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62 Cards in this Set
- Front
- Back
Clear liquid diet
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a) Need for adequate GI function (coffee ok)
b) No orange juice c) Listen for bowel sounds, flatulence, or BM |
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Full liquid diet
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a) Difficulty chewing
b) Cannot swallow solid foods or thin liquids c) Cream of wheat, ice cream, ensure, milkshake |
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Pureed diet
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a) Smooth consistency
b) Edentulous pts c) Pts without their dentures or no teeth d) Pts usually hate it |
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Mechanical soft diet
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a) Required only minimal chewing before swallowing
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Soft diet
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a) Transition from liquid to regular diet
b) Pt with GI problems c) Pt with trouble swallowing |
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Diet as tolerated
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a) Common Rx postop
b) Permit’s pt’s preferences c) Situations to be taken into account d) Allows for progression as tolerance improves (without stomach problems) |
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Cardiac diet
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a) Low-fat
b) Low salt |
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Renal diet
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a) Low salt
b) Low fluid c) 3 ps: i) Low potassium ii) Low phosphorous iii) Low protein |
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Verification of NG tube placements
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1) Ausculate over stomach
2) X-ray to confirm placement (do not start until confirmed) 3) Check for residual (check every shift; aspirate and see how much is coming out) a) If less than 350 mL return to pt if more than 350 mL hold TF and check in 1 hour. 4) Can be dislodged by vomiting or coughing (mark on the tube once confirmed so that if it becomes dislodged it can be noticed) 5) Can be knotted or kinked in the GI tract |
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What to watch for when pts on enteral feedings?
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1) Clogged feeding tubes
a) Use warm water (DO NOT force) b) Use smaller syringes = higher pressure c) Use of carbonated beverages (cranberry juice or sodas) 2) Aspiration 3) Distended abdomen 4) Nausea/vomiting 5) Diarrhea 6) Abdominal pain 7) Constipation 8) Dehydration 9) HOB – 30 – 45 degrees 10) Tube patency a) Flush with 30 mL of h2o before/after feeding, drug administration, residual checks |
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Monitoring and assessment for pts on parenteral feedings
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1) Monitor VS 4-8 hours
2) Daily weights 3) Blood glucose (every 4-6 hours) 4) Electrolytes 5) CBC 6) Chem panel 7) Change solutions every 24 hours 8) Change lipids every 12 hours including tubing (lipids in glass) |
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TPN
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a) 20-50% glucose
b) 60 mEq k (or more) c) Need dedicated catheters central line |
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PPN
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a) 10-12.5% glucose
b) K is less than 60 mEq peripheral line |
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What to watch for in swallow evaluations?
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a) Collecting food under tongue or in checks:
i) Takes a long time to eat or doesn’t touch food b) Excessive eating time c) Coughing d) Coking e) Drooling f) Wet voice (gurgling voice after eating or swallowing) (say ahhhhh) |
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Starting a swallow evaluation
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a) Sit upright
b) Speech therapist c) Ice chips to sips of water d) Small bites only e) Spoon vs. straw (risk for aspiration with straw is much higher) f) Thick liquids (easier to swallow) |
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Who’s at risk for aspiration? How can it be prevented?
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a) Those on TF; check residual volume; only feed when HOB is 30-45;
b) Swallow evals – change diet according to swallow eval |
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Sequelaes of protein‐calorie malnutrition
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1) Generalized loss of muscle and body fat
2) Muscle wasting 3) Inability to heal 4) Decreased immune function 5) Uses carbohydrates from the liver and muscle to meet metabolic needs 6) Once stores are depleted converts protein into glucose for energy 7) Gluconeogensis occurs (formation of glucose by the liver) 8) Fat stores are used in 4-6 weeks 9) Once fat stores are depleted body proteins from internal organs are no longer spared 10) Liver function becomes impaired 11) Protein synthesis diminished 12) Plasma oncotic pressure ↓ (shift from vascular space to interstitial space) 13) Albumin leaks into interstitial space (edema present) |
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Sequelaes and complications for obesity
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1) HTN
2) CAD 3) Stroke 4) Metabolic disorder (insulin resistant syndrome) 5) Obstructive sleep apnea |
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Recognize tests used to dx metabolic syndrome
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1) Has 3 or more of conditions:
a) Waist circumference: men equal to or greater than 40; woman equal to or greater than 35 b) Triglycerides greater than 150 c) HDL less than 40 in men less than 50 in women d) Bp greater than or equal to 130/85 mm Hg e) Fasting glucose is greater than or equal to 100 mg/dL |
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Patient education for discharge post‐bariatric procedures
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1) Diet should be high in protein low in carbs, fat, roughage, and consist of 6 small meals a day
