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59 Cards in this Set
- Front
- Back
Respiratory acidosis
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PaCO2 up
Ph down |
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Respiratory alkalosis
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PaCO2 down
Ph up |
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Metabolic acidosis
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HCO3 down
Ph down |
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Metabolic alkalosis
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HCO3 up
Ph up |
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Basic disorder
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Ph normal one component normal and one is high or low
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Partially compensated
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Ph abnormal both components high or low
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Fully compensated
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Ph normal both components high or low
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cut off rule
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7.40Ph
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Ph
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7.35-7.45
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Paco2
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35-45 mm Hg
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HCO3
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22-26 mEq/l
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PO2
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80-100 mm Hg
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SAO2
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95%
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causes of respiratory acidosis
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COPD, sedation, head trauma, drug overdose, chest trauma (pneumothorax)CNS disorders, sleep apnea, pulmonary edema
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causes of metabolic acidosis
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diarrhea, lactic acidosis, ketoacidosis, diabetes, starvation, acute alcoholism, hyperkalemia
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causes of Respiratory alkalosis
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anxiety/fear, hypoxia, pain, fever, sepsis, CHF, asthma, ARDS, pulmonary embolus
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causes of metalbolic alkalosis
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Vomiting, N/G suctioning, diurectic, increased antacids, hypokalemia, adminstration of HCO3
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respiratory acidosis
symptoms |
restleness, dizziness, dyspnea, headache, coma, tremors, disorientation
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metabolic acidosis
symptoms |
drowziness, headache, coma, seizures, hyperventilation, hyperkalemia, nausea/vomiting, cardiac dysrhythmias, diarhhea
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respiratory aklalosis
symptoms |
anxiety, lightheadedness, palpations/diaphoresis, tinnitus, nausae/vomiting, chest tightness, cardiac dysrhythmias
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metabolic alkalosis
symptoms |
numbness/tingling, seizures, hypokalemia, hypoventilation, cardiac dysrhythmias, confusion, nervousness
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fluid spacing
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first space- ICF and ECF
second space- abnormal ISF (edema) third space-areas normallly without fluid |
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cations
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postive ions
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anions
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negative ions
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electrolyte functions
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promote neuromuscular function
maintain fluid osmolarity regulate H+ distribution body fluids between compartments |
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Potassium K+
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3.5-5.1
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Magnesium
Phosphate |
Mg+1.5-2.5
PO4 3.5-4.5 |
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Sodium Na+
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135.-145
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Calcium Ca+
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8.5-10
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Anions
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Bicarbonate 20-30
Chloride 96-106 Phosphate 2.8-4.5 Protein 6-8 |
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Potassium
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major ICF cation
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sodium
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major ECF cation
effects H2O dist. ECF& ICF Na pump effects nerve impulse transmission kidneys regualte ECF Na via ADH effect on H2O |
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what happens in low sodium
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aldersterone up
sodium reabsorption H2O reabsorption Blood volume Cardiac output |
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potassium
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main ICF cation
controls osmotic pressure neuromuscular activity(heart) effects skeletal muscle and cardiac excitability regulates ABB effects glucose transport Aldersterone conserves Na & H2O pormotes K excretion |
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Potassium acid base balance
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hyperkalemia=acidosis
hypokalemia=alkalosis |
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digitalis
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hypokalemia can enhance effect of drug causing digitalis toxicity
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sodium/potassium relationship
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inverse in the kidneys factors that cause Na retension(blood loss,increased aldersterone)cause K loss
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hyponatremia S & S
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anorexia, nausea/vomiting, lethargy, CONFUSION, muscle cramps, twitching, seisures
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hyponatremia causes & tx
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diuretics, GI loss, diaphoresis, water excess, hypotonic solutions(slowly to avoid intracanial pressure prob)
Give NS IV & oral Na |
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Hypernatremia S&S
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thirst, hyperpyrexia, dry mouth, lethargy, irratilbility, siezures, hallucinations
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hypernatremia causes & tx
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water loss, hypertonic tube feedings, diabetes insipidus, hyperventalation, diarrhea, renal failure
give IV solutions without NA |
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hypokalemia S&S
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fatigue, anorexia, n/v, muscle weakness, Slow GI motility, paresthesia,
Heart-Flat T waves, ST wave depression, U wave presence, ventricular arryth., PVC's, bradycardia, enhanced digitalis effect |
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hypokalemia causes
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diuretics, diarrhea, vomiting, gastric suction, hyperaldosteronism(renal issue),bulimia, shift form ECF into ICF(effect serumK)
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treat hypokalemia
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Dose 20mEq/l or run of 10mEq/l in 100ml in hour oral/IV potassium no bolus/no IV push must be diluted use pump assess renal function restrict foods high in K renal dialysis |
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hyperkalemia S&S
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muscle weakness, dysrhythmia's, flaccid paralysis, anxiety/irritability, paresthesia, diarrhea, abd. cramping
Tall T-waves, prolung PR inteval, ST depression, loss of Pwave wide QRS |
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Hyperkalemia treat
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20-20% IV Glucose with regular insulin(pushes K into cells) Kayexalate enema, dialysis(if needed)Monitor EKG
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hyperkalemia causes
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insufficient renal function, K shift from ICF to ECF, trauma(burns), hyperglycemia, metabolic acidosis
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hypocalcemia s&s
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diarrhea, numbness,tingling seizures, Increased DTR, tetany, ekg changes, laryngeal stridor
Chvosteks sign Trousseaus sign |
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hypocalcemia causes
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Renal failure
hypoparathyroidism malabsorption pancreatitis |
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Hypocalcemia tx
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administer oral/IV calcium gluconate
Airway patency with laryngeal stridor |
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Hypercalcemia S&S
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muscle weakness, constipation, anorexia, n/v, dysrhythmias, lethargy, bone pain, confusion/memory loss, nephrolithiasis
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PTH: (PARATHYROID HORMONE) relationship to calcium
hypocalcemia |
a ↓Ca ↑PTH→
a 1. ↑ BONE RESORPTION→CA MOVES OUT OF BONE a 2. ↑ GI ABSORPTION a 3 ↑ RENAL REABSORPTION OF Ca |
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balancing factor for calcium
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hypercalcemia causes
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signs of hypercalcemia
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LETHARGY /MUSCLE WEAKNESS /
FATIGUE |
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interventions for hypercalcemia
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PHOSPHATE FACTS
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PHOSPHATE 3.5-4.5 mg/dl
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normal level of phosphate
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HYPOPHOSPHATEMIA
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