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17 Cards in this Set
- Front
- Back
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what is cushing triad's
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bradycardia
widened pulse pressure increased BP |
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decerebrate position vs
decorticate rigidity |
extension of legs and hands
extension legs, flexion hands |
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signs of high ICP
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nasuea
headache vomiting conciosness posture ± Cushing LATE |
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Cheynes Stokes respiration is:
depends on |
apnea with alternating hyperventilation
depends on level of brainstem damage |
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Cheynes Stokes respiration at
midbrain pons |
sustained hyperventilation
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Cheynes Stokes respiration at
upper medulla |
rapid shallow respiration with ataxic breathing in final stages
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why do we have pulmonary edema in the increased ICP
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^ sympathetic activity as a result of increased ISP on HPT and medulla or spinal cord (cervical)
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types of cerebral edema
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vasogenic, cytotoxic, interstitial
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vasogenic edema is
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due to increased permeabiltiy of capillary endothelial tissue
mass occupying lesion |
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cytotoxix
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Cx: cellular atp failure
failure of normal homeostatic mechanisms to maintain cell size |
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interstitial edema
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increased water in the body
hydrocephalus i |
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subdural hematoma vs extra dural
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subdural (crescent)
extra dural (pea) |
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herniation: transtentorial
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from temporal on the cbl
compressing: III nerve ipsilateral cerebral peduncle ipsilateral |
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herniation: subfalcine
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hemisphere to hemisphere
compressing: 1. cingulate gyrus 2. ACA compression 3. may lead to other hernias due to infarction |
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herniation: central
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bilateral temporal lobe
compressing III nerve cerebral peduncle decerebrate posture |
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herniation: upper (infratentorial)
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CBL==> brainstem
3rd ventricle superior cerebellar a. |
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herniation: tonsillar
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CBL on the brainstem
dysconjugate eye movement resp arrest |
