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12 Cards in this Set
- Front
- Back
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3 Morphological variants of acute inflammation
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1) suppurative inflammation
2) fibrinous inflammation 3) serous inflammation |
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Bacteria that promotes purulent inflammation
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pyogenic
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Chemical mediators of arterior dilation
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NO, histamine
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Chemical mediators of neutrophil activation
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leukotriene, chemokines, bacteria
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Chemical mediators of epithelial activation
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serotonin, histamin, leukotrienes
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4 cardinal signs of inflammation
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pain, redness, swelling, heat
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Outcomes of inflammation determined by (3)
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1) severity of tissue damage
2) stem cells w/ in 3) agent causing it |
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3 possible outcomes of inflammation
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1) Resolution
2) Fibrous Healing (scar) 3) chronic inflammation |
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acute abscess vs. chronic abscess
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pus vs. pus encapsulated by granulous/ fibrous tissue
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3 cell types in chronic inflammatory infiltrates
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-lymphocytes, macrophages, plasma cells
-following non-resolution of acute -certain injurious agents |
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Chronic inflammation signs
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co-existing tissue destruction
organization/repair |
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Langhan's giant cells vs. foreign body giant cells
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L-horseshoe nuclei
G-similar to surrounding macrophages |
