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57 Cards in this Set

  • Front
  • Back
Body Fluids
Two compartments
Intracellular fluids (ICF)
Extracellular fluids (ECF)
Interstitial fluid
Intravascular fluid
Composition of Body Fluids
Electrolytes
Ions
Cations
Sodium, potassium, calcium & magnesium
Anions
Chloride, bicarbonate, phosphate
Movement of Body Fluids
Osmosis
Osmotic pressure
Solutions
Hypertonic
Isotonic
hypotonic
Adult Daily Fluids
Fluid gains
Oral fluid 1100-1400cc
Solid food 800-1000cc
Metabolism 300cc

Total Gain= 2200-2700
Fluid losses
Kidneys 1200-1500cc
Skin 500-600cc
Lungs 400cc
GI 100-200cc
Total Losses 2200-2700
Intravenous therapy
Isotonic
Same osmolality as body fluids- ex. Lactated ringers, D5W, NS
Hypotonic
Less osmolality than body fluids-ex. 1/2 NS
Hypertonic
Greater osmolality than body fluids- ex. D5W in LR

Important method to replace fluids & electrolytes

Goes directly into blood stream- not the digestive system
Cation: Sodium
Na 135-145 meq/l
Most plentiful electrolyte in the ECF
Na in ECF accounts for 95% of body’s physiologically active sodium
Main role: control water distribution
Hyponatremia
Na < 135 meq/l
Associated with fluid volume imbalances
Cell swells as H2O is pulled into cell
Diluted ECF
Hyponatremia-Causes
Increased sodium excretion
Excessive diaphoresis
Diuretics: thiazide type
Wound drainage: esp GI
Hyperlipidemia
Renal disease
Inadequate sodium intake
Dilution of serum sodium
Renal failure
excessive ingestion of hypotonic fluids
Freshwater drowning
Hyperglycemia
Congestive heart failure
Hyponatremia Assessment
Weak , rapid pulse
Respiratory arrest
Pulmonary edema
Skeletal muscle weakness
Muscular twitching & seizures
anorexia
Headache
Personality change-lethargy & confusion
Increased motility of GI system
Nausea & vomiting
Abdominal cramping
Hyponatremia Interventions
Monitor systems
If hypovolemia- IV saline infusion
If hypervolemia - diuretics are administered to promote excretion of water rather than sodium
If ADH problem- may give drugs
Instruct patient to increase oral intake
Sodium Food Source
Table salt
Soy sauce
Cottage cheese
American cheese
Milk butter
Ketchup
Mustard
bacon
Snack food
Canned food
Lunch meat
White & whole bread
Hotdogs
Processed food
Cured pork
Hypernatremia
Na>145 meq/l
Gain of sodium
Shrinkage of cells
Excess water loss
Overall sodium excess
Hypernatremia-Causes
Decreased water intake
Increased water loss- fever, hyperventilation, diarrhea, infection, etc
Increased sodium intake
Decreased sodium excretion-renal failure, corticosteroids, Cushing’s syndrome
Hypernatremia- Assessment
Decreased cardiac output
Thirst
Muscle irritability and convulsions
Skeletal muscle weakness
Lethargy, stupor or coma
Elevated body temperature
Increased specific gravity
Decreased urine output
Dry, flaky skin
Restlessness, disorientation
Hypernatremia-Interventions
Monitor systems
If from fluid loss-IV glucose & H2O (D5W)
