Md2 Block4 Ns Test Review Alternate

Front 1

1. When light is presented to the right eye, both pupils constrict. When light goes to left eye, left pupil paradoxically dilates. Where is the lesion

Back 1

Optic nerve lesion

Front 2

2. What can u expect in the visial field

Back 2

extension of the blind spot, central scotoma (area in visual field where you can see anything),

Front 3

3. There is aneurysm detected at the junction of the anterior cerebral artery and the anterior communicating artery. What kind of visual field issue can you detect here

Back 3

Suppresion of optic chiasm from the upper part. Bitemporal Hemianopsia, inferior Quandrantanopsia

Front 4

4. Supratentorial tumor compressing the optic chiasm

Back 4

Bitemporal inferior quadrantanopsia

Front 5

5. Pt with homonymous hemianopsia with no macular sparing. Where is the lesion

Back 5

Optic radiation

Front 6

6. There is a light on the left eye and the right eye has no reaction, but left eye has reaction. Then the light on the right eye, and it does not react, but left eye constricts. Where is the lesion

Back 6

Lesion of right Occulomotor nerve

Front 7

7. What else you might expect in this patient

Back 7

Inability to do adduct the right eye during convergence

Front 8

8. The lateral half of the right optic nerve rear to the entrance to the brain is damaged. What can you expect in visual field

Back 8

Nasal visual field of right eye

Front 9

9.8. The lateral half of the right optic nerve rear to the entrance to the brain is damaged. What can you expect in visual field?

Back 9

Ganglion cells make action potentials in eye

Front 10

10. The left optic tract proximal to LGN is damaged. What kind of visual field defect

Back 10

Right side visual defect- Right homonymous hemianopsia

Front 11

11. Lesion in the left temporal lobe What causes this =
Aka=

Back 11

(Meyer’s loop).
Right Upper Quadrant Anopia

Front 12

12. What is the problem with parkinsons disease

Back 12

Rhythmic tremor, mask-like expression less face, they walk like an old man- the lesion of substantia nigra and projects to corpus striatum Basal Ganglia. Difficulty to start movement.

Front 13

13. Young man complains of impotence and has bitemporal hemianopsia. On MRI, there is supracellar mass. High prolactin level. Which structure is involved in that

Back 13

Hypohyseal Portal System
above chiasm

Front 14

14. Which structure is destroyed in order to help the patient with parkinsons disease

Back 14

Medial Globus Pallidus

Front 15

15. Huntingtons disease- Which neurotransmitters

Back 15

GABA and AcH. Occurs in Caudate nucleus putamen

Front 16

16. Kluver Busci Syndrome- not aware of dangers, blind for the dangers. When does this syndrome appear
What causes this

Back 16

- Hippocampus and bilateral lesion of Amygdala.
-Traumatic or infection.
-This presents with = Hyperphagia and hyperorality, hypersexuality

Front 17

17. lesion of Hippocampus when you have lesion of temporal lobe of brain. How will it affect you

Back 17

Hippocampus is for storage of long term memory. Hippocampus is important for learning.

Front 18

18. The hypothalamus is in connection to the hippocampus =

Back 18

thru the septal area

Front 19

19. When do you have hemiballismus

Back 19

Lesion of subthalamic nucleus

Front 20

20. In schizophrenia, there is a change in

Back 20

Dopamine

Front 21

21. Stroke caused by occlusion of right anterior cerebral artery.

Back 21

You will lose pain and temperature sensation of left leg.