2) Fluids should not be ingested with meal and in some cases fluids should be restricted to less than 1000 ml/day 3) Fluids and foods high in carbs tend to promote diarrhea and dumping syndrome 4) Diet must be understood otherwise = anemia, vitamin deficiencies, diarrhea, psychiatric problems 5) Peptic ulcer formation, dumping syndrome, and small bowel obstruction may be seen late in the recovery and rehabilitative stage. 6) Long term follow up care must be stressed 7) Massive weight loss often leaves patients with large amounts of flabby skin |
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Ways to reduce dumping syndrome
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1) Small, frequent meals
2) No fluids 30-45 mins before or after meals 3) Lay down after meals (semi-folwers) 4) Avoid concentrated sweets 5) Low card moderate protein and fat |
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alcohols withdrawals, DTs
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2) Alcohol hallucinosis-within 48 hrs
a) s/sx of hangover b) > HR, B/P c) Irritability d) Diaphoresis e) Oriented f) Nighmares 3) Delirium Tremens (DTs) – 48-72 hrs a) Sever hypertension b) Tachycardia c) Delirium d) Severe tremors e) Faulty memory f) Rambling speech g) Seizures h) > hallucinations i) Death: seizure, stroke, MIs |
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Assessment for alcohol withdraws
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a) Last drink? (exact time)
b) What did you drink? c) How much? d) How long have you been drinking? e) Do you feel the need to cut down drinking? f) Are you annoyed by people who complained about your drinking? g) Do you feel guilty about drinking? h) Do you need an eye-opener when you wake up? |
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Tx for alcohol withdraws
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1) Nitritional & fluid replacements
2) Banana bag- NS with supplemenets 3) Precautions 4) Fall 5) Seizure 6) Meds 7) Benzodiazepines-Ativan 8) May have to give in IV drip 9) CNS depressant 10) Monitor VS |
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ETOH consequences/ deficiencies
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a) Alcoholic cirrhosis
b) Irreversible inflammatory disease c) Hepatic tissue fibrosed & nodular d) Malnutrition e) inhibits export of proteins and alters metabolism of vitamins/minerals f) Hypoglycemia g) Hypomagnesemia h) Hypokalemia i) Folic deficiency j) Thiamine deficiency |
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What is third spacing and patient population at risk
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a) Third spacing
i) Fluid accumulation in part of body where it is not easily exchanged with ECF ii) Interstitial spaces & connective tissues between the cells iii) fluid is trapped iv) unavailable for functional use. v) Examples: ascites, burns, massive bleeding |
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Hypervolemia
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i) Too much extracellular fluid
ii) Edema iii) Peripheral iv) Pitting v) Fluids in lungs (crackles) vi) > weight |
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Hypovolemia
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i) Too little extracellular fluid
ii) < weight iii) > pulse iv) Dry skin / mucous membranes v) < Skin turgor |
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Hypernatremia
Nursing Implementation |
Serum sodium levels must be reduced gradually to avoid cerebral edema
Fluid balance • Increase fluid intake • D5W • Diuretics • Decrease fluid retention |
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Hypernatremia
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i) Elevated serum sodium occurring with water loss or sodium gain
ii) Causes hyperosmolality leading to cellular dehydration iii) Primary protection is thirst from hypothalamus i) Na > 145 mEq ii) Too much sodium iii) concentrated (dehydration) iv) Manifestations (1) Thirst, lethargy, agitation (2) Impaired LOC (3) Dry mucous membranes |
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Hyponatremia
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i) Na < 135
ii) Too little sodium iii) Diluted (water excess) iv) Manifestations Fatigue, confusion, nausea, vomiting |
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Hyponatremia
Nursing Management |
Treat by water excess
• Fluid restriction Treat sodium loss • Give NS if needed (NPO) |
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Hyperkalemia
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i) K > 5.0
ii) High serum potassium caused by (1) Massive intake (2) Impaired renal excretion (3) Shift from ICF to ECF iii) Common in massive cell destruction (1) Burn, crush injury, or tumor lysis |
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Hyperkalemia Manifestations
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i) Confusion
ii) Paresthesias-numbness and tingling iii) Weak or paralyzed skeletal muscles iv) Abdominal cramping v) EKG changes vi) > T waves vii) Widened QRS Interval |
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Hypokalemia
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a) K < 3.5
b) Potassium loss i) Use of Lasix diuretic ii) Use of insulin-K goes into ICF-< serum K level iii) Prolonged vomiting, diarrhea |
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hypokalemia Manifestations
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i) • Most serious are cardiac
ii) • Inverted T waves iii) • Skeletal muscle weakness iv) • Weakness of respiratory muscles v) • Decreased gastrointestinal motility |
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Hypercalcemia
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i) High serum calcium levels caused by
(1) Hyperparathyroidism (two thirds of cases) (2) Malignancy (3) Vitamin D overdose (4) Prolonged immobilization (5) Osteoporosis (6) Ca++ is liberated into bloodstream |
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Hypercalcemia Manifestations
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(1) Confusion, disorientation
(2) Fatigue, weakness (3) Muscle spasm (4) Shortened QT interval |
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Hypocalcemia
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i) Low serum Ca levels caused by:
(1) Decreased production of PTH (a) Gland removal (2) Vitamin D deficiency (a) Hypertrophy of parathyroid (PTH) gland (i) Demineralization (3) Multiple blood transfusions (4) Decreased intake |
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Hypocalcemia Manifestations
