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24 Cards in this Set
- Front
- Back
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neonate
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birth to 1 month
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weanling
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4-6months
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foal
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by mares side
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yearling
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12-18 months
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adult
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>2yrs
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Recurrent Exertioal Rhabdomyolysis
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Thoroughbreds
-issue with ryanidine receptor and Ca -excessive unaccostomed exercise -myoglobinuria and components of muscle enzymes in blood -myoglobin may precipatate in kidneys and cause kidney failure Equally dangerous can be the leakage of potassium into the bloodstream, which under certain circumstances can interfere with propagation of the heartbeat. Another danger is posed by the possible leakage of excessive calcium into the cell, creating a state of hypocalcemia in the bloodstream, which can lead to irregular heartbeat, muscle spasms, and other symptoms. |
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Exercise Induced Pulmonary Hemorrhage
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Thoroughbred
QH Standardbred -intense exercise |
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Gastric ulcers
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housing, concentrates
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Osteochondrosis desicans
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increased Energy w. growth that is too fast
OCD in neck=wobblers |
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SCID
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Arabians
severe combined immunodeficiency -no competent immune system |
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cerebellar abiotrophy
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Arabians
recessive mode of inheritance for this condition. This means that a horse can "carry" the disease gene but not be affected by it. However, breedings between two carrier horses will produce an affected foal 25% of the time. -Foals affected with CA appear normal at birth. Around six weeks of age (although sometimes as late as four months), the disease causes the death of neurons in the cerebellum of affected foals, leading to head tremor (intention tremor) and a lack of balance equilibrium (ataxia). Affected horses may show exaggerated action of the forelegs, a wide-based stance, and be unable to rise from a reclining position. They tend to startle easily and often fall due to ataxia. The neurological problems may not be apparent to owners and are frequently thought to be a consequence of a fall rather than the cause of it. CA symptoms vary in severity. Some foals show very severe symptoms, including the exaggerated gaits and a dramatic lack of balance. Others have little more than the head tremor, which may only manifest itself during goal-directed movement. Regardless of the severity of the symptoms, CA foals are often euthanized or restricted to life as pasture pets, as they are never coordinated enough to be ridden safely. |
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Idiopathic epilepsy
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Arabians
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Hyperkalemic Periodic Paralysis
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QH
Impressive line **genetic test! inherited autosomal dominant disorder which affects sodium channels in muscle cells and the ability to regulate potassium levels in the blood. It is most commonly associated with horses, but also occurs in humans, where it may be called Gamstorp episodic adynamy. It is characterized by muscle hyperexcitability or weakness which, exacerbated by potassium or cold, can lead to uncontrolled shaking followed by paralysis. -mutation which causes this disorder is dominant on SCN4A with linkage to the sodium channel expressed in muscle. The mutation causes single amino acid changes in parts of the channel which are important for inactivation. In the presence of high potassium levels, including those induced by diet, sodium channels fail to activate properly. |
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Polysaccharide Storage Myopathy
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QH
monday morning sickness -glycogen storage in muscle cells |
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HERDA
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QH
Hereditary , regional, equine, derm problem w. subQ tissues **Genetic test! Affected foals rarely show symptoms at birth. The condition typically occurs by the age of two, most notably when the horse is first being broke to saddle. There is no cure, and the majority of diagnosed horses are euthanized because they are unable to be ridden and are inappropriate for future breeding. |
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Granulomatous enteritis
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Standardbred
inflamm bowel disease |
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Ileocolonic agangiosis
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American Paint Horse
lethal white foal 2 overos bred together -can't doordinate intestinal movement **genetic test! |
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Equine Recurrent Uveitis
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Appaloosa
-25% affected horses are appaloosas moon blindness! |
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Squamous cell carcinoma
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Appaloosa
-penis/prepuce/3rd eyelid |
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Junctional epidermolysis bullosa
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Belgian
mucocutaneous jxn ie: coronoid bands |
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PSSM
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polysaccharide storage myopathy
QH Draft warmbloods |
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esophageal disorders
-megaesophagus esophageal hypertrophy neonatal dysphagia |
Friesian
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fecalith
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mini
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Equine core vaccs
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EEE/WEE
WNV Tetanus Rabies |
