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98 Cards in this Set
- Front
- Back
PMI, Point of Maximal Impulse
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left 5th ICS MCL, does not continue into second heart sound
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Abnormalities of PMI
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displacement due to hypertrophy, cardiomyopathy, ischemic disease, amplitude increase due to hyperthyroidism. Anemia, pressure overlaod of the left ventricle
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Pathology and PMI
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lateral → volume overload, high amplitude impuse → left ventricular hypertrophy, sustained low amplitude → dialted cardiomyopathy
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Systole
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ventricular contraction
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Diastole
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ventricular relaxation
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S1
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closure of the mitral valve followed by triscuspid valve
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S2
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closure of the aortic valve followed by the pulmonary valve
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S3
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extra heart sounds, after mitral valve opening. Rapid deceleration of blood against ventricular walls or from pathologic changes in compliance
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S4
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marks atrial contraction immediately preceding S1
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Spliting of S2
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during inspiration S2 splits into two distinct sounds, closure of the aortic and then pulmonary semi-lunar valves
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Changes with inspiration
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thoracic volume increases, intrathroacic pressure decreases, venous flow to the right heart increases, delayed closure of pulomonary valve, increased pulomary capacitance, decreased left ventricular ejection volume
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Aortic valve
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R 2nd ICS RSB
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Pulmonary Semilunar
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left 2nd/3rd ICS LSB
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Tricuspid
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left 4th/5th ICS LSB
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Mitral
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4th ICS MCL, is the PMI
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Murmur
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turbulent blood flow within the heart
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Bruit
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mumur like sound of vascular origin, external to the heart
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Thrill
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humming vibrations felt on skin overlying area of turbulent blood flow upon palpation
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Jugular Venous Pressure
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measured in cm. 3 or 5 cm is above normal
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JVP pathology
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increased pressure suggest right sided congestive heart failure, constrictive pericarditis, tricuspid stenosis, or SVC obstruction
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Grade I Murmur
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faint, tuned in
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Grade II murmur
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quiet but heart immediately after putting stethoscope on chest
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Grate III murmur
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moderately loud
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Grade IV murmur
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loud with palable thrill
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Grade V murmur
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very loud, thrill, my be heard when stethoscope is partly off chest
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Grade VI
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ery loud, thrill, heard with stethoscope entirely off chest
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Aortic Stenosis
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Right 2nd ICS parasternally, radiates into carotids,
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Aortic Regurgitation
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left 2nd to 4th ICS parasternally, Erbs Point, best heard when patient is sitting, learning forward, and breath held after exhalation
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Mitral stenosis
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apex, no radiation, use bell for presystolic low pitched rumble
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Mitral Regurgitation
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apex, radiaton to left axilla, does not become louder in inspiration
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Innocent murmur
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2nd to 4th left ICS between LSB and apex, little radiation, decreases or disappears on sitting
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Erb’s point
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left 3rd ICS, accentuates aortic murmurs
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Left lateral decubitus position
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accentuates S3, S4, and mitral murmurs
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Diaphgragm
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high pitched sounds (S1, S2, and aortic/mitral regurgitation)
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Bell
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low pitched sounds (S3, S4, mitral stenosis)
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Abdominal Quadrants
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RUQ, LUQ, RLQ, LRQ
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Abdiomnal sections
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epigrastric, umbilical, suprapubic, r/l hypochondrium, lumbar, inguinal
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What is normally palpable in the abdomen (8)
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stomach liver, spleen, bladder, aorta, masses, enlarged kidneys, pregnant uterus
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Kidney Tenderness
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percussion or palpation at costovertebral angels
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Bruits
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stenosis of renal arteries, abdominal aorta, or iliac arteries. Best place to listen is below costal margins at MCL and epigastric aorta
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Liver span
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percussion right MCL, 6-12 ccm on right MCL and 4-8 at mid sternal.
