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98 Cards in this Set

  • Front
  • Back
Pain distribution: esophagus
Substernal
Pain distribution: duodenum, liver, gallbladder, colon, (hepatic flexure)
RUQ
Pain distribution: stomach
Mid-epigastric
Pain distribution: pancreas
Generalized upper abdominal
Pain distribution: small intestine
Peri-umbilical
Pain distribution: colon (splenic flexure)
Left upper quadrant
Pain distribution: appendix, cecum
Right lower quadrant
Pain distribution: sigmoid colon, rectum
Left Lower Quadrant
the palpatory examination also includes the examination for primary ____ dysfunction and ____ findings.
somatic, viscerosomatic
Sympathetic Innervations: Esophagus
T2-T8
Sympathetic Innervations: Upper GI tract
T5-T9
Sympathetic Innervations: Middle GI tract
T10-T11
Sympathetic Innervations: Lower GI tract
T12-L2
Sympathetic Innervations: Stomach, Liver, Gallbladder, Spleen, Parts of Pancreas & Duo
T5-T9
Sympathetic Innervations: Parts of Pancreas & Duodenum, Jejunum, Ilium, Right Colon
T10-T11
Sympathetic Innervations: Left Colon, Rectum
T12-L2
T lvls for Greater Splanchnic Nerve
T5-T9
T lvls for LesserSPlanchinic Nerve
T10-T11
Synapses at the Celiac Ganglion
Greater Splanchnic Nerve (T5-9)
Synapses at the Superior Mesenteric Ganglion
Lesser Splanchnic Nerve (T10-11)
T lvls for Least Splanchnic Nerve
T12
T lvls for Lumbar Splanchnic Nerve
L1-2
Synapses at the Inferior Mesenteric Ganglia
Least Splanchnic Nerve (T12) and Lumbar Splanchnic Nerve (L1-2)
Innervates the Left Colon and Pelvic Organs
Least Splanchnic Nerve (T12) and Lumbar Splanchnic Nerve (L1-2)
Autonomic that causes Decreased peristalsis
Sympathetic
Autonomic that causes Relaxation of gallbladder and ducts
Sympathetic
Autonomic that causes Increased Vascular Tone leading to decreased oxygen and nutrients to tissues (extreme case bowel angina and ischemic bowel)
Sympathetic
Parasympathetic Innervation of greater curvature of the stomach and pyloric sphincter
Left Vagus CN X
Parasympathetic Innervation of Upper GI tract, Liver, Gallbladder and Right Half of the Colon
Right Vagus CN X
Parasympathetic Innervation of Lower 2/3 of Esophagus
Vagus Nerve CN X
Parasympathetic Innervation of the left colon and pelvis
Pelvic Splanchnic Nerves (S2-4)
Autonomic that causes Increases acid secretion
Parasympathetic
Autonomic that causes Contraction of Gallbladder and Ducts
Parasympathetic
Autonomic that causes Peristalsis (i.e. Sprue and diarrhea)
Parasympathetic
Viscerosomatic reflexes Synapse with____, Results in ___.
internuncial neurons, facilitation
Sympathetic reflexes are found between ___
T1 and L2
Parasympathetic reflexes are found in the ___ and ___ regions
high cervical and Sacral
This relationship may explain the high cervical paravertebral manifestations of the vagal viscero-somatic reflex
After exiting the skull, the vagus nerve interdigitates with a connecting loop between C1 and C2 within the cervical plexus
Parasympathetic of Left half of the transverse colon, descending colon, sigmoid, and rectum
Sacral (S2-S3-S4)
The pelvic diaphragm is innervated by the ___
pudendal nerve (S2-3-4) of the pelvic splanchnic nerves
Upper G.I. sympathetic reflexes: Esophagus
T3 right
Upper G.I. sympathetic reflexes:Stomach
T5-T8 Left
Upper G.I. sympathetic reflexes: Duodenum
T7 -T8 right
The parasympathetic reflex from stomach is the___
high cervical vagal reflex
upper G.I. reflex.
The combination C2 left, T3 right, T5 left and T7 right
Reflex for Small intestine
Bilateral(R>L) T8-10
Reflex for descending colon
L2-3 left
T12 right with tenderness over the tip of the 12th rib right appendix
(anterior Chapman tenderpoint)
The 12th dermatome on the right is the____
appendiceal viscerosomatic reflex
Positional variation of the____ can result in perplexing variability in the clinical presentation of acute appendicitis
appendix
Referred pain patterns are found in the same distribution as ____
viscerosomatic reflexes
discoloration caused by massive nontraumatic ecchymoses in the skin of the lower abdomen and flanks. It results from the infiltration of the extraperitoneal tissues with blood
Grey-Turner’s sign
faintly blue coloration as a result of retroperitoneal bleeding
Cullen’s sign
Effect of osteopathic manipulative treatment on length of stay for pancreatitis: A randomized pilot study RESULTS
Average length of stay 3.5 days shorter in OMT versus control
Which part of the pancreas is NOT reproperitoneal
small part of tail
What is the sympathetic Innervation of the Pancreas?
T5-T9 -> Greater Splanhnic -> Celiac Ganglion
Where are the TART sympathetic pancreas changes?
Paraspinal, Midline collateral ganglia
What is the Viscerosomatic Reflex of the pancreas? What type of dysfunction
T7 on Right or bilateral; non-neutral
Where are the Anterior Chapman’s Reflex for the pancreas?
Lateral to costal cartilage between 7th and 8th ribs on right
Where are the Posterior Chapman’s Reflex for the pancreas?
Between transverse processes of T7-8 on the right
What is the parasympathetic innervation of the pancreas?
Vagus
Path of the pancreas lymph?
