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166 Cards in this Set

  • Front
  • Back
elevated homocysteine
stable angina
Q waves
prior MI
ST segment depression
subendocardial ischemia
wall motion abnormalities on stress echo
exercise induced ischemia
rx for pharmacologic stress test
adenosine or dipyridamole (which vasodilate coronaries, and therefore makes little difference in diseased ones)
or
dobutamine (which increases O2 demand by increasing HR, BP, contractility)
ACS means
atherosclerotic plaque rupture and coronary occlusion
beware of stress testing a patient with...
unstable angina
how to differentiate USA and non-STEMI
cardiac enzymes
ESSENCE trial
Enoxaparin led to better outcomes than heparan in USA and non-STEMI
tx for unstable angina
aspirin
beta blocker
LMWH heparin (enoxaparin)
nitrates
GPIIb/IIIa inibitors as adjunct
TIMI score
age >=65
more than three CAD risk factors
known CAD (stenosis >50%)
2 episodes of severen angina in past 24 hrs
apsirin use in past 7 days
elevated cardiac enzymes
ST changes >= .5mm

Risk is about 4-5 per point
CARE trial
statins
transient ST elevations during exertion
Prinzmetal
substernal chest pain > 30 minutes
MI
inferior ECG changes
hypotension
elevated JVP
hepatomegaly
clear lungs
RV infarct
Peaked Ts
early ischemia
ST elevation
transmural injury
ST is TS
elevated troponins, no heart issues
renal failure
CAPRICORN trial
beta blocker carvedilol reduces risk of death in patients with post-MI LV dysfunction
ST elevations V1-V4
anterior MI
Q waves in V1-V4
Anterior MI
R wave in V1 V2
ST depression in V1V2
T elevation in V1V2
Posterior

P is RST for Vs
Q waves in I, aVL
Lateral MI
Q waves in II, III, aVF
Inferior MI
HOPE trial
ACEi, ramipril, reduces mortality in MI, stroke and renal disease in high-risk CAD pts
GUSTO trial
tPA and Heparin is the best thrombolytic regime
tx for VT
IV amiodarone
rx to correct severe/symptomatic bradycardia
atropine
tx for 1st and 2nd degree (type I) AV block
none
what is CK useful for
an early subsequent MI (because they are shorter duration)
incomplete free wall rupture...

what is it called?
what is tx?
ventricular pseudoaneurysm
surgery
dyspnea
orthopnea
PND
nocturnal cough
confusion and memory impairment
diaphoresis and cool extremities at rest
Left sided HF
S3
ventricular gallop
left sided heart failure
S3 follows S2
tx for mild CHF
ACEi
loop if volume overload
tx for moderate CHF
ACE, loop
add beta blocker
tx for sever CHF
loop, ACE
add digoxin
add spironolactone if needed
where to hear S3
apex
where to hear S4
base
peripheral pitting edema
nocturia
JVD
hepatomegaly/mepatojugular reflex
ascites
RV heave
Right sided heart failure
RV heave
right sided heart failure
BNP >100
CHF vs. COPD
Kerley B lines
on CXR
pulmonary congestion secondary to dilatation of pulmonary lymphatic vessels
RALES trial
spiRonolactone Reduces Rigor moRtis in patients with class thRee or fouR heaRt failuRe
When to use digitalis
EF <30%, but doesn't reduce mortality
nausea/vomiting, anorexia
ectopic ventricular beats
AV block
A Fib
visual disturbances, disorientation
digoxin toxicity
wide QRS
PVCs
PVCs increase risk for?
A Fib
what cardiac trouble can be caused by hyper/hypo thyroidism
A Fib
what cardiac trouble can be caused by excessive alcohol drinking (holiday heart)
A fib
sequela of A fib
embolic stroke
preferred tx for rate control for acute A Fib in a stable patient
Calcium blockers
B blocer alternative

