• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/31

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

31 Cards in this Set

  • Front
  • Back

35-45

PaCO2

21-28

HCO3

80-100

PaO2

7.35-7.45

pH

Normally associated with acidosis

Hyperkalemia

Breathing

controls the amount of free hydrogen ions by controlling the amount of CO2 in arterial blood

CO2 rises above normal


triggers the neurons to increase the rate and depth of breathing. (Respiratory alkalosis)

Hyperventilation

causes respiratory depression, inadequate chest expansion, airway obstruction, and reduced alveolar capillary diffusion

Respiratory acidosis

What causes Kussmaul respiration

Metabolic acidosis

Breathing is reduced when?

Respiratory acidosis

Hydration


Drugs or other treatments to control the problem

Interventions for Metabolic Acidosis

Improver ventilation


oxygenation


maintain patent airway (drug and oxygen therapy)

Respiratory acidosis interventions

Causes Hypocalcemia/Hypokalemia

Alkalosis

Prevent further loss of hydrogen, potassium, calcium and chloride ions, to restore fluid balance and to monitor changes

Alkalosis Intervention

Metabolic alkalosis R/T


bicarbonate therapy


diuretic use


vomiting


nasogastric suction


Metabolic acidosis treatment

Sodium bicarbonate

Inserted in veins of the upper arm (sterile technique)


Therapies lasting 1-4 weeks


Indications: Fluids for hydration and drug therapy


pH of fluids should be 5-9

Midline Catheters

-Inserted antecubital fossa (inner aspect of the bend of arm) or midline of upper arm


-Insert early in course of therapy


-Basilic vein preferred


-Common complications: phlebitis, thrombophlebitis, DVT


-Accommodates all types of infusion therapies, blood sampling

PICC line

-allows access to rich vascular network in the red marrow of bones


-used previously for only peds patients


-Trauma, burns, cardiac arrest, diabetic ketoacidosis

Intraosseous Infusion Therapy

increased tissue pressure in confined anatomic space causes decreased perfusion

Compartment Syndrome

Noninflammatory degenerative joint disease


-localized


-Progressive loss of cartilage


-joint pain


Osteophytes (Bone spurs)

Osteoarthritis

-Joint pain/stiffness


-Crepitus


-Herberden's nodes


-Bouchard's nodes


-Joint effusion(excessive joint fluid )


-Atrophy of skeletal muscles

Osteoarthritis clinical manifestations

Integrative therapy for OA


reduces inflammation

Glucosamine

inflammatory systemic disease


-Destructive to joints


-Chronic, progressive and autoimmune

RA

-Associated with advanced RA


-Dry mouth (xerostomia)


-Dry Eyes


-Dry Vagina (Sometimes)

Sjögren's Syndrome

ESR


Serum complement (C3/C4)


Serum protein


Immunoglobulins

RA Labs

-Autoimmune disorder, chronic, progressive inflammatory


-Causes major body organs and systems to fail


-Characterized by spontaneous remissions and exacerbations (flare ups)

Systemic lupus erythematosus (SLE)

Dry, scaly "butterfly" rash on the face


-Discoid lesions


-Alopecia


-Polyartyhritis


-Joint changes

SLE Clinical Manifestations

Urate crystals deposit in joints and other body tissues, causing inflammation


-Tophi

Gout

Drug therapy


Nutrition therapy--> Limit proteins, foods that causes flare ups



Gout Treatment

-Hip dislocation


-VTE


-Infection


-Anemia


- Neurovascular compromise

Hip Replacement Complications