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98 Cards in this Set

  • Front
  • Back
PMI, Point of Maximal Impulse
left 5th ICS MCL, does not continue into second heart sound
Abnormalities of PMI
displacement due to hypertrophy, cardiomyopathy, ischemic disease, amplitude increase due to hyperthyroidism. Anemia, pressure overlaod of the left ventricle
Pathology and PMI
lateral → volume overload, high amplitude impuse → left ventricular hypertrophy, sustained low amplitude → dialted cardiomyopathy
Systole
ventricular contraction
Diastole
ventricular relaxation
S1
closure of the mitral valve followed by triscuspid valve
S2
closure of the aortic valve followed by the pulmonary valve
S3
extra heart sounds, after mitral valve opening. Rapid deceleration of blood against ventricular walls or from pathologic changes in compliance
S4
marks atrial contraction immediately preceding S1
Spliting of S2
during inspiration S2 splits into two distinct sounds, closure of the aortic and then pulmonary semi-lunar valves
Changes with inspiration
thoracic volume increases, intrathroacic pressure decreases, venous flow to the right heart increases, delayed closure of pulomonary valve, increased pulomary capacitance, decreased left ventricular ejection volume
Aortic valve
R 2nd ICS RSB
Pulmonary Semilunar
left 2nd/3rd ICS LSB
Tricuspid
left 4th/5th ICS LSB
Mitral
4th ICS MCL, is the PMI
Murmur
turbulent blood flow within the heart
Bruit
mumur like sound of vascular origin, external to the heart
Thrill
humming vibrations felt on skin overlying area of turbulent blood flow upon palpation
Jugular Venous Pressure
measured in cm. 3 or 5 cm is above normal
JVP pathology
increased pressure suggest right sided congestive heart failure, constrictive pericarditis, tricuspid stenosis, or SVC obstruction
Grade I Murmur
faint, tuned in
Grade II murmur
quiet but heart immediately after putting stethoscope on chest
Grate III murmur
moderately loud
Grade IV murmur
loud with palable thrill
Grade V murmur
very loud, thrill, my be heard when stethoscope is partly off chest
Grade VI
ery loud, thrill, heard with stethoscope entirely off chest
Aortic Stenosis
Right 2nd ICS parasternally, radiates into carotids,
Aortic Regurgitation
left 2nd to 4th ICS parasternally, Erbs Point, best heard when patient is sitting, learning forward, and breath held after exhalation
Mitral stenosis
apex, no radiation, use bell for presystolic low pitched rumble
Mitral Regurgitation
apex, radiaton to left axilla, does not become louder in inspiration
Innocent murmur
2nd to 4th left ICS between LSB and apex, little radiation, decreases or disappears on sitting
Erb’s point
left 3rd ICS, accentuates aortic murmurs
Left lateral decubitus position
accentuates S3, S4, and mitral murmurs
Diaphgragm
high pitched sounds (S1, S2, and aortic/mitral regurgitation)
Bell
low pitched sounds (S3, S4, mitral stenosis)
Abdominal Quadrants
RUQ, LUQ, RLQ, LRQ
Abdiomnal sections
epigrastric, umbilical, suprapubic, r/l hypochondrium, lumbar, inguinal
What is normally palpable in the abdomen (8)
stomach liver, spleen, bladder, aorta, masses, enlarged kidneys, pregnant uterus
Kidney Tenderness
percussion or palpation at costovertebral angels
Bruits
stenosis of renal arteries, abdominal aorta, or iliac arteries. Best place to listen is below costal margins at MCL and epigastric aorta
Liver span
percussion right MCL, 6-12 ccm on right MCL and 4-8 at mid sternal.