vs either for chronic AFib
first step in A fib tx
rate control with calcium blocker
next step after rate control in A Fib
cardioversion (electrical > pharm)
INR range to prevent CVA
2-3
AFFIRM trial
better in A Fib tx to controle Rate
tx for chronic AFib
rate control with beta blocker or Ca channel blocker (vs just CCB in chronic A Fib)
atrial rate > 400
A fib
atrial rate of 250-350
A flutter
causes of A flutter
COPD
RHD, CAD, CHF
ASD
cardiac sequelae of COPD
Flutter
multifocal atrial tachycardia
multifocal atrial tachycardia tx
oxygenation and ventilation
what does multifocal tachycardia indicate
severe pulmonary disease
variable P waves and variable PR and RR intervals (at least 3 different)
multifocal atrial tachycardia
narrow QRS
P waves burried inside the QRS
PSVT, generally due to AV nodal reentrant tachycardia
causes of PSVT (narrow QRS, possibly with no visible Ps)
ischemia
digoxin toxicity (2:1 block)
AV node reentry
excessive caffeine or alcohol consumption
tx for PSVT
IV adenosine preferred
also can use IV verapamil, esmolol
PSVT prevention rx
digoxin (verapamil or bb as alternatives)
ablation of AV node or accessory tract
difference in tx of PSVT and WPW
avoid digoxin (because it acts on AV node) in WPW; just use ablation
difference in location of PSVT and WPW
PSVT is either in AV node or between A and V

WPW is only between the A and V
tachycardia
short PR
delta wave
WPW
tx for torsades
IV magnesium
acute Canon A wave
VT
wide QRS tachycardia
suspect VT
VT tx
first, ICD
second, amiodarone
no atrial P waves
no QRS identified
VFib
tx for pulseless electrical activity
AMIodarone (in our AMI with VFib)
P and Q waves unrelated
3rd degree AV block
p waves lengthen progressively until a beat dropped
2nd degree block type I
heart d/o causedby doxorubicin
dilated cardiomyopathy
heart d/o caused by thiamine deficiency
dilated cardiomyopathy
heart deficiency caused by Chagas disease
dilated cardiomyopathy
heart irregularity caused by pheochromocytoma
dilated cardiomyopathy
S3 and S4 murmurs
dilated cardiomyopathy
inheritance of HCM
AD
loud S4
HCM
intensity of a murmur increases with valsalva and standing, but decreases with handgrip
HCM
bisferious pulse (rapidly increasing carotid pulse with two upstrokes)
HCM
tx for HCM
beta blockers are first choice
also CCBs
diuretics for fluid retention
mymectomy
MV replacement
viral causes of pericarditis
echovirus
coxsackie
Dressler syndrome
Percarditis post MI (weeks to months)
drugs that cause lupus pericarditis
procainamide
hydralazine
chest pain with breathing
pericarditis
pericardial friction rub
pericarditis
scratching, high pitched, 3 component sound heard during expiration
pericardial rub
PR depression
pericarditis
tx for pericarditis
NSAIDS
when is ventricular filling impeded..
in constrictive pericarditis?
in tamponade?
early and late diastole
vs
all diastole
JVD fails to decrease during inspiration
Kussmaul's sign for constrictive pericarditis
how to image for pericardial effusion and tamponade
echo
enlarged heart without pulmonary vascular congestion
pericardial effusion
narrowed pulse pressure
pulsus paradoxus
cardiac tamponade
hypotension
muffled heart sounds
JVD
cardiac tamponade (Beck's triad)
loud S1
mitral stenosis
LA enlargmenet
MS
LV hypertrophy, leading to dilation, dysfunciton, MR
AS
LV enlargement and hypertrophy
AR
LA/LR dilatation
MR
crescendo decrescendo systolic murmur
AS
S4
AS
parvus et tardus
AS
precordial thrill
AS
widened pulse pressure
AR
holosystolic murmur at apex
MR
tx for MR
timely surgery
blowing holosystolic murmur at LLSB
TR
A fib usually present with this murmur
TR
midsystolic clicks
mid to late systolic murmur
MVP
click:
increased by valsalva and handgrip
decreased by squatting
MVP
migratory polyarthritis
erythema marginatum
chorea
subcutaneous nodules
RH disease
Acute endocarditis usually caused by
staph aureus

