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79 Cards in this Set

  • Front
  • Back

Diuretic therapy can cause what

hypokalemia

What type of loop diuretic

Furosemide

What are some cardiac effects of K+ hypokalemia

increased risk of digoxin toxicity


ventricular dysrhythmias


flattening and inversions of T wave


presence of U wave


cardiac arrest

Hyperkalemia us usually related to

renal dysfunction


excess K+ supp

what are some cardiac effects of hyperkalemia

ventricular dysrhythmias


tall peaked T waves on ECG


asystole

In older adult


what happens to cardiac valves

calcification


mucoid degeneration


mainly mitral and aortic valve


In older adult


what happens with the conduction system

Pacemaker cells decrease in number


fibrous tissue and fat increase SA node


few muscle fibers remain in the atrial myocardium and bundle of his


conduction time increase

In older adult


what happens left ventricle

increases in size


stiff and less distensible


diastolic filling decrease by 50%

In older adult


what happens aorta and other Lg arteries

thicken


stiffen


less distensible


systolic BP increases to compensate for stiff arteries


systemic vascular resistance increases


hypertropy

In older adults


what happens to barorecptors

less sensitive


Nursing interventions


cardiac valves

assess HR


rhythm


Hears sounds


Question about dyspnea

Nursing interventions


conduction system

HR < 60


ECG for dysrhythmias


Nursing interventions


left ventricle

ECG for


widening QRS complex


longer QT interval

Nursing interventions


aorta and other Lg arteries

BP


Not increase in systolic, diastolic, and pulse pressure


activity intolerance


SOB


peripheral pulses

Nursing interventions


baroreceptors

BP standing, lying and sitting


dizziness? when change position


Teach to change position slowly

Rationales


cardiac valves what are you looking for

Murmurs


rhythm changes

Rationales


conduction system what are you looking for

SA node lose it inherent rhythm


Atrial dysrhythmias

80% of older adults experience what conduction system malfunction

PVC's premature ventricular contractions

Ventricular changes result in decreased what in the left ventrical

decreased


stroke volume


ejection fraction


CO during exercise


less able to meet increased O2 of heart

Rationales


what happens in the aorta and arteries

HTN

Rationales


Baroreceptors have to do with

orthostatic

What are some cardiac markers

troponin


creatine kinase


myoglobin

Is a myocardial muscle protein released into the bloodstream with injury to myocardial muscle

troponin

MI is also known as

acute coronary syndrome

is an enzyme specific to cells of the brain, myocardium, and skeletal muscle

creatin kinase CK

what does CK in the blood mean

tissue necrosis or injury

Earliest marker detected - maybe 2 hrs after MI

Myoglobin

Which cardiovascular assessment lab checks for CAD

Serum Lipids

an amino acid that is produced when proteins break down

homocysteine

What are some serum lipids that check for CAD

HDL


LDL


triglyceride


total cholesterol

Highly sensitive C-reaction protein hsCRP


Most studied marker of inflammation

PT

checks


anticoagulants


Warfarin


coumadin

PTT

Heparin

is inability of heart to pump adequate blood to meet metabolic needs of body

HF

What cause HF

myocardial damage from MI


incompetent valves


inflammatory cond. of heart


cardiomyopathy


pulmonary HTN (cor pulmonale)


What happens in the compensatory phase of HF

1. CO falls


a. sensed by baroreceptors


b. stimulate SNS


2. release of epinephrine and norepinephrine


3. increase in HR and vasoconstriction


4. increased in filing pressures


5. increase in SV and CO

Compensatory mechanisms increase cardiac metabolic demands and in time?