Inaccurate liver span
COPD, right pleural effusion, gas in colon
Spleen
percussion of lowest intercostal space in left anterior axillary line of a supine pt. resonant or tympanic sound, dull sound →splenomegaly
Rebound tenderness
peritoneal inflammation, deep and slow palpation in the abdomen away from suspected site. If pain is felt when hand is removed→ contralateral imflammation
Rovsing’s sign
appendicitis, pressing deeply and evenly in LLQ then quickly removing figures, + if RLQ pain during left sided pressure
Psoas Sign
irritation of psoas muscle by inflamed appendix , right knee and ask to rasie thigh against pressure or flex leg at hip on left side
Murphy’s sign
acute cholecysttis, hook fingers under pts right cosdtal margin and ask pt to take deep breath, + if pain during inspiration while palpating the liver margin
Ascites
free fluid in abdomen, indication of underlying heart failure, liver disease, nephritic syndrome or metastases. Elecvated hydrostatic pressure and decreased osmotic pressure
Shifting dullness
sensitive sign for ascites. Tympanic percussion will shift to superior position
Bulging flanks
fluid in abdomen will move to flank region in supine position in ascetic t
Flank dullness
percussion should be tympanic but will be dull with ascites
Prominent fluid wave
fluid wave when pt compresses subcut fat in abdomen upon flick from doc
Epitrochlear
LN on medial surface of arm
Lateral Axillary
LN in the lateral axillary area, drains radial side of arm and hand
Horizontal Group
LN superficial inguinal, underlies inguinal ligament
Vertical Group
LN superficial inguinal, proximal segment of greater saphenous vein
Deep
LN small saphenous vein at the popliteal space, drains heel and outer foot
Radial Pulse
radial side of palmar surface of wrist
Brachial pulse
medially just under tendon of bicepts at the antecubital crease
Femoral pulse
femoral triangle below the inguinal ligament and about midway between ASIS and symphysis pubis
Popliteal pulse
flex pts knee, fingertips just meet midline behind the knee and press deep into, not easily palpable
Dorsalis pedis
between first and second metatarsals on dorsum of foot, not easly palpable
Posterior tibialiss
behind and below the medial malleolus, absent in 15% of healthy individuals
Allen test
determine arterial insufficiency in the upper extremity by ensuring patency of ulnar artery. Tight fist and compress both radial and ulnar arteries → release pressure over ulnar. Palm will flush in seconds if artery is patent
Major veins of the lower extremity
femoral vein, great saphenous vein, small saphenous vein, popliteal vein, communicating vein
Pitting edema
increased peripheral venous pressure, depression caused by pressure from thumb
Non-pitting edema
stasis of lymph fluid in the tissues
Orthostatic edema
pitting, swollen foot, returns to normal upon postural changes
Lymphedema
firm, non pitting edema, leathery skin
Lipedema
deposition of fat, non pitting, no swelling
Edema of advance chronic venous insufficiency
soft pitting with lesions, can become hard, skin thickening and pigmentation, swelling of foot
Internal rotation
anterior surface of limb inward
External rotation
anterior surface of limb outward
Supination
rotation of hand so palmar surface is upward
Pronation
rotation of hand so palmar surface faces down
Abduction
movement away from midline
Adduction
movement towards midline
Inversion
plantar surface of foot inward
Eversion
planar surface of foot outward
Biceps tendon reflex
C5C6
Triceps tendon reflex
C6C7C8
Brachioradialis tendon reflex
C5C6
Patella
L2L3L4
Achilles
S1S2
4+
brisk, hyperactive, with clonus
3+
brisker than average, not indicative of disease
2+
average, normal
1+
somewhat diminished, lower than average
0
reflex absent
Clonus
involuntary contractions of muscles between flexion and textion that occurs in conditions of increased tone
5
active movement against gravity with full resistance without evident fatigue
4
active movement against gravity with some resistance
3
active movement against gravity
2
cannot act against gravity
1
slight contraction detected
0
no contraction
venous thromboembolism
blood clot or deep vein thrombosis, can lead to pulmonary embolism
Clinical paramaters of venous thromboembolism
active cancer, paralysis, pareisis, cast in lower extremities, bedridden, localized tenderness along distribution of deep venous system, entire leg swelling, calf swelling, pitting edema. Collateral superficial veins