PaCO2?

in chronic?
2 for every 10
5-6 for every 10 in chronic