Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
34 Cards in this Set
- Front
- Back
|
what is the most common gastro-intestinal infection |
gastric infection with heliobacter pylori |
|
|
what appearance does vomited blood have |
coffee grounds |
|
|
what is the definition of gastritis |
inflammation of gastric mucosa |
|
|
what is the definition of chronic gastritis |
presence of chronic mucosal inflammatory changes leading to mucosal atrophy and epithelial metaplasia |
|
|
when present in chronic gastritis where are H. pylori bacteria found |
nested with in mucus layer overlying superficial mucosal epithelium |
|
|
in intestinal metaplasia what is gastric epithelium replaced with |
columnar and golbet cells |
|
|
h. pylori induced proliferation of lymphoid tissue with in gastric mucosa is implicated as a precursor to what |
gastric lymphoma |
|
|
what is hypochlorhydric |
this is the condition of insufficient gastric acid to release vitamin B12 from the protein in food |
|
|
what does chronic gastritis lead to the development of |
peptic ulcer and gastric carcinoma |
|
|
what is the severe erosive form of gastritis that is an important cause of acute gastrointestinal bleeding |
acute gastritis |
|
|
acute gastritis is frequently associated with heave use of what drug |
aspirin |
|
|
where doe peptic ulcers most commonly occur |
duodenum and stomach |
|
|
what is the definition of gastric ulceration |
mucosal alimentary tract breach extending through muscularis into submucosa or deeper |
|
|
how are erosions different from ulcers |
erosions are a breach of the mucosal epithelium only |
|
|
what is the fundamental requisite for peptic ulceration |
mucosal exposure to gastric acid and pepsin |
|
|
what percent of individuals worldwide infected with H.pylori actually develop ulcer |
10-20% |
|
|
what are the gastruduodenal effects of NSAIDS |
acute erosive gastritis, acute gastric ulceration and peptic ulceration |
|
|
synthesis suppression of what is key to NSAID induced peptic ulceration |
mucosal prostoglandin synthesis |
|
|
alcohol, corticosteroids and peronality and psychological stress are possible etiologic factors for what |
peptic ulcers |
|
|
pts with NSAID associated peptic ulcers only have gastritis if they have a co-infection from what |
h. pylori |
|
|
what is the chief complication of peptic ulcers |
bleeding |
|
|
true or false; there is a high frequency of malignant transformation of gastric ulcers |
false; there is is a very rare occurrence |
|
|
what ulcers are associated with extensive burns |
curling ulcers |
|
|
what ulcers are associated with traumatic or surgical injury |
cushing ulcers |
|
|
where are acute stress ulcers found |
anywhere in the stomach |
|
|
what color is the base of a acute stress ulcer stained |
dark brown |
|
|
what is the major risk factor for tumors in the stomach |
chronic gastritis associated with h. pylori infection |
|
|
which stomach polyps are essentially innocuous |
hyperplastic and fundic gland polyps |
|
|
adenomatous polyp leads to a definite risk of harboring what |
adenocarcinoma |
|
|
what is the most common malignant tumor of the stomach |
gastric carcinoma |
|
|
what are the 2 distinct morphological forms of gastric carcinoma |
intestinal and diffuse |
|
|
which morphological appearance in gastric cancer is seen in 50% of gastric carcinomas in the USA |
diffuse |
|
|
what is the major risk factor for intestinal gastric cancer |
environmental factors |
|
|
what is the most important prognostic indicator for cure of gastric carcinoma |
stage of tumor at time of resection |
