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62 Cards in this Set

  • Front
  • Back
Clear liquid diet
a) Need for adequate GI function (coffee ok)
b) No orange juice
c) Listen for bowel sounds, flatulence, or BM
Full liquid diet
a) Difficulty chewing
b) Cannot swallow solid foods or thin liquids
c) Cream of wheat, ice cream, ensure, milkshake
Pureed diet
a) Smooth consistency
b) Edentulous pts
c) Pts without their dentures or no teeth
d) Pts usually hate it
Mechanical soft diet
a) Required only minimal chewing before swallowing
Soft diet
a) Transition from liquid to regular diet
b) Pt with GI problems
c) Pt with trouble swallowing
Diet as tolerated
a) Common Rx postop
b) Permit’s pt’s preferences
c) Situations to be taken into account
d) Allows for progression as tolerance improves (without stomach problems)
Cardiac diet
a) Low-fat
b) Low salt
Renal diet
a) Low salt
b) Low fluid
c) 3 ps:
i) Low potassium
ii) Low phosphorous
iii) Low protein
Verification of NG tube placements
1) Ausculate over stomach
2) X-ray to confirm placement (do not start until confirmed)
3) Check for residual (check every shift; aspirate and see how much is coming out)
a) If less than 350 mL return to pt if more than 350 mL hold TF and check in 1 hour.
4) Can be dislodged by vomiting or coughing (mark on the tube once confirmed so that if it becomes dislodged it can be noticed)
5) Can be knotted or kinked in the GI tract
What to watch for when pts on enteral feedings?
1) Clogged feeding tubes
a) Use warm water (DO NOT force)
b) Use smaller syringes = higher pressure
c) Use of carbonated beverages (cranberry juice or sodas)
2) Aspiration
3) Distended abdomen
4) Nausea/vomiting
5) Diarrhea
6) Abdominal pain
7) Constipation
8) Dehydration
9) HOB – 30 – 45 degrees
10) Tube patency
a) Flush with 30 mL of h2o before/after feeding, drug administration, residual checks
Monitoring and assessment for pts on parenteral feedings
1) Monitor VS 4-8 hours
2) Daily weights
3) Blood glucose (every 4-6 hours)
4) Electrolytes
5) CBC
6) Chem panel
7) Change solutions every 24 hours
8) Change lipids every 12 hours including tubing (lipids in glass)
TPN
a) 20-50% glucose
b) 60 mEq k (or more)
c) Need dedicated catheters
central line
PPN
a) 10-12.5% glucose
b) K is less than 60 mEq
peripheral line
What to watch for in swallow evaluations?
a) Collecting food under tongue or in checks:
i) Takes a long time to eat or doesn’t touch food
b) Excessive eating time
c) Coughing
d) Coking
e) Drooling
f) Wet voice (gurgling voice after eating or swallowing) (say ahhhhh)
Starting a swallow evaluation
a) Sit upright
b) Speech therapist
c) Ice chips to sips of water
d) Small bites only
e) Spoon vs. straw (risk for aspiration with straw is much higher)
f) Thick liquids (easier to swallow)
Who’s at risk for aspiration? How can it be prevented?
a) Those on TF; check residual volume; only feed when HOB is 30-45;
b) Swallow evals – change diet according to swallow eval
Sequelaes of protein‐calorie malnutrition
1) Generalized loss of muscle and body fat
2) Muscle wasting
3) Inability to heal
4) Decreased immune function
5) Uses carbohydrates from the liver and muscle to meet metabolic needs
6) Once stores are depleted converts protein into glucose for energy
7) Gluconeogensis occurs (formation of glucose by the liver)
8) Fat stores are used in 4-6 weeks
9) Once fat stores are depleted body proteins from internal organs are no longer spared
10) Liver function becomes impaired
11) Protein synthesis diminished
12) Plasma oncotic pressure ↓ (shift from vascular space to interstitial space)
13) Albumin leaks into interstitial space (edema present)
Sequelaes and complications for obesity
1) HTN
2) CAD
3) Stroke
4) Metabolic disorder (insulin resistant syndrome)
5) Obstructive sleep apnea
Recognize tests used to dx metabolic syndrome
1) Has 3 or more of conditions:
a) Waist circumference: men equal to or greater than 40; woman equal to or greater than 35
b) Triglycerides greater than 150
c) HDL less than 40 in men less than 50 in women
d) Bp greater than or equal to 130/85 mm Hg
e) Fasting glucose is greater than or equal to 100 mg/dL
Patient education for discharge post‐bariatric procedures
1) Diet should be high in protein low in carbs, fat, roughage, and consist of 6 small meals a day
2) Fluids should not be ingested with meal and in some cases fluids should be restricted to less than 1000 ml/day
3) Fluids and foods high in carbs tend to promote diarrhea and dumping syndrome
4) Diet must be understood otherwise = anemia, vitamin deficiencies, diarrhea, psychiatric problems
5) Peptic ulcer formation, dumping syndrome, and small bowel obstruction may be seen late in the recovery and rehabilitative stage.
