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81 Cards in this Set

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  • Back
Hypotonicity

Abnormal decrease in muscle tone. lesion to LMN

Hypertonicity

Abnormal increase in muscle tone/ resistance to active/passive movement. Lesion to UMN




Hyporflexia

Lack of reflexes: decrease in deep tendon reflexes. Lesion to LMN

Hyperflexia


Too much sensitivity to reflexes. Increase in deep tendon reflexes. Lesion UMN




Spasticity

Form of Hypertonicity: increase muscle tone. Difficulty actively/passively moving spastic muscle groups, either flexor or reflexors (one not both). Lesion UMN

Rigidity (two types)


1. Lead pipe: no movement


2. Cogwheel: little movement


Form of hypertonicity. Difficulty actively &passively moving the muscle groups on both sides of joint. Lesion UMN: result of basal ganglia dysfunction.


Clonus


Uncontrolled oscillation of a muscle that occurs in a spastic muscle group.


Lesion UMN


Praxis ( 3 types)


1. ideational- ability to cognitively understand motor demands.


2. ideomotor planning I- store motor planning abilities- can access it. Ability to plan to access appropriate motor plan


3. Ideomotor planning II- implement appropriate motor plan, or put into action




Apraxia


No motor plan.


Lesion UMN.

Hemiballismus


Damage of basal ganglia


Involuntary hyperkinetic movement involving ipsalateral arm and leg.


Athetosis

Constant writhing movement cause by injury to deeply situated structures caused by lack of oxygen. Damage to Basal Ganglia. Lesion UMN. Ex: cerebral palsy

Chorea

Sudden involuntary jerky movements that involve the axial and proximal limb areas. Damage to Basal Ganglia.

Dystonia


Damage to Basal Ganglia. Lesion UMN.


Repetitive muscle contractions result in abnormal postures. worsen over time

Tardive Dyskinesia


Movement disorder resulting from chronic use of dopamine receptor antagonists. Decrease sensitivity to dopamine receptors. Damage to basal ganglia.



Tremor (2 types)

Tremor- involuntary oscillating movements of muscles.


1. Static (dynamic)- trying to use muscles


2. Resting- involuntary at rest.


No lesion but damage to cerebellum

TICS


Repetitive brief rapid involuntary movement. Purposeless (but ppl feel they have to do it) sound, movements, muscles, meaningful utterances. Waxing/waning: could be there for a week, month, year...


Dopamine involvement; recovery from strep throat.


No lesion areas


Ataxia

Uncoordinated movements due to cerebellar lesions.

Dysmetria (two types)

-inability to judge distance; perceive objects


1. hypermetria: overshoot


2. hypometria: understood
neocerrebellar lesions in posterior lobe



Adiachokinesia


Inability to perform rapid alternating movements.


neocerrebellar lesions in posterior lobe.




Asthenia


Muscle weakness sign of cerebellar damage.


Neocerrebellar lesions in posterior lobe.




Fasciculations


Wavelike contractions on tongue/body.


Presents itself when damage in the LMN

atrophy


Muscle/body deterioration


Sign of severe LMN damage



reflexes


Damage in UMN presents as hyperreflexia.


Damage in LMN presents as hyporeflexia.

Muscle tone


Damage in UMN presents as increased/spastic.


Damage in LMN presents as decreased/flaccid.

Babinkski sign


If present; toes spread; sign of UMN damage.


If absent; toes goes down; LMN damage



Upper Motor Neurons (innervation and origins)


Location of cell body: lie in the motor area of cerebral cortex


Innervation: NIL


Functions: controls activity of LMN


Examples