• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/96

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

96 Cards in this Set

  • Front
  • Back

GERD what is it

backwards flow of gastric secretions into esophagus

GERD causes

transient relaxation or incompetent sphincter, increased stomach pressure



-position, anesthesia, tight clothing, hiatal hernia, CA, NG tube, purging, surgery

GERD s&sx

heartburn post meal, when bending or lying down, sore throat, hoarseness, regurg of sour material

complications of GERD

esophagus strichters, can lead to dysphagia and later Barrett's esophagus

Barrett's esophagus

change in lining of esophagus cells that then leads to an increase for esophageal cancer

GERD tx

antacids, PPI's, H2 blockers


diet- no coffee, alcohol, smolking, reduce fat, no food prior to bed


maintain ideal body wt, small meals

what is a hiatal hernia

part of stomach protrudes through esophageal hiatus of diaphragm into thoracic cavity



normally asymptomatic

hiatal hernia sliding vs paraesophogeal

sliding- gastroesophogeal junction and fundus of stomach slide through esophageal hiatus



paraesophogeal- junction is normal place but part of stomach herniates through, can become strangulated pt may develop bleeding gastritis

hiatal hernia tx

diet and lifestyle chgs, med similar to GERD


surgery


Nissan- fundus is sutured down

function of gallbladder

bile is made in liver and STORED in gallbladder

cholelithiasis

formation of stones in gallbladder

cholecystitis

inflammation of gallbladder

choleangitis

inflammation of bile ducts

why do gallstones form

abnormal bile compostition, inflammation of gallbladder

risk factors for cholelithiasis

female, fair skin, fat, 40


prolonged TPN (bile just sits there and collects)


age, fam hx, native americans, obesity, high cholesterol diets, pregnancy

common s&sx cholelithiasis

early- fullness after fatty meals


RUQ steady pain, n/v


jaundice if blockage

complications of cholelithiasis

pancreatitis, liver dmg, empyema of gallbladder, gangrene gallbladder, intestinal obstruction

chole dx

serum bilirubin levels- elevated bili


CBC- elevation in WBC


amylase & lipase- elevated w/pancreatitis


ultrasound of gallbladder- inflammation & stones

chole tx

lap cholecystectomy


if unable to do surgery can try to dissolve stones with long term meds

lap cholecystectomy

minimally invasive, normally outpatient surgery

liver function (review)

manufactures bld proteins- clotting, immune system, O2 transport


manufactures bile- digestion


stores sugar- forms glycogen


rids harmful substances- drugs/alcohol


breaks down sat fat produces cholesterol

order of events portal HTN to ascites & fluid retention

obstruction of flow bld thru liver


portal HTN thru portal system


ascites


decreased fluid vol (decreased BP)


release of renin by kidneys


increase in aldosterone secretion by glands


retention of Na+ & H2O to increase vol

hepatitis

inflammation of liver due to virus



metabolic function and bile elimination are disrupted

Hepatitis that has a vaccination

A & B

s&sx that relate to all acute hepatitis

malaise, dark urine, anorexia, n/v, jaundice

Hep A/B which is a CHRONIC condition

B!



A is acute lasting about 1-2 months

Hep A transmission

stool


fecal-oral route, contaminated food/water

Hep B transmission

blood and bodily fluids



liver cells damaged by immune response

Hep C transmission

blood and bodily fluids


injection drug users primary factor

Hep B associated delta virus (HDV)

blood and bodily fluids


infects people who already have Hep B

Hep E transmission

fecal oral route


contaminated water in developing nations


rare in US

primary worldwide cause of Hep C

chirrosis

"big one" hepatitis

Hep B!


damages liver through bodily fluids


high risk in healthcare workers, increases risk of liver cancer

disease pattern

incubation --> prodromal --> icteric --> convalescent phase

incubation period w/hep

period after exposure


no symptoms

prodromal phase

flu like symptoms


malaise, anorexia, fatigue, aches, n/v, diarrhea, chills, fever, RUQ pain

icteric phase

5-10 days after prodromal symptoms


jaundice of sclera, elevation of bili levels, light brown or clay colored stool, dark urine

convalescent phase

in uncomplicated cases, symptoms improve spontaneous recovery occurs within 2 wks of jaundice

other types of hepatitis

chronic- cirrhosis, live CA, tx- enzymes elevated


fulminant- rapidly progressive, liver fx w/in 2-3 wks


toxic- alcohol, meds, drugs


hepatobiliary - interruption of normal flow of bile (gall stones)

med/nursing care with hepatitis

prevention- vaccine for A & B


meds, rest, avoid alcohol & substances detoxified in liver

hepatitis dx LFT's

ALT- liver specific


AST- heart and liver


ALP- liver and bones


GGT- rises w/hepatitis


LDH- ldh5 liver specific


serum bilirubin- elevation

med for acute hep C

interferon A (alpha)


to prevent chronic hep

med for chronic hep B

interferon A (alpha)


lamivudine

med for chronic hep C

interferon A (alpha) with ribavirin (rebetol) antiviral

acute hep tx

rest, nutrition, avoid substances filtered by liver (ETOH)

chirrosis

functional liver tissue replaced with scar tissue



end stage chronic liver disease, progressive and irreversible, 10th leading cause of death in US

early chirrosis s&sx

liver enlargement & tenderness


dull ache in RUQ


wt loss, weakness, anorexia, diarrhea, constipation,

later states of chirrosis s&sx

impaired metabolism causing bleeding, ascites, gynecomastia, jaundice, periph edema, low wbc and platelets