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(1) Positive Trousseau’s sign
(2) Positive Chvostek’s sign (3) Prolonged QT interval (4) Tingling around the mouth or in the extremities-paresthesias (pins & needles) (5) Bone pain / Fractures (a) Demineralization (i) Mobilization of calcium from bone |
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Hyperphosphatemia
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i) Causes
(1) • Renal failure (2) • Chemotherapy (3) • Excessive ingestion of phosphate (4) • In dairy products |
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Hyperphosphatemia Manifestations
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iii) Calcified deposition in joints, arteries, skin, kidneys, heart, lung, blood vessels,
iv) Crystal deposits (1) Neuromuscular irritability and tetany |
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Hypophosphatemia
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(1) Low serum PO4
(a) − caused by Malnourishment/malabsorption (ii) Alcohol withdrawal (iii) Use of phosphate-binding agents (iv) During parenteral nutrition with inadequate replacement |
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Hypomagnesemia
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i) Causes
(1) Prolonged fasting or starvation (2) Chronic alcoholism |
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Hypomagnesemia Manifestations
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(1) Confusion
(2) Hyperactive deep tendon reflexes (3) Involuntary twitching (4) Seizures (5) Cardiac dysrhythmias |
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Hypermagnesemia
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i) High serum Mg caused by
(1) Increased intake or ingestion of products containing magnesium when renal insufficiency or failure is present (2) Milk of magnesia (MOM) (3) Maaalox |
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Hypermagnesemia Manifestations
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(1) Nausea/vomiting
(2) Impaired reflexes (3) Loss of deep tendon reflex (DTR) (4) Lethargy or drowsiness (5) Respiratory and cardiac arrest |
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Sodium values
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i) Na + (135-145 mEq)
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Potassium values
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i) K + (3.5-5 mEq)
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Potassium food sources
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i) Banana, sweet potato, potato, clams, plain yogurt, halibut, soybeans,
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Calcium food sources
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i) Fortified cereal, tofu, spinach, mollasus, turnip, oatmeal, soybeans, greens, yogurt, cheese, milk
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Magnesium food sources
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Pumpkin, halibut, mixed nuts (pine nuts), tuna, artichokes, brown rice
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Phosphorous food sources
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i) Garlic, wheat bran, cheese, popcorn, nuts, liver, tuna, eggs
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Hyperkalemia meds
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i) IV glucose
ii) Insulin-Regular for IV administration iii) Calcium gluconate iv) Kayexalate (1) Exchanges K for Na (2) Diarrhea-uses sorbitol for evacuation of K+ v) Needs to be on telemetry monitoring |
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Hypocalcemia meds
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i) Give calcium PO
(1) Calcium carbonate (Tums) (2) Calcitriol (3) Sensipar ii) Give Calcium IV (1) A vesicant |
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Hyperphosphatemia meds
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i) Phosphate binding agents
(1) PhosLO (2) Renagel ii) Calcium supplements |
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Precautions for giving IV potassium
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Use with caution in patients with cardiac disease, severe renal impairment, hyperkalemia
Never give rapid bolus K+ Maximum adult dose is 100-200 mmol/24 hour Do not give higher than 10 mmol in a periphal IV as it can cause pain and sclerosis Use iv pump when giving higher signs to control rate and observe for signs of extravasation Avoid iv potassium if pt is dehydrated or has sever renal impairment. Adequate urine flow is required before iv k+ can be administered. K+ can leak and cause tissue damage |
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Precautions for giving IV calcium
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Can cause serious tissue damage and death if leaks
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Hypotonic
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i) • More water than electrolytes
(1) • Pure water lyses RBCs ii) • Water moves from ECF to ICF by osmosis iii) • Usually maintenance fluids (1) • ½ or 0.45% NS iv) Lower osmotic pressure than the cells v) Lowers serum osmolality, causing body vi) fluids out of blood vessels and into the vii) cells and interstitial space viii) Indication: cellular dehydration ix) Monitor: can cause shift of fluid into cells b) • Intravascular fluid depletion and cardiovascular collapse |
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Do not give Hypotonic to :
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a) Those at risk for third spacing
b) Liver pts c) Sever malnutrition d) Burn / trauma pts e) Neuro patients f) Increase in intracranial pressures |
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Hypertonic
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i) • Initially expands and raises the osmolality of ECF
ii) • Require frequent monitoring of (1) • Blood pressure (2) • Lung sounds (3) • Serum sodium levels iii) Greater osmotic pressure the cell iv) Raises serum osmolality, pulling fluids v) from cells and interstitial tissues into the vi) vascular space vii) Indication: intravascular dehydration viii) with interstitial and intracellular fluid ix) overload x) Infuse slowly to prevent circulatory xi) overload (1) • High risk IV fluids |
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Do not give Hypertonic to Those with:
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a) Cardiac disease
b) Renal disease c) Cannot tolerate extra fluid |