Hemodynamics

Front 1

Hemorrhage

Back 1

extravasation of blood into the extravascular space

Front 2

Hematoma

Back 2

localized collection of blood outside the blood vessels,[1] usually in liquid form within the tissue; due to hemorrhage

Front 3

Hemorrhagic (hypovolemic) shock

Back 3

Caused by great losses of blood; usually more than 20%

Front 4

Thrombin

Back 4

Thrombin is a "trypsin-like" serine protease protein that in humans is encoded by the F2 gene.[2][3] Prothrombin (coagulation factor II) is proteolytically cleaved to form thrombin in the first step of the coagulation cascade, which ultimately results in the stemming of blood loss. Thrombin in turn acts as a serine protease that converts soluble fibrinogen into insoluble strands of fibrin, as well as catalyzing many other coagulation-related reactions.

Front 5

Virchow's triad

Back 5

Three main factors that induce thrombos formation :
(1) endothelial injury
(2) stasis or turbulent blood flow
(3) hypercoagulability of the blood

Front 6

Aneurysm

Back 6

is a localized, blood-filled balloon-like bulge in the wall of a blood vessel. Leads to a hemorrhage.

Front 7

Embolism/Embolus

Back 7

An embolus is a detached intravascular solid, liquid, or gaseous mass that is carried by the blood to a site distant from its point of origin]-> not all thrombotic in nature, but for the most part should be considered thrombotic (unless otherwise stated).
An embolism (plural embolisms; is the event of lodging of an embolus (a detached intravascular mass capable of clogging arterial capillary beds at a site far from its origin) into a narrow capillary vessel of an arterial bed which causes a blockage (vascular occlusion) in a distant part of the body.
*Traveling clot (traveling thrombosis)

Front 8

Thrombosis

Back 8

Thrombosis is the formation of a blood clot inside a blood vessel, obstructing the flow of blood through the circulatory system. When a blood vessel is injured, the body uses platelets and fibrin to form a blood clot to prevent blood loss. Alternatively, even when a blood vessel is not injured, blood clots may form in the body if the proper conditions present themselves. If the clotting is too severe and the clot breaks free, the traveling clot is now known as an embolus.

Front 9

Vascular occlusion

Back 9

Sudden blockage of a blood vessel, usually with a clot, but may be any blockage.
*can be performed medically in order to reduce pressure on aneurysm, restrict hemorrhage and to reduce nutrient to tumors.

Front 10

Arterial thrombi

Back 10

Arterial thrombi are frequently occlusive; the most common sites in decreasing order of frequency are the coronary, cerebral, and femoral arteries. They typically cosist of a friable meshwork of platelets, fibrin, red cells, and degenerating leukocytes. Although these are usually superimposed on a ruptured atherosclerotic plaque, other vascular injuries (vasculitis, trauma) may be the underlying cause.

Front 11

Venous thrombi

Back 11

Venous thrombosis (phlebothrombosis) is almost invariably occlusive, with the thrombus forming a long cast of the lumen. Because these thrombi form in the sluggish venous circulation, they tend to contain more enmeshed red cells (and relatively few platelets) and are therefore known as red, or stasis, thrombi. The veins of the lower extremities are most commonly involved (90% of cases); however, upper extremities, periprostatic plexus, or the ovarian and periuterine veins can also develop venous thrombi. Under special circumstances, they can also occur in the dural sinuses, portal vein, or hepatic vein.