Front 22

22. After head trauma, pt loses interest in family and work. Which part of brain is effected by this trauma

Back 22

Frontal Lobe

Front 23

23. Pt had a stroke and is unable to move right arm and leg. Speech was too slurred to comprehend and then they thought the patient has a stroke. Speech was fluent and grammatically correct. The lower 2/3 of the right face is drooping. Tongue points to right side. Her right arm and leg were weak. Where in the CNS is the stroke

Back 23

Left internal capsule or basilar pons

Front 24

25. Lesion of internal capsule of posterior limb on the right. What can you expect

Back 24

Altered sensation of left side of face

Front 25

26. Damage to the CST and CBT can cause

Back 25

weakness on opposite side of lesion

Front 26

27. Damage to 2 tracks cause weakness of right side

Back 26

.CST and CBT

Front 27

28. Her forehead is spared because

Back 27

the upper face is spared due to bilateral innervations to superior facial nucleus

Front 28

29. How can her speech deficit be classified

Back 28

Dysarthria

Front 29

30. Which artery is involved in stroke in this woman

Back 29

Middle Cerebral artery

Front 30

31. Fainting followed by several hours of unconsciousness. When conscious, pt is unable to speak. On examination, there is spastic paralysis of right arm, but no atrophy. Tongue is protruding to right but is not atrophic. Right facial muscles below the eye were paralyzed. This deficit is called =

Back 30

Broca’s aphasia

Front 31

32. Section of splenium of corpus callosum.

How can you fix this?

Back 31

By cutting of splenium, Right and Left Occipital lobe

Front 32

33. Stroke involving post cerebral artery. You can expect

Back 32

homonymous hemianopsia with macular sparing

Front 33

34. History of hypertension, right sided clumsiness, pt numbness, miosis, ptosis and anhydrosis and opthalmos of right side. Also has left sided body numbness, vertigo and hoarseness. Stroke in what territory appeared

Back 33

Right vertebral artey

Front 34

35. A pt does not understand what someone is saying. But pt misuses the words.

Back 34

Sensory Apahasia

Front 35

36. If there is damage of subthalamic nucleus. it is =

Back 35

Hemibalismus

Front 36

37. Pt cannot read or write but can speak fluently with normal speech but can misuse words. Pt cannot do calculations. Lesion is in =

hint : fishing

Back 36

Angular Gyrus
near wernickes area

Front 37

38. Homonymous Hemianopsia with macular sparing

where is the Leision

Back 37

Posterior Cebrel artery-

Front 38

39. Pt unable to move right arm and right leg and the lower 2/3 of face are drooped down. The tongue is pointing to the right side. Speech is slurred. Dysarthria. Pt can follow commands. Where is the lesion from this stroke

Back 38

Left side of brain capsular interna or Pons

Front 39

40. Which tracts are involved in this lesion

Back 39

CST and CBT

Front 40

41. Sensory Aphasia

Back 40

- expect speech to be fluent, but they misuse wrong words. They do not understand what somebody is saying

Front 41

42. Angular Gyrus Lesion

Back 41

- connects auditory info and speech. Intact speech but use the wrong words. They cannot read or understand & cannot do calculations

Front 42

43. Patient has fainting and several hours of unconsciousness. When conscious, pt unable to speak. On examination, spastic paralysis of right arm without atrophy. Tongue protruded to the right but was not atrophic. Right facial muscles below the eye were paralyzed.
What is it.
What can you expect about the speech of this person

Back 42

Broca Apahsia

Front 43

Broca Apahsia

Back 43

43. unable to speak.
spastic paralysis of right arm without atrophy.
Tongue protruded to the right not atrophic.
Right facial muscles below the eye paralyzed.

Front 44

44. Section of the splenium of corpus calosum might be expected to prevent the seizure from spreading from which of the following structures?

Back 44

Right and left occipital lobe

Front 45

44. Section of the ___ - ___ - ___ might be expected to prevent the seizure from spreading from
Right and left occipital lobe

Back 45

splenium of corpus calosum

Front 46

45. Impairment of the ability to perform learned complex movements and ataxia. Where is the lesion?

Back 46

Premotor cortex

Front 47

45. Impairment of the ability to perform ___ ___ ____ ____ . shows that a liesion is in the premotor cortex.

Back 47

learned complex movements and ataxia

Front 48

46. When you have spasticity, which part of the brain is involved?

Back 48

UMN-internal capsule

Front 49

46. When you have ____ , UMN-internal capsule in the brain is involved?

Back 49

spasticity