decrease cardiac function and ability to compensate

Where does Right sided HF back up to

venous circulation


body

where does left sided HF back up into

pulmonary circulation


lungs

What happens to the body in acute HF with overload in lungs

pulmonary edema


restlessness


anxiety


increased crackles


tachypnea


tachycardia


pink frothy sputum


decreased SO2 and PO2

Assessment


S/S of HF left

dyspnea on exertion (often 1st sign)


orthopnea


paroxysmal nocturnal dyspnea


new S3 (ventricular gallop)


crackles


pulmonary edema (acute life threat)


angina


weakness


Assessment


S/S of HF right

edema lower extremities


JVD


nausea (fluid congestion in abdominal organs)


abdominal discomfort


weight gain

Assessment


S/S of HF both sides

unexplained fatigue


decreased exercise tolerance


AMS

What are labs for HF

chest x ray


ECG


CVP elevated in right HF


PA pressure monitoring

Priority nursing for HF


what might happen

reduced CO


fluid overload


alteration in gas exchange


inability to tolerate activity


inadequate knowledge

Planning and implementation


HF acute phase

Monitor BP, pulse, respiration, ECG, and CVP to detect changes in the CO


Pt in sitting position to decrease pulmonary congestion


Lung auscultation


Give O2


Give meds


monitor serum electrolytes to detect hypokalemia


I&O



Planning and implementation


HF chronic phase

Get a baseline assessment for fluid staus


monitor daily weight


assess the fluid status - regular intervals

What kinds of Meds do you give HF

1. angiotensin converting enzyme ACE


2. ARB's angiotensin II receptor blockers


3. Diuretics ie loop diuretics (furosemide - Lasix)


4. Vasodilators


5. Morphine


6. Digoxin (lanoxine)


7. Dopamin

What do ACE do for HF?

reduce afterload


increase CO

ACE monitor for

hypotension


orthostatic (especially)


ARB's do for HF

interrupt vasoconstrictor and


aldosterone secreting effects of angiotensin II

Diuretics do for HF

decrease preload and


pulmonary congestion


What happens when you decrease pulmonary congestion

decreases cardiac work


increases CO

Vasodilators what do for HF


example of drug


monitor for

nitroglycerine


reduce preload


monitor hypotension

Morphine do for HF


monitor for?

sedate


vasodilate


decreases cardiac workload


monitor for hypotension and


respiratory depression

Digoxin (lanoxin) do for HF


what do you do before you give this drug

improve contractility


increase SV and CO


apical pulse for 1 min


slow conduction through AV node

What drug is used in critical care of HF

dopamine (Intorpin)


when decompensation of CO leads to hypotension


monitor BP and IV site frequently

Pt in acute HF should not be started on these drugs

Beta blockers

What beta blockers are approved for chronic HF

metoprolol


bisoprolol


carvedilol

Defects in cardiac valve structure of function that interfere with proper cardiac circulation

Valvular disorders

What are the 2 main valvular disorders

stenosis


regurgitation

heart valve leaflets are fused tighter, opening is narrow, stuff, and unable to open or close properly

stenosis

incomplete closure of the heart valve, resulting in backflow of blood

regurgitation

Priority nursing problems


what can go wrong

reduced cardiac output


potential for thrombus formation or infection


inadequate knowledge


can't do exercise or activities

What are some planning and implementation for valvular disorders

monitor heart sounds


signs of decreased CO, restrict activity


monitor for signs of endocarditits


Medication therapy


Valvular disorders


What are some things you might have to treat

Antibiotic for endocarditis


Antidysrhythmic and anticoagulant for A-fib

What do you need to monitor lab wise


Valvular disorders

PT/INR


Why - warfarin

What Vit do you want to maintain with Valvular disorders

Vit K


green leafy vegetables

What are the expected outcomes for Valvular disorders

no signs of endocarditis


describes s/s to report to MD


optimal level of function

inflammation of inner layer of heart, usually involves cardiac valves

endocarditis

Caused by staphylococcus aureus

acute endocarditis

Caused by streptococcus viridans

subacute endocarditis

Acute endocarditis s/s

high fever with chills


signs of HF


WBC elevation

Subacute endocarditis s/s

High fever unknown origin


cough


dyspnea


anorexia


malaise


normal WBC


anemia


elevated ESR erythrocyte sedimentation

Priority nursing


what can go wrong be prepared

potential for injury related to thrombus formation


inadequate knowledge related to prevention of repeated infection

Planning and implementation


what do you need to do as a nurse

s/s of infection


anti embolism stockings


What symptoms do endocarditis need to report to MD

fever


anorexia


malaise

What is the most common complication of infective endocarditis

HF

g

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