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Inaccurate liver span
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COPD, right pleural effusion, gas in colon
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Spleen
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percussion of lowest intercostal space in left anterior axillary line of a supine pt. resonant or tympanic sound, dull sound →splenomegaly
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Rebound tenderness
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peritoneal inflammation, deep and slow palpation in the abdomen away from suspected site. If pain is felt when hand is removed→ contralateral imflammation
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Rovsing’s sign
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appendicitis, pressing deeply and evenly in LLQ then quickly removing figures, + if RLQ pain during left sided pressure
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Psoas Sign
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irritation of psoas muscle by inflamed appendix , right knee and ask to rasie thigh against pressure or flex leg at hip on left side
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Murphy’s sign
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acute cholecysttis, hook fingers under pts right cosdtal margin and ask pt to take deep breath, + if pain during inspiration while palpating the liver margin
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Ascites
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free fluid in abdomen, indication of underlying heart failure, liver disease, nephritic syndrome or metastases. Elecvated hydrostatic pressure and decreased osmotic pressure
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Shifting dullness
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sensitive sign for ascites. Tympanic percussion will shift to superior position
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Bulging flanks
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fluid in abdomen will move to flank region in supine position in ascetic t
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Flank dullness
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percussion should be tympanic but will be dull with ascites
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Prominent fluid wave
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fluid wave when pt compresses subcut fat in abdomen upon flick from doc
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Epitrochlear
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LN on medial surface of arm
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Lateral Axillary
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LN in the lateral axillary area, drains radial side of arm and hand
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Horizontal Group
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LN superficial inguinal, underlies inguinal ligament
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Vertical Group
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LN superficial inguinal, proximal segment of greater saphenous vein
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Deep
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LN small saphenous vein at the popliteal space, drains heel and outer foot
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Radial Pulse
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radial side of palmar surface of wrist
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Brachial pulse
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medially just under tendon of bicepts at the antecubital crease
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Femoral pulse
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femoral triangle below the inguinal ligament and about midway between ASIS and symphysis pubis
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Popliteal pulse
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flex pts knee, fingertips just meet midline behind the knee and press deep into, not easily palpable
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Dorsalis pedis
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between first and second metatarsals on dorsum of foot, not easly palpable
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Posterior tibialiss
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behind and below the medial malleolus, absent in 15% of healthy individuals
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Allen test
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determine arterial insufficiency in the upper extremity by ensuring patency of ulnar artery. Tight fist and compress both radial and ulnar arteries → release pressure over ulnar. Palm will flush in seconds if artery is patent
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Major veins of the lower extremity
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femoral vein, great saphenous vein, small saphenous vein, popliteal vein, communicating vein
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Pitting edema
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increased peripheral venous pressure, depression caused by pressure from thumb
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Non-pitting edema
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stasis of lymph fluid in the tissues
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Orthostatic edema
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pitting, swollen foot, returns to normal upon postural changes
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Lymphedema
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firm, non pitting edema, leathery skin
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Lipedema
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deposition of fat, non pitting, no swelling
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Edema of advance chronic venous insufficiency
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soft pitting with lesions, can become hard, skin thickening and pigmentation, swelling of foot
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Internal rotation
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anterior surface of limb inward
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External rotation
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anterior surface of limb outward
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Supination
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rotation of hand so palmar surface is upward
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Pronation
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rotation of hand so palmar surface faces down
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Abduction
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movement away from midline
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Adduction
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movement towards midline
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Inversion
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plantar surface of foot inward
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Eversion
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planar surface of foot outward
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Biceps tendon reflex
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C5C6
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Triceps tendon reflex
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C6C7C8
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Brachioradialis tendon reflex
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C5C6
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Patella
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L2L3L4
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Achilles
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S1S2
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4+
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brisk, hyperactive, with clonus
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3+
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brisker than average, not indicative of disease
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2+
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average, normal
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1+
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somewhat diminished, lower than average
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0
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reflex absent
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Clonus
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involuntary contractions of muscles between flexion and textion that occurs in conditions of increased tone
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5
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active movement against gravity with full resistance without evident fatigue
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4
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active movement against gravity with some resistance
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3
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active movement against gravity
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2
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cannot act against gravity
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1
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slight contraction detected
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0
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no contraction
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venous thromboembolism
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blood clot or deep vein thrombosis, can lead to pulmonary embolism
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Clinical paramaters of venous thromboembolism
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active cancer, paralysis, pareisis, cast in lower extremities, bedridden, localized tenderness along distribution of deep venous system, entire leg swelling, calf swelling, pitting edema. Collateral superficial veins
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