6) Long term follow up care must be stressed
7) Massive weight loss often leaves patients with large amounts of flabby skin
Ways to reduce dumping syndrome
1) Small, frequent meals
2) No fluids 30-45 mins before or after meals
3) Lay down after meals (semi-folwers)
4) Avoid concentrated sweets
5) Low card moderate protein and fat
alcohols withdrawals, DTs
2) Alcohol hallucinosis-within 48 hrs
a) s/sx of hangover
b) > HR, B/P
c) Irritability
d) Diaphoresis
e) Oriented
f) Nighmares
3) Delirium Tremens (DTs) – 48-72 hrs
a) Sever hypertension
b) Tachycardia
c) Delirium
d) Severe tremors
e) Faulty memory
f) Rambling speech
g) Seizures
h) > hallucinations
i) Death: seizure, stroke, MIs
Assessment for alcohol withdraws
a) Last drink? (exact time)
b) What did you drink?
c) How much?
d) How long have you been drinking?
e) Do you feel the need to cut down drinking?
f) Are you annoyed by people who complained about your drinking?
g) Do you feel guilty about drinking?
h) Do you need an eye-opener when you wake up?
Tx for alcohol withdraws
1) Nitritional & fluid replacements
2) Banana bag- NS with supplemenets
3) Precautions
4) Fall
5) Seizure
6) Meds
7) Benzodiazepines-Ativan
8) May have to give in IV drip
9) CNS depressant
10) Monitor VS
ETOH consequences/ deficiencies
a) Alcoholic cirrhosis
b) Irreversible inflammatory disease
c) Hepatic tissue fibrosed & nodular
d) Malnutrition
e) inhibits export of proteins and alters metabolism of vitamins/minerals
f) Hypoglycemia
g) Hypomagnesemia
h) Hypokalemia
i) Folic deficiency
j) Thiamine deficiency
What is third spacing and patient population at risk
a) Third spacing
i) Fluid accumulation in part of body where it is not easily exchanged with ECF
ii) Interstitial spaces & connective tissues between the cells
iii) fluid is trapped
iv) unavailable for functional use.
v) Examples: ascites, burns, massive bleeding
Hypervolemia
i) Too much extracellular fluid
ii) Edema
iii) Peripheral
iv) Pitting
v) Fluids in lungs (crackles)
vi) > weight
Hypovolemia
i) Too little extracellular fluid
ii) < weight
iii) > pulse
iv) Dry skin / mucous membranes
v) < Skin turgor
Hypernatremia
Nursing Implementation
Serum sodium levels must be reduced gradually to avoid cerebral edema
Fluid balance
• Increase fluid intake
• D5W
• Diuretics
• Decrease fluid retention
Hypernatremia
i) Elevated serum sodium occurring with water loss or sodium gain
ii) Causes hyperosmolality leading to cellular dehydration
iii) Primary protection is thirst from hypothalamus
i) Na > 145 mEq
ii) Too much sodium
iii) concentrated (dehydration)
iv) Manifestations
(1) Thirst, lethargy, agitation
(2) Impaired LOC
(3) Dry mucous membranes
Hyponatremia
i) Na < 135
ii) Too little sodium
iii) Diluted (water excess)
iv) Manifestations
Fatigue, confusion, nausea, vomiting
Hyponatremia
Nursing Management
Treat by water excess
• Fluid restriction
Treat sodium loss
• Give NS if needed (NPO)
Hyperkalemia
i) K > 5.0
ii) High serum potassium caused by
(1) Massive intake
(2) Impaired renal excretion
(3) Shift from ICF to ECF
iii) Common in massive cell destruction
(1) Burn, crush injury, or tumor lysis
Hyperkalemia Manifestations
i) Confusion
ii) Paresthesias-numbness and tingling
iii) Weak or paralyzed skeletal muscles
iv) Abdominal cramping
v) EKG changes
vi) > T waves
vii) Widened QRS Interval
Hypokalemia
a) K < 3.5
b) Potassium loss
i) Use of Lasix diuretic
ii) Use of insulin-K goes into ICF-< serum K level
iii) Prolonged vomiting, diarrhea
hypokalemia Manifestations
i) • Most serious are cardiac
ii) • Inverted T waves
iii) • Skeletal muscle weakness
iv) • Weakness of respiratory muscles
v) • Decreased gastrointestinal motility
Hypercalcemia
i) High serum calcium levels caused by
(1) Hyperparathyroidism (two thirds of cases)
(2) Malignancy
(3) Vitamin D overdose
(4) Prolonged immobilization
(5) Osteoporosis
(6) Ca++ is liberated into bloodstream
Hypercalcemia Manifestations
(1) Confusion, disorientation
(2) Fatigue, weakness
(3) Muscle spasm
(4) Shortened QT interval
Hypocalcemia
i) Low serum Ca levels caused by:
(1) Decreased production of PTH
(a) Gland removal
(2) Vitamin D deficiency
(a) Hypertrophy of parathyroid (PTH) gland
(i) Demineralization
(3) Multiple blood transfusions
