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21 Cards in this Set
- Front
- Back
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T3/T4
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"Thyroid"
1. Inc BMR 2. Add to blood anything used to make energy --- so that we can do gluconeogenesis & glycogenesis in liver --- therefore it stims insulin secretion so liver can uptake glucose 3. Tissue Growth |
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Cortisol
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Adrenal Cortex
1. @ Liver: Gluconeogenesis & Glucogenesis 2. @ TT: Stims PRO Catab & Inhibs PRO Synth |
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Aldosterone
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Adrenal Cortex
1. Na+ & H2O reabsorption to Inc BV 2. K+ Excretion |
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Catecholamines
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Adrenal Medulla
1. Add to blood anything that can be used to make energy 2. Inc CV & Resp. Fnctn |
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Ghrelin
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1. Stim GH
2. Inhib Leptin --> Inc Appetite |
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GH
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Ant Pituitary
1. Linear Growth 2. Preserve Lean Body Mass (PRO In Muscle) by using fats for energy |
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IGF
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Liver
1. Primary: GH-Like Effects 2. Secondary: Insulin-Like Effects |
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GH
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Ant Pituitary
1. Linear Growth 2. Preserve Lean Body Mass (PRO In Muscle) by using fats for energy |
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What is the 'SYMPATHETIC' response on 'DIGESTIVE' (SECRETION)?
What receptor is involved in this? |
INHIBITED
CONTRACTION OF INTESTINAL SPHINCTERS (ALPHA-1 RECEPTOR) |
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Insulin
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Panc
1. Remove from blood anything used to make energy |
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Pancreatic Polypeptide
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Panc F Cells
1. Add to blood anything that can be used to make energy 2. EXCEPT supresses appetite |
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Endocrine Adipocyte
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1. CTL Food Intake Along w/ stomach
2. CTL metab of substrate distribution along w/ counter-reg hormones |
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Leptin
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1. Dec Appetite
2. Inc Lipolysis 3. Permissive for LH, FSH |
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PTH
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1. Inc Blood Ca++ from bone
2. Dec Blood Pi by kidney excretion |
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Vit D
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Inc Blood Ca++ & Pi by stim intest absorption
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Calcitonin
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1. Dec Blood Ca++ from Bone
2. Inc Pi reabsorption in kidney 3. Dec Ca++ reabsorption |
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Adiponectin
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1. Glycogenesis
2. Lipolysis |
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Resistin
***Stim by what to do what?*** |
1. Inhib Glycogenesis
2. Stim Glycogenolysis 3. Stim by insulin to competitively inhib insulin --> above effects |
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TNF-a
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1. Inhib PPAR-gamma -->Inhib of pre-adipocytes to Adipocytes
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MCP-1
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Stim Macs to release TNF-a
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SOCS
***When does it become overexpressed?**** |
1. Inhib Leptin & IL-6
2. Inhib Insulin Receptor ***Becomes overexpressed if you have too many cytokines*** |