If from inadequate renal excretion of sodium- diuretics that promote sodium loss
Dialysis if needed
Restrict sodium & fluid intake
Cation:Potassium
The principle cation in the intracellular compartment (98% of K* inside cell)
K* 3.5-5 meq/l
Regulates many metabolic activities & necessary for:
Glycogen deposits in the liver & skeletal muscles
Transmission & conduction of nerve impulses
Cardiac contraction
Potassium- Food Source
Bananas
Avocado
Pork, veal, beef
Fish
Tomatoes
Potatoes
Mushrooms
strawberries
Hypokalemia
K* below 3.5 meq/l
Potentially life threatening because every body system is affected.
80% excreted daily from kidneys
15% lost through bowel
5% is lost through sweat glands
Adults need 40-80meq/l per day
Hypokalemia-Causes
Inappropriate or excessive medications
Increased secretion of aldosterone
Vomiting
Diarrhea
Wound drainage- esp. GI
Prolonged nasogastric suction
Heat-induced diaphoresis
Renal disease
Inadequate potassium intake-NPO
Movement of K* from ECF to ICF
Dilution of serum potassium
Hypokalemia-Assessment
Weak, thready pulse
Peripheral pulse difficult to palpate
ECG changes
Shallow respirations
Anxiety, lethargy, confusion, coma
Decreased GI motility
Nausea, vomiting
Constipation
Abdominal distention
Paralytic ileus
Decreased specific gravity
Increased urine output
Hypokalemia-Interventions
Monitor systems
Monitor electrolyte values
Give IV or PO K* supplement
Institute safety measures for pt with muscle weakness
Instruct patient and family about food choices
Instruct patient and family about foods that are high fiber to prevent constipation
Hyperkalemia
Serum potassium > 5.1meq/l
Hyperkalemia-Causes
Excessive potassium intake
Decreased potassium excretion
Renal failure, adrenal insufficiency, potassium sparing diuretics
Movement of potassium from ICF to ECF
Tissue damage, acidosis, hypercatabolism
Hyperkalemia-Assessment
Irregular heart rate, slow weak pulse
Decreased blood pressure
EKG changes
Dysrhythmias
Profound weakness of muscles
Respiratory failure
Muscle twitches, cramps
Hyperactive bowel, diarrhea
Hyperkalemia-Interventions
Monitor all systems- place on cardiac monitor
Discontinue any potassium supplements
Potassium-restricted diet
Potassium excreting diuretic
Kayexalate
Possible dialysis- monitor renal function
Calcium
8.5-10.0 mg/dl
cation Essential to action potentials & muscle contractions
A cofactor in many enzyme systems
Critical to metabolism
Clotting factor
Required for synaptic release of neuro-transmitters
Two forms of calcium
Ionized
Constitutes 45% of total calcium
Physiologically active form of calcium
Varies directly with plasma albumin concentration
Ph changes vary calcium
Nonionized form
40% is bound to albumin
15% is complexed to anions including phosphate, lactate, citrate & bicarbonate
Calcium-Food Source
Broccoli
Low-fat yogurt
Milk
Rhubarb
Spinach
Green beans
Carrots
Collard greens
Hypocalcemia
< 8.6mg/dl
Causes