(4) Decreased intake
Hypocalcemia Manifestations
(1) Positive Trousseau’s sign
(2) Positive Chvostek’s sign
(3) Prolonged QT interval
(4) Tingling around the mouth or in the extremities-paresthesias (pins & needles)
(5) Bone pain / Fractures
(a) Demineralization
(i) Mobilization of calcium from bone
Hyperphosphatemia
i) Causes
(1) • Renal failure
(2) • Chemotherapy
(3) • Excessive ingestion of phosphate
(4) • In dairy products
Hyperphosphatemia Manifestations
iii) Calcified deposition in joints, arteries, skin, kidneys, heart, lung, blood vessels,
iv) Crystal deposits
(1) Neuromuscular irritability and tetany
Hypophosphatemia
(1) Low serum PO4
(a) − caused by Malnourishment/malabsorption
(ii) Alcohol withdrawal
(iii) Use of phosphate-binding agents
(iv) During parenteral nutrition with inadequate replacement
Hypomagnesemia
i) Causes
(1) Prolonged fasting or starvation
(2) Chronic alcoholism
Hypomagnesemia Manifestations
(1) Confusion
(2) Hyperactive deep tendon reflexes
(3) Involuntary twitching
(4) Seizures
(5) Cardiac dysrhythmias
Hypermagnesemia
i) High serum Mg caused by
(1) Increased intake or ingestion of products containing magnesium when renal insufficiency or failure is present
(2) Milk of magnesia (MOM)
(3) Maaalox
Hypermagnesemia Manifestations
(1) Nausea/vomiting
(2) Impaired reflexes
(3) Loss of deep tendon reflex (DTR)
(4) Lethargy or drowsiness
(5) Respiratory and cardiac arrest
Sodium values
i) Na + (135-145 mEq)
Potassium values
i) K + (3.5-5 mEq)
Potassium food sources
i) Banana, sweet potato, potato, clams, plain yogurt, halibut, soybeans,
Calcium food sources
i) Fortified cereal, tofu, spinach, mollasus, turnip, oatmeal, soybeans, greens, yogurt, cheese, milk
Magnesium food sources
Pumpkin, halibut, mixed nuts (pine nuts), tuna, artichokes, brown rice
Phosphorous food sources
i) Garlic, wheat bran, cheese, popcorn, nuts, liver, tuna, eggs
Hyperkalemia meds
i) IV glucose
ii) Insulin-Regular for IV administration
iii) Calcium gluconate
iv) Kayexalate
(1) Exchanges K for Na
(2) Diarrhea-uses sorbitol for evacuation of K+
v) Needs to be on telemetry monitoring
Hypocalcemia meds
i) Give calcium PO
(1) Calcium carbonate (Tums)
(2) Calcitriol
(3) Sensipar
ii) Give Calcium IV
(1) A vesicant
Hyperphosphatemia meds
i) Phosphate binding agents
(1) PhosLO
(2) Renagel
ii) Calcium supplements
Precautions for giving IV potassium
Use with caution in patients with cardiac disease, severe renal impairment, hyperkalemia
Never give rapid bolus K+
Maximum adult dose is 100-200 mmol/24 hour
Do not give higher than 10 mmol in a periphal IV as it can cause pain and sclerosis
Use iv pump when giving higher signs to control rate and observe for signs of extravasation
Avoid iv potassium if pt is dehydrated or has sever renal impairment. Adequate urine flow is required before iv k+ can be administered.
K+ can leak and cause tissue damage
Precautions for giving IV calcium
Can cause serious tissue damage and death if leaks
Hypotonic
i) • More water than electrolytes
(1) • Pure water lyses RBCs
ii) • Water moves from ECF to ICF by osmosis
iii) • Usually maintenance fluids
(1) • ½ or 0.45% NS
iv) Lower osmotic pressure than the cells
v) Lowers serum osmolality, causing body
vi) fluids out of blood vessels and into the
vii) cells and interstitial space
viii) Indication: cellular dehydration
ix) Monitor: can cause shift of fluid into cells
b) • Intravascular fluid depletion and cardiovascular collapse
Do not give Hypotonic to :
a) Those at risk for third spacing
b) Liver pts
c) Sever malnutrition
d) Burn / trauma pts
e) Neuro patients
f) Increase in intracranial pressures
Hypertonic
i) • Initially expands and raises the osmolality of ECF
ii) • Require frequent monitoring of
(1) • Blood pressure
(2) • Lung sounds
(3) • Serum sodium levels
iii) Greater osmotic pressure the cell
iv) Raises serum osmolality, pulling fluids
v) from cells and interstitial tissues into the
vi) vascular space
vii) Indication: intravascular dehydration
viii) with interstitial and intracellular fluid
ix) overload
x) Infuse slowly to prevent circulatory
xi) overload
(1) • High risk IV fluids
Do not give Hypertonic to Those with:
a) Cardiac disease
b) Renal disease
c) Cannot tolerate extra fluid