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45 Cards in this Set
- Front
- Back
Cushing's Triad
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increased systolic pressure
widened pulse pressure decrease in pulse and respiratory rates results from increased intracranial pressure (ICP) |
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contrecoup
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an injury that occurs at a site opposite the side of impact
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coup
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local damage that occurs at the site of impact
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Battle's Sign
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echymossis over the mastoid process caused by fracture of the temporal bone
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cerebral perfusion pressure
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a measure of the amount of blood flow to the brain calculated by subtracting the intracranial pressure from the mean systolic arterial blood pressure
normal range: 70-80mmHg 60mmHg is the critical minimum to adequately perfuse the brain |
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decerebrate posturing
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a position in which a comatose patient's arms are extended and internally rotated and the legs are extended with the feet in forced plantar flexion; usually observed in patient's who have compression of the brainstem
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decorticate posturing
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a position in which the comatose patient's upper extremities are rigidly flexed at the elbows and at the wrists; usually observed in patients who have a lesion in the mesencephalic region of the brain
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epidural hematoma
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accumulation of blood between the dura mater and the cranium
rapid developed lesion (arterial) possible lucid period |
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raccoon's eyes
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ecchymosis of one or both orbits caused by fracture of the base of the sphenoid sinus
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subarachnoid hematoma
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a collection of blood or fluid in the subarachnoid space
sudden and severe headache |
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subdural hematoma
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a collection of blood in the subdural space (dura and surface of the brain) - venous
more common than epidural hematoma acute - symptoms within 24 hours subacute - symptoms between 2-10 days chronic - symptoms after 2 weeks |
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Head injuries - Paramedic should obtain this info
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- Mechanism of injury
- events leading up to injury - time of injury - associated medical problems - allergies - medications - last oral intake |
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dental malocclusion
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misalignment of teeth (often caused by mandibular fractures)
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Le Fort I fracture
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involves the maxilla up to the level of the nasal fossa
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Le Fort II fracture
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involves the nasal bones and medial orbits (generally shaped like a pyramid)
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Le Fort III fracture
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complex fracture in which the facial bones are separated from the cranial bones
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Signs and Symptoms of an eye contusion injury
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- traumatic dilation or constriction of the pupil
- pain - photophobia (abnormal sensitivity to light) - blurred vision - tear-shaped pupil |
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Signs and Symptoms of traumatic hyphema
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- traumatic dilation or, less commonly, constriction of the pupil
- decrease in visual acuity - blood in the anterior chamber (may be visible w/ penlight) |
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Signs and Symptoms of globe or scleral rupture
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- decrease in visual acuity to hand movements or light perception
- lowered intraocular pressure (soft eye) - pupil irregularity - hyphema |
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linear fractures
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seen as straight lines on x-ray film. account for 80% of all fractures to the skull.
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basilar skull fracture
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major impact trauma. signs and symptoms include battle's sign, raccoon's eyes, hemotympanum, and CSF leakage.
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Cranial nerve I (olfactory nerve)
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- loss of smell
- impaired taste (dependent on food aroma) - Hallmark of basilar skull fracture |
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Cranial nerve II (optic nerve)
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- blindness in one or both eyes
- visual field defects |
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Cranial nerve III (oculomotor nerve)
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- ipsilateral, dilated, fixed pupil
- especially compression by the temporal lobe - mimics direct ocular trauma |
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Cranial nerve VII (facial nerve)
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- immediate or delayed facial paralysis
- basilar skull fracture |
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Cranial nerve VIII (auditory nerve)
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- deafness
- basilar skull fracture |
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Mean arterial pressure (MAP)
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diastolic pressure plus 1/3 pulse pressure (systolic - diastolic)
Normal range: 85-95mmHg Maintaining a systolic blood pressure of at least 90mmHg also may help maintain adequate MAP |
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Intracranial pressure (ICP)
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normal range: 10-15mmHg
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Cerebral blood flow
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as ICP approached MAP the gradient for flow decreases and cerebral blood flow decreases.
when ICP increases - CPP decreases as CPP decreases, vessels in the brain dilate - results in increased ICP and further cerebral vasodilation |
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Early signs and symptoms of increased ICP
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headache, nausea and vomiting, altered LOC
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Glasgow Come Scale < or = 8
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Tracheal intubation and ventilatory support is recommended
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neurogenic shock
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- a relatively bradycardic response (e.g., a pulse rate of 80 with a blood pressure of 80mmHg
- Warm and Dry skin - no evidence of significant blood loss or hypovolemia - paralysis and loss of spinal reflexes |
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Cerebrum
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sensory, emotions, willed movement, memory
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cerebellum
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motor coordination, smooth movement
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brainstem
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essential functions include heart rate, respirations, temperature regulation
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The Brain
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- Intracranial volume = 1900cc's in adults
- occupies 80% of the cranial volume - normal blood flow = 800ml/min - O2 consumption = 20% of the total body - Only 2% of total body weight, yet gets 15% of cardiac output |
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Reticular Activating System
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responsible for the state of alertness
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decussation of pyramids
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cross over point for pyramid nerve fiber from the cerebrum down the brain stem to the opposite sides of the body (contralateral physical findings)
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Diffuse Axonal Injury Presentation
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- unconsciousness
- hypertension ( > 140mmHg) - Hyperthermic (104-105 degrees F) - posturing (decorticate or decerebrate) - excessive sweating |
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CO2 is a potent vasodilator
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PaCO2 above 45 torr = cerebral blood vessels DILATE = increased vascular volume = Increased ICP
PaCO2 below 30 torr = cerebral blood vessels CONSTRICT = decreased vascular volume = decreased ICP |
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head injury assessment
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- history/mechanism of injury
- level of consciousness (most important) - AVPU - Glascow Coma Scale q 5 mins - vital signs q 5 mins - Pupil assessment - if unilateral dilation, document which side - motor, sensory, pulse X 4 extremities |
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increasing intracranial pressure signs (initial)
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- decreased LOC/unconscious
- pupils - unilateral/bilateral enlargement,maybe reactive to light - decorticate posturing with pain stimulus - cheyne-stokes breathing |
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increasing intracranial pressure signs (ongoing)
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- unconscious
- pupils - unilateral/bilateral may be reactive to light or fixed - decerebrate posturing with pain stimulus - central neurogenic hyperventitlation |
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increasing intracranial pressure signs (late)
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- unconscious
- pupils - unilateral/bilateral dilation, fixed, non-reactive - flaccid posturing - ataxic (or medullary) breathing |
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head trauma management
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- airway/c-spine
- GCS <9 or takes an oral airway - Intubate! - BVM 100% O2, 12-15 BPM, watch SpO2 - If herniation continues, increase ventilations up to 20BPM - early aggressive hyperventilation may drop PaCO2 too rapidly and may worsen situation - start 1-2 large bore IV's depending on other injuries - monitor BP, do not allow hypotension (SBP < 90) to occur - head bleeds do not cause hypotension. if the BP is dropping the patient is bleeding from somewhere else! - reassess the LOC and GCS q 5 mins |