complications with chirrosis

portal hypertension


splenomegaly


ascites


esophageal varcies


hepatic encepholopathy


hepatorenal syndrome

hepatic enceohalopathy

increased ammonia levels = decreased brain function

hepatorenal syndrome

renal failure with azotemia (high urea levels)

liver disorder dx lab results

elevated AST, ALT, alkaline phosphate, GGT


CBC & platelets- decrease


prothrombin time- prolonged


decrease Na+, K+, Mg+, phosphate


elevated bilirubin & ammonia


decrease albumin

liver disorder dx tests

abd ultrasound- liver size, nodule, ascites


upper endoscopy


liver biopsy

meds

diuretics


lactulose


vit k


antacids

diet/fluid mgmt

limit protein 60-80g/day with elevated ammonia


otherwise 75-100g/day


high carb, moderate fat

surgical tx

TIPS- go around liver with shunt


balloon tamponade


liver tx


pancreatitis

inflammation of pancrease


low mortality rate 10%

risk factor of pancreatitis

alcohol


gall stones

acute pancreatitis

interstitial- mild form, inflammation & edema of pancreas


necrotizing- inflammation hemorrhage, necrosis of pancreatic tissue

acute cause

unknown!



could be trauma, surgery, tumors, alcohol, gallstones

acute s&sx

severe abd pain


nausea & vomiting


jaundice


hypotension


bleeding

acute pancreatitis complications

renal


ARDS


local necrosis, abscess, pseudocyst

chronic pancreatitis

gradual destruction of pancreas tissue


#1 cause alcohol


ducts are blocked causing inflammation and fibrosis

chronic panc s&sx

recurrent LUQ pain


n/v, wt loss, steatorrhea (fatty stools)

chronic panc complications

malabsporption


pseudocyst


diabetes


panc CA

#1 test for pancreatitis

serum amylase


2-3 times nor

other dx for pancreatitis

serum lipase- elevated for 7-14 days


serum typsinogen- elecated w/acute, decreased w/chronic


ultrasound, CT, ERCP

tx for pancreatitis

NPO then to low fat diet


hydration


pain control


meds

meds for pancreatitis

antibiotics


PPI, antacids, H2 blockers


enzyme supplements


octreotide (sandostatin)- reduces pressure

peritonitis

infection or chemical irritant in peritoneal cavity


bacteria enters normally sterile space

peritonitis s&sx

3rd spacing causes hypotension


"acute abdomen"- board like


elderly- confusion, decrease output, abd complaints

complications of peritonitis

life threatening 40%


abcess


sepsis

dx peritonitis

elevated WBC


abd x-ray shows free air


shows GI perforation

peritonitis tx

broad spectrum anitbiotics


analgesics


surgery- fix perforation


IV fluids


NG

ulcerative colitis

rectum thru colon


anscess leads to scarring= narrows colon

ulcerative colitis s&sx

rectal bleeding


diarrhea (5-10 stools/day)


anemia


malnutrution


LLQ pain



*increased risk of CA* (repeated "injury")

crohn's disease

any segment of bowel- common is iliem and ascending colon


ulcers transmural and lead to fistulas


crohn's s&sx

rectal bleeding, diarrhea, anemia, malnutrition


RLQ pain relieved by defecation


fever, fatigue, malaise, wt loss, anemia

crohn's complications

scarring causing strictures


perforation


hemorrhage


increased risk of CA

dx

colonoscopy, upper GI, stool culture


albumin & folic acid- low due to malabsorption


LFT's may be elevated

medications for bowel disorders

sulfasalazine- abx with topical effect in colon (ulcerative colitis)


corticosteroids, immunisupression, metronidazole (flagyl)


anti-diarrheal

neoplastic disorders of bowel

colon CA 2nd leading cause of cancer death in US

polys

considered pre malignant


tissue mass protruding from wall of bowel


most often in sigmoid and rectum

CDC guidelines

fecal occult blood test at age 50


flexible sigmoidoscopy q 5 yr


colonoscopy q 10 yr

colorectal cancer risks

fam hx


IBD


high fat diet



commonly metastasized to lymph then liver, lungs, brain, kidney

colorectal CA s&sx

none until late stages


bleeding, wt loss, pain, abd mass, anemia

screening

**early detection & intervention**


digital done at age 40


mechanical vs functional bowel obstruction

mechanical- adhesions, tumore, strictures


partial or complete- intusseption, volvulous, foreign body, stricture



functional- neurological or muscular


post surgery, hypokalemia, meds, peritonitis

bowel obstruction s&sx

colicky pain, vomiting, decreased bowel sounds

bowel obstruction complications

hypovolemia


perforation


septic shock

obstruction tx

NG


IV fluids


surgery


early ambulation after surgery

diverticular disease

sacs form in colon mainly sigmoid


diverticulitis is when the colon is inflamed from food getting caught in diverticuli

diverticular dx

abd xray- free air, perforation


barium enema- contraindicated


abd CT or colonoscopy


WBC- shift to left with diverticulitis

diverticular compications

perforation, peritonitis, abscess

s&sx diverticular disease

L side pain


n/v


fever


distension

tx of diverticular disease

meds- abx analgesic



low fiber diet with exacerbation to rest bowel then high fiber diet



no seeds, popcorn



surgery- resction, Hartman (reconnecting to give bowel rest)