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12 Cards in this Set

  • Front
  • Back
3 Morphological variants of acute inflammation
1) suppurative inflammation
2) fibrinous inflammation
3) serous inflammation
Bacteria that promotes purulent inflammation
pyogenic
Chemical mediators of arterior dilation
NO, histamine
Chemical mediators of neutrophil activation
leukotriene, chemokines, bacteria
Chemical mediators of epithelial activation
serotonin, histamin, leukotrienes
4 cardinal signs of inflammation
pain, redness, swelling, heat
Outcomes of inflammation determined by (3)
1) severity of tissue damage
2) stem cells w/ in
3) agent causing it
3 possible outcomes of inflammation
1) Resolution
2) Fibrous Healing (scar)
3) chronic inflammation
acute abscess vs. chronic abscess
pus vs. pus encapsulated by granulous/ fibrous tissue
3 cell types in chronic inflammatory infiltrates
-lymphocytes, macrophages, plasma cells
-following non-resolution of acute
-certain injurious agents
Chronic inflammation signs
co-existing tissue destruction
organization/repair
Langhan's giant cells vs. foreign body giant cells
L-horseshoe nuclei
G-similar to surrounding macrophages