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51 Cards in this Set
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Q650. when do diaphragms and cervical caps need to be refitted postpartum?
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A650. 6 weeks
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Q651. what are the (3) hormonal contraceptives of choice postaprtum?; Why?
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A651. Depo-provera,; Norplant,; Progesterone-only minipill b/c they are less likely to decrease milk production in breast-feeding patients
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Q652. What are the causes of postpartum hemorrhage?; (6)*
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A652. Coagulation Defect;; Atony;; Rupture;; Placenta (POC) retained;; Implantation site bleed;; Trauma
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Q653. what are the steps in managing a postpartum hemorrhage?; (4 steps)
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A653. 1. RULE OUT cervical/vaginal lacerations; 2. if still bleeding: give Uterotonic agents (Oxytocin); 3. if still bleeding: D&C; 4. if still bleeding: Laparotomy with bilateral O'Leary sutures to tie off uterine arteries
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Q654. Dx:; fever, high WBC, uterine tenderness 5 - 10 days post C- section; Tx?; (2)
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A654. Endomyometritis; Tx: D&C, broad-spectrum Antibiotics until afibrile for 48 hrs
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Q655. what is the underlying cause of labial fusion?
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A655. excess Androgens
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Q656. MC form of enzymatic deficiency assoc with ambiguous genitalia; what is deficient?
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A656. Congenital Adrenal hyperplasia; (21-hydroxylase deficiency)
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Q657. Dx:; hyperandrogenism, salt wasting, hypotension, hyperkalemia, hypoglycemia, ambiguous genitalia
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A657. Congenital Adrenal Hyperplasia; (21-hydroxylase deficiency)
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Q658. what main lab is elevated in Congenital Adrenal Hyperplasia?; what is Tx?
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A658. 17-alpha-hydroxyprogesterone; Tx: Cortisol (and a mineralcorticoid if pt is salt-wasting)
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Q659. what is the name of the fertilized oocyte 2 - 4 days after fertilization?; what is it called in the next stage?
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A659. Blastomere / Morula; next: Blastocyst
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Q660. Dx:; patient at puberty with primary amenorrhea and cyclic pelvic pain, lower abdominal girth
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A660. Imperforate hymen
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Q661. Definition:; build-up of blood behind the hymen in person with imperforate hymen; Tx?
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A661. Hematocolpos; Tx: surgery
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Q662. (2) causes of Vaginal Agenesis
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A662. Mayer-Rokitansky-Kuster-Hauser syndrome (MRKH); Androgen Insensitivity
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Q663. Dx:; normal female karyotype with ovaries and secondary sexual characteristics, but congenital absence or hypoplasia of vagina, cervix, uterus and fallopian tubes
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A663. Mayer-Rokitansky-Kuster-Hauser syndrome (MRKH)
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Q664. what is the Tx for Mayer-Rokitansky-Kuster-Hauser syndrome (MRKH)?
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A664. Create vagina: with dilators; or McIndoe procedure (surgically creating vagina with skin grafts)
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Q665. Dx:; woman with scant pubic hair and small breasts with vaginal agenesis or absence and absence of uterus; cause?
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A665. Androgen Insensitivity; cause: nonfunctioning androgen receptors (normal levels of Testosterone)
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Q666. Dx:; symmetric white, thinned skin on labia, perineum and perianal region. Shrinkage and agglutination of labia with occasional pruritis or dysparunia; Tx?
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A666. Lichen Sclerosis; Tx: Topical steroids (Clobetasol)
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Q667. Dx:; localized thickening of the vuvlar skin from edema with chronic pruritis, possible raised white lesion on labia majora or clitoris
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A667. Squamous cell hyperplasia
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Q668. Dx:; multiple shiny, flat, purple papules usu on the inner aspects of the labia minora, vagina and vestibule. Often erosive and causing pruritis and mild inflammation
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A668. Lichen Planus
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Q669. Dx:; Thickened white epithelium, slight scaling, usually unilateral and circumscribed on vulva, with pruritis; (2) Tx?
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A669. Lichen Simplex Chronicus; Tx: Ultraviolet light, Topical steroids
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Q670. Dx:; Red, moist and sometimes scaly lesions on vulva, which may also be found on scalp, axilla, groin and trunk
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A670. Vulvar Psoriasis
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Q671. Dx:; palpable red granular spots and patches in the upper third of the vagina on the anterior wall
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A671. Vaginal Adenosis
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Q672. how are vulvar lesions Dx?
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A672. Biopsied
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Q673. MC benign tumor on the vulva
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A673. Epidermal Inclusion cysts
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Q674. Definition:; Disease that causes occlusion of the sweat glands on mons pubis and labia majora, causing cyst formations; Tx?
