Study your flashcards anywhere!

Download the official Cram app for free >

  • Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

How to study your flashcards.

Right/Left arrow keys: Navigate between flashcards.right arrow keyleft arrow key

Up/Down arrow keys: Flip the card between the front and back.down keyup key

H key: Show hint (3rd side).h key

A key: Read text to speech.a key

image

Play button

image

Play button

image

Progress

1/30

Click to flip

30 Cards in this Set

  • Front
  • Back
main cholesterol carriers
LDL
remove excess cholesterol from blood and tissue
HDL
exogenous pathway
chylomicrons
transfer TG to cells
lose TG and becomes LDL
VLDL
secreted by liver as bile
LDL
if not used, deposit on walls
LDL
HDL is made where?
liver and intestine
responsible for reverse cholesterol trasnport
HDL
transfers TG
VLDL
Chylomicrons made where?
intestine
magic number for LDL
100 mg/dL
formula for VLDL
TG/5
total cholesterol formula
Total = HDL + LDL + VLDL
magic number for HDL
40-60 mg/dL
good/bad total choleterol number
<200/>240
magic number of Triglycerides
150 mg/dL
freqency of lab tests for atherosclerosis
2x over 6 month period
homozygous hypercholesterolemia etiology
no cell surface receptors for LDL; unable to metabolize
range of LDL for homo hypercholesterolemia
5-7x LDL of normal
LDL range for hetero hypercholesterimia
2x normal
purpose of lipoprotein lipase
cleave TG off CM
etiology of dysbetaliopoproteinemia
less apoprotein, unable to carry other cholesterol
etiology of hyperchylomicroemia
abnormal enzyme lipolipase
two main lipids in blood
cholesterol and Triglyceride
responsibilities of cholesterol
animal cell membrane development
nerve tissue formation
steroid hormones
classification of lipoprotein
amount of TG or apoprotein
contains very little TG
HDL
less dense than HDL
more dense than CM
LDL
foam cells
macrophages ingests LDL and oxidizes
binds with excess cholesterol and retursn it to the liver
HDL