Acute Aureus
organisms for native valve endocarditis:
viridans
aureus, epidermidis
enterococci
HACEK (hemophilus, actinobacillus, cardioabacteium, Eikenella, Kingella)
endocarditis on both sides of aortic valve
Libman Sacks
thrombotic endocarditis
Marantic
associated with metastases
Duke's criteria
for endocarditis

major:
bacteremia sustained
endocardial involvement, esp by echo or new regurg

minor:
predisposing condition
fever
vascular phenomena
immune phenomna
positive blood cultures
positive echo
wide fixed splitting of S2
low rumble murmur
ASD
harsh blowing holosystolic murmur
VSD
heart defect associated with congenital rubella
PDA
machinery murmur
PDA
mid systolic murmur heard over back
coarctation of aorta
coarctation of aorta in short woman
turners
hypertensive emergency
systolic >200 and/or diastolic >120
AND
end organ damage
tx plan for HTN emergency
25% decrease in 1-2 hours and then gradually by IV nitroprusside, labetalol or NG
tx plan for HTN urgency
oral agents over 24 hours
widened mediastinum on CXR
aortic dissection
diagnostic tools for aortic dissection
CXR and TTE
tx for aortic dissection
IV beta blockers (to slow)
and
IV sodium nitroprusside to lower BP

type a proximal - surgery
medical for type b
pain in back or lower abdomen radiating to groin, buttocks, leg
Grey Turners Sign for AAA
echymoses around umbilicus
Cullen's sign for AAA
abdominal pain
HTN
palpable uplsatile abdominal mass
ruptured AAA

tx = emergent laparotomy
size indication for surgery on AAA
> 5cm
cramping leg pain reliably reproduced by same walking distance

relieved at rest, or by hanging leg over bed
PVD - chronic arterial insufficiency
systolic BP at ankle/arm
claudication is <0.7
resta pain <0.4
first treatment for claudication
stop smoking
diagnostic tool for PVD? Acute arterial occlusion?
both are by arteriogram
pallor
pain
pulselessness
paresthesias
paralysis
polar (cold)
Acute arterial occlusion
risk with arteriograms
cholesterol embolization
what should you not anticoagulate for?
cholesterol emboli!
how to treat syphyllitic aortitis/leutic heart
IV penicillin and surgical repair
endothelial injury
venous stasis
hypercoagulability
Virchow's triad for DVT
PTT target for DVT
1.5-2 x
(vs 2-3 for Afib)
brawny induration of leg
CVI (chronic venous insufficiency/venous stasis disease)
migratory superficial thrombophlebitis
malignancy, often of pancreas
throbmobplebitis at IV site on arm or with LE varicostiy
superficial thrombophlebitis
tx for thrombophlebitis
no tx unless painful

with pain and cellultis:
be rest, elevation, hot compresses
swelling
redness
indurated vein
superficial thrombophlebitis
cardiac output up means what kind of shock?
septic
PCWP up means what kind of shock?
cardiogenic
SVR up and PCWP down means
hypovolemic shock
CO down and SVR down means
neurogenic shock
severe peripheral vasodilatation
flushing
warm skin
septic shock
peripheral vasoconstriction
cool skin
hypovolemic shock
two or more of:
fever
hyperventilation
tachycardia
Increased WBCs
SIRS
SIRS plus positive blood cultures (2 sets from 2 different sites)
sepsis
hypotensin induced by sepsis
septic shock
peripheral vasodilation with decreased SVR
warm, well-perfused skin
bradycardia and hypotension
neurogenic shock
diastolic murmur that changes character with changing body positions
atrial myxoma