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A674. Fox-Fordyce Disease; Tx: I&D
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Q675. how do you differentiate an epidermal cyst from a sebaceous cyst?
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A675. Epidermal - solitary cyst; Sebaceous - collection of cysts
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Q676. where are the Skene glands located?
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A676. Paraurethral
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Q677. where are the Bartholian glands located?
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A677. Bilaterally at 4 and 8 o'clock on labia majora
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Q678. what is first step in Tx if a Bartholian cyst first appears in woman over 40yo?
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A678. Biopsy to RULE OUT Bartholian gland carcinoma
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Q679. Tx of a Bartholian Abscess; what is Tx for recurrent Bartholian Abscesses?
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A679. Tx: I&D with placement of Word catheter; Recurrent: Marsupialization
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Q680. Definition:; Cervical dilated retention cysts
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A680. Nabothian cysts
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Q681. Definition:; Cervical cysts that lie deep in the stroma and are from remnants of Wolffian ducts
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A681. Mesonephric cysts
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Q682. even though cervical polyps are not premalignant, why are they removed?
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A682. to avoid masking bleeding from other sources and to avoid misidentification for an endometrial polyp
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Q683. MC Uterine formation anomaly; cause?
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A683. Septate uterus; cause: Problems with fusion of Paramesonepheric ducts
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Q684. what are anomalies of the uterus assoc with (non-gyn medical)?; (2)
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A684. Urinary tract anomalies; Inguinal hernias
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Q685. Dx:; amenorrhea or dysmenorrhea, dyspareunia, cyclic pelvic pain, infertility or recurrent pregnancy loss or premature labor
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A685. Uterine anatomic anomalies; (Septate uterus)
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Q686. Dx:; small uterine cavity, second-trimester pregnancy loss, malpresentation and possible premature labor
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A686. Bicornuate uterus
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Q687. Tx of Septate and Bicornuate uteri
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A687. Surgical removal of septum
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Q688. Definition:; Benign, estrogen-sensitive smooth muscle tumors of the uterus; found in what percentage of reproductive-age women?
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A688. Fibroids (Uterine Leiomyomas); in 20 - 30% of reproductive-age women
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Q689. Incidence of Fibroids in Black women; (3) causes to increase risk of developing fibroids
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A689. 3 - 9 x higher in Black Risks:; Non-smoking,; Obese,; PeriMenopausal
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Q690. what distinguishes a Fibroid from adenomyosis?
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A690. Fibroid has a Pseudocapsule
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Q691. Top (2) MC Sx in patient with Fibroids
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A691. Asymptomatic (50 - 65%) (MC otherwise is Prolonged bleeding)
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Q692. Drug Tx for Fibroids; (3); MOA of these drugs collectively
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A692. Provera,; Danzol,; GnRH agonists (Lupron) MOA - shrink fibroids by reducing circulating Estrogen
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Q693. If drugs dont work, what is the name of the surgical Tx for Fibroids?; Only Difinitive Tx?
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A693. Myomectomy (removal of one or more Fibroid surgically); Only Difinitive Tx: Hysterectomy
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Q694. what causes Endometrial Hyperplasia?
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A694. continuous endogenous or exogenous Estrogen in absence of Progesterone
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Q695. In endometrial hyperplasia, what proliferates in endometrium?; (2)
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A695. Glandular and Stromal elements of endometrium
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Q696. Risk factors for getting Endometrial Hyperplasia; (9)
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A696. CLONED PHD:; Chronic Anovulation,; Late Menopause (> age 55),; Obesity,; Nulliparity,; Estrogen-producing tumors (granulosa-theca cell tumor),; Diabetes,; PCOS,; Hypertension,; Drugs - Tamoxifen
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Q697. Dx:; long periods of Oligomenorrhea or amenorrhea followed by irregular or excessive uterine bleeding
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A697. Endometrial Hyperplasia
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Q698. main Dx evaluation used to Dx Endometrial Hyperplasia; what is second choice?
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A698. Endometrial biopsy (or D&C...second choice)
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Q699. Risk of malignant transformation from Endometrial Hyperplasia in:; 1. Simple Hyperplasia; 2. Complex Hyperplasia; 3. Atypical Simple Hyperplasia; 4. Atypical Complex Hyperplasia
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A699. Simple = 1%; Complex = 3%; Atypical Simple = 8%; Atypical Complex = 29%
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Q700. what is the initial Tx for all types of endometrial hyperplasia in child-bearing patient?; Non-child bearing patient?
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A700. Child bearing:; Progestin therapy for 3 months; (followed by resampling of Endometrium); Non-child bearing:; Hysterectomy
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