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36 Cards in this Set

  • Front
  • Back
Sx of anemia
fatigue
dyspnea
palpitations
dizziness
dysphagia
Sx of severe anemia
pallor
tachycardia
postural HTN
palmar creases appear pink
Sx of pernicious anemia
-skin has "lemmon yellow" tint b/c simultaneous presence of pallor and jaundice
-no papillae on tongue
gingival hypertrophy
acute leukemia
sx of lead poisoning
dark line of lead sulfide deposited in gums at base of teeth
clinical Sx of iron deficiency anemia
koilonychia (spoon shaped fingernails)
pencil (cigar) cells
iron def anemia
Fe/TIBC for iron def anemia
Fe/TIBC <= 10%
Fe/TIBC for chronic dz
>15%
iron studies for sideroblastic anemia
increaed Fe, norm TIBC, increased Fe/TIBC ,
inc ferritin
hypersegmented polymorphs
megaloblastic aenmia (B12/folate def)
spur cells (acanthocytes)
liver dz (spur/burr cells in Abetalipoprotenia (can't make VLDL, LDL)--thus liver dz)
target cells
liver dz
hemoglobinopathies
post-splenectomy
microspherocytes
autoimmune hemolytic anemia
hereditary spherocytosis
Howell Jolly bodies
-megaloblastic anemias (B12 def, pernicious anemia, folate def)
-post-splenectomy
rouleax formation
-usu seen in multiple myeloma
-stacked RBC's caused by coating of individual cells by charged proteins that neutralize the normally negative surface charge that by repulsion keeps RBC's from agglutinating
increased UB indicates
hemolysis
increased LDH indicates
hemolysis
decreased haptoglobin
hemolysis
hemolytic transfusion rxns have + or - Coombs test?
+ Coombs test
cause of iron deficiency
BLOOD LOSS!
how evaluate iron deficiency
evaluate for BLOOD LOSS!
Do upper and lower GI endoscopy. Can't just supplement with iron, must control the bleed
best route for iron therapy
oral (rather than parenteral)
what should you do for pts wo don't respond to B12 or folate?
evaluate for concurrent iron def:
-BM irons tores are reliable indicators of iron status at any time
-Peripheral blood iron studies: must wait 7-10 days after initation of foldate/B2 Tx to get accurate results
Tx TTP
plasma exchange
pathogen of Autoimmune hemolytic anemia (AIHA)
binding of Auto-Ig +/- complement to RBC surface-->destroy RBC's in spleen
-75% of Auto-Ig are IgG, 25% IgM
Tx for Autoimmune hemolytic anemia
first corticosteroids;
if fail-then splenectomy
tx for Cold Agglutinin Dz
(Cold Agglutinin Dz=Auto-ab against RBC's are IgM)
-cold avoidance
(corticosteroids and splenectomy are ineffective)
Sx of iron def anemia
(1-3 relate to mouth)
1. PICA
2. Dysphagia
3. no papillae oin tongue
4. koilonychia
gingival hypertrophy
AML
neurologic problems
ALL
target cells
-liver dz
-hemoglobinopahties (target cells)
-post-splenectomy
microspherocytes
autoimmune hemolytic anemia
tear drop cells
BM infiltration
leukoerythroblastosis
BM infiltration
Howell-Jolly bodies
megaloblastic anemias
post-splenectomy