Lymph vessels follow blood vessels to the Pancreaticosplenic nodes -> Celiac nodes -> Left Thoracic duct
Where are the pancreas lymph capillaries? Where are there none?
The lymph capillaries commence around the acini and their continuations (exocrine). none in the islets (endocrine)
Why do JSCS on psoas with pancreatitis?
because of the close physical relationship between the psoas and the pancreas, any dysfunction of the psoas can contribute to the pain of pancreatitis
Why do Thoracic/lumbar mobilization important with pancreatitis?
encourage lymph flow by removing myofascial restrictions, removing somatic (myofascial) input to a facilitated segment. It is a myofascial release and/or soft tissue technique
Why do OA decompression/suboccipital release with pancreatitis?
vagus nerve exits via the jugular foramen and the occiput forms one border of this opening
Why do A/P Treatment Diaphragm with pancreatitis?
thoracic diaphragm is the primary mover of lymph through the thoracic duct. It is a myofascial technique
Why do Iliosacral mobilization with pancreatitis?
sacrum is attached to occiput via the dura, so treating the sacrum also treats the OA. Also, treating the sacrum allows for better movement of the pelvic diaphragm, which will encourage lymph flow. It is myofascial/soft tissue technique
____ includes a myriad of disorders that involve inflammatory changes in the gastric mucosa, including erosive gastritis caused by a noxious irritant, reflux gastritis from exposure to bile and pancreatic fluids, hemorrhagic gastritis, infectious gastritis, and gastric mucosal atrophy.
Gastritis
____ refers to a discrete mucosal defect in the portions of the gastrointestinal tract (gastric or duodenal) exposed to acid and pepsin secretion.
Peptic ulcer disease (PUD)
lesser curvature stomach parasympathetic innervation
right vagus
greater curvature & pyloric sphincter stomach parasympathetic innervation
Left vagus
What are the stomach lymph nodes?
Left gastric,Celiac, Sub-pyloric, Right gastro-omental nodes
Impaired stomach lymph flow leads to:
Increased tissue congestion and impaired nutrient absorption from the bowel
Lymph flow from the GI tract & below the diaphragm drain from the small lymphatic channels to the ___ up to the Thoracic Duct (Left)
Cysterna chyli
What are the Sympathetic Dominant Complaints?
Constipation
Abdominal Pain
Flatulence
Distension
What are the Parasympathetic Dominant Complaints?
Nausea and vomiting
Diarrhea
Hypermotility related cramping/pain
Inc. tissue congestion
Impaired Absorption
Inc. risk of pancreatic complications in gallbladder disease/dysfunction
Impaired Lymph Flow
Usually self-limited (85-90%)
Analgesics
IV Fluids
No oral alimentation
Nasogastric suction
OMT
Treatment of Acute Pancreatitis
Lab significant for:
Increased serum amylase
Increased serum lipase
Leukocytosis
Hyperglycemia
Acute Pancreatitis
Put the pancreas at “rest”
Parenteral nutrition should be started in cases of severe pancreatitis that prolongs recovery
Once stable, diet should be progressed as tolerated.
Medical Nutrition Therapy: Acute Pancreatitis
First goal is to optimize nutritional support
Second goal is to decrease pain by minimizing stimulation of exocrine pancreas
Consider pancreatic enzyme replacement
Low fat diet
Medical Nutrition Therapy: Chronic Pancreatitis
What does parasympathetic innervation do to the pancreas?
Increases pancreatic juices rich in enzymes
Stimulates the production of bile
May cause a headache because of interchange with somatic innervation in neck
Peak incidence in sixth decade
More common in males
Less common
Gastric Ulcers
Incidence greatest between ages of 25-75
Equal incidence in males and females
Occur in 6 to 15% of the western population
Duodenal Ulcers
Increased mucosal sensitivity to H+ concentration and alters mucosal barrier
Vasoconstriction
Decreased peristalsis which leads to constipation
Relaxation of gallbladder and ducts (no secretion)
Sympathetics T5-T9
Increased tone leads to
Increased acid secretion
Increased peristalsis which can cause diarrhea
Contraction of gallbladder and ducts (secretion)
Increased Parasympathetics - Vagus (OA, AA, C2) tone
Hypermotility related cramping/pain
Diarrhea
Nausea & vomiting
GI parasympathetic complaints
abdominal pain
constipation
flatulence
distension
GI sympathetic complaints
Osteopathic Considerations for PUD: Sympathetics
Rib Raising
T5-9
T10-11
Chapman’s Reflexes
Collateral (celiac) Ganglia
Osteopathic Considerations for PUD: ParaSympathetics
Vagus Nerve:
OA, AA, C2,
Cranial
Sacrum
Osteopathic Considerations for PUD: Lymph
Thoracic Inlets:
Abdominal Diaphragm via Cervical C3-C5 (phrenic nerve)
Thoracolumbar Junction (diaphragm attachment)
Osteopathic considerations for GERD:Sympathetics
Rib raising: T5-T9
Chapman’s reflexes: collateral ganglia
Osteopathic considerations for GERD: ParaSympathetics
SI joint, OA, OM suture
Osteopathic considerations for GERD: Lymphatics
Thoracic inlets: abdominal diaphragm
Osteopathic considerations for Pancreatitis: Sympathetics
T5-T9, T7 right
Chapman’s reflexes: Anterior: Lateral to costal cartilage between 7th, 8th ribs on right
Posterior- Between transverse processes of T7-8 on the right
Osteopathic considerations for Pancreatitis: ParaSympathetics
OA
Osteopathic considerations for Pancreatitis: Lymph
Pancreaticosplenic nodes -> Celiac nodes
Thoracic inlets: abdominal diaphragm
Thoracolumbar junction (diaphragm attachments)