Inhibition of calcium absorption in GI tract
Lactose intolerance, renal failure, malnutrition
Increased calcium excretion
Renal failure, diarrhea, wound drainage
Decreased ionized fractions of calcium
Hyperproteinemia, alkalosis, pancreatitis
Immobility, hyperphosphatemia
Hypocalcemia-Assessment
Decreased heart rate & contractility
Cardiac dysrhythmias
Hypotension
Tetany/seizures
Positive Trousseau’s & Chvostek’s sign
Hyperactive bowel
Abdominal cramping & diarrhea
Hypocalcemia-Interventions
Monitor systems esp. cardiac
Oral calcium supplements or IV calcium
Give medications that reduce nerve & muscle excitability
Seizure precautions
Monitor patient for fractures
Instruct patient in high calcium foods
Hypercalcemia-
Causes >10mg/dl

Increased calcium absorption
Excessive oral intake calcium & vitamin D
Decreased calcium excretion
Renal failure, thiazide diuretics
Increased bone resorption of calcium
Malignancy, immobility, hyperthroidism
Hemoconcentration
Dehydration, adrenal insufficiency, lithium
Hypercalcemia-Assessment
Increased heart rate which changes to bradycardia and cardiac arrest
Increased blood pressure
Bounding peripheral pulses
Ineffective respiratory movement
Profound muscle weakness
Increased urine output, renal calculi
Decreased GI motility, anorexia, distention
Hypercalcemia-Interventions
Monitor all systems-cardiac monitor
Discontinue solutions containing Calcium
DC thiazide diuretics
Give IV normal saliene
Give meds to inhibit calcium resorption:
Phosphorus, calcitonin, biphosphonates, aspirin
Magnesium
1.6-2.6 mg/dl cation
Appears in blood stream only in small amounts
Essential for neuromuscular function
Excreted primarily from kidneys
Many uses as a smooth muscle relaxer
Magnesium
Food Sources
Green leafy veg
Avocado
Milk
Potatoes
Peas
Pork
Peanut butter
cauliflower
Hypomagnesemia-Causes
<1.6mg/dl
Insufficient magnesium intake
Diarrhea, celiac disease, crohn’s disease
Increased magnesium secretion
Diuretics, alcoholics, malnutrition
Intracellular movement of magnesium
Sepsis,
Insulin administration, hyperglycemia
Hypomagnesemia-Assessment
EKG changes
Dysrhythmias
Hypertension
Decreased GI motility
Anorexia
Nausea
Abdominal distention
Shallow respirations
Seizures
Tetany
Confusion
Psychosis
Facial twitches
Positive Trousseau
Positive chvostek
Hypomagnesemia-Intervention
Monitor systems
IV or po magnesium supplements
Monitor magnesium level every 12 hours if receiving IV Mg
Hypermagnesemia-Causes
>2.6mg/dl
Increased magnesium intake
Magnesium containing antacids and laxatives such as MOM
IV Mg supplements
Decreased renal excretion of magnesium
Hypermagnesemia-Assessment
Bradycardia
Peripheral vasodilation
Hypotension
Dysrhythmias
ECG changes-wide QRS
Decreased respiratory effort
Diminished or absent tendon reflexes
Skeletal muscle weakness
Drowsiness, lethargy
coma
Hypermagnesemia-Interventions
Monitor systems
Oral & IV Mg containing medications are stopped
Antacids should contain aluminum hydroxide not magnesium hydroxide
Phosphorus-
2.7-4.5 mg/dl anion
Regulates many enzyme actions critical for energy transformations
Excreted primarily in kidney but also stool
Calcium and phosphorus are reciprocal-as one rises the other falls.
Phoshorus-Food Source
Fish
Pork
Beef
Chicken
Organ meats
Nuts
Whole-grain breads & cerals
Hypophosphatemia-Causes
<2.7mg/dl
Insufficient intake
Malnutrition, starvation
Increased phosphorus excretion
Hyperparathyroidism, renal failure, malignancy
Intracellular shift
Hyperglycemia, hyperalimentation,
Respiratory alkalosis
Hypophosphatemia-Assessment
Decreased cardiac output
Slowed peripheral pulses
Reversible cardiomyopathy
Shallow respirations
Weakness
Rhabdomyolysis
Confusion
Seizures
irritability
Decreased bone density
Hypophosphatemia-Intervention
Monitor systems
Administer oral or IV phos
Assess renal system before administering
Move patient carefully
Monitor for fractures
Hyperphosphatemia-Causes
>4.5mg/dl

Remember that when level is high- Calcium is low
Decreased renal excretion
Tumor lysis syndrome
Increased intake
hyperparathyroidism
Hyperphosphatemia-Assessment
Decreased heart rate & contractility
Cardiac dysrhythmias
Hypotension
Tetany/seizures
Positive Trousseau’s & Chvostek’s sign
Hyperactive bowel
Abdominal cramping & diarrhea
Hyperphosphatemia-Intervention
Administer phosphate binding medication such as aluminum hydroxide gel to increased phos excreted in stool
Instruct patient on diet restrictions
Chloride
100-108mg/dl anion

Principle negative ion in the serum
With sodium, maintains osmotic pressure in the serum
Kidneys selectively excrete chloride ions depending on acid-base balance
Hypochloremia Causes
<100
Vomiting
Gastric suction
Diarrhea
Diuretics
Alkalotic states
Hypochloremia-Assessment
Anorexia
Lethargy
Confusion
Muscle twitching
Seizures
Respiratory difficulty
Hypochloremia-Intervention
Monitor system
Seizure precautions
IV fluids
Hyperchloremia-Causes
>108
Increased sodium intake
Renal failure
dehydration
Hyperchloremia-Assessment
Thirst
Elevated body temperature
Weakness
Disorientation
Lethargy, stupor & coma
convulsions
Hyperchloremia-Interventions
Monitor systems
IV fluids
Good oral care