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9 Cards in this Set

  • Front
  • Back
Describe Cole Hypothesis for Bronchiectasis
1. Environmental/Microbial Insult
2. Genetic Predisposition: defect in host defense
3. LRTI
4. Bronchial Inflammation
5. LRT damage
---back to 3--
-results in progressive lung Dx
Name for layer between Mucus and Ciliated Cells
Periciliary Fluid Layer
--watery fluid in that bathes cilia, what they beat in
With Brochiectasis, what can happen to following cells
Cilia:
Goblet Cells:
Submucosal Glands:
Ciliated Cell Metaplasia: become squamous or columnar/loose cilia
Goblet Cells: hyperplasia
Submuc Cells: hypertrophy
In Bronchiectasis, where is inflammation most prominent?
--Effect on other airways?
In Small Airways--bronchioles
-inflm mediators spill over into larger; effect Elastin w/
-proteases

-inflam can also spill over into interstitium appearing as Pneumonitis
Test Question:
Necessity for Bronchiectasis (2)
Host Immune Problem
Recurrence of Bronchial Obstruction
name and describe the 3 main types of of "reid" dilations
Tubular: smooth dilation
Varicose: dilated with multiple indentations
Cystic: dilated bronchi end in blind sacs
What is typical result of FVC on bronchiectasis if severe
Decreased FVC (restricted dx)

-more often, as it is an obstructive, FVC follows obstructive pattern
What is current Gold Standard for bronchiectasis
High Res CT

--see airway dilation
lack of tapering of these vessels
bronchial wall thickeniing
mucopurulent plugs
What is ABPA
Allergic Brochno Pulmonary Aspergillosus
--common with bronchiectasis
---elevated IgE and anti-Aspergillus ABs
-not actual infxn of aspergillius, buta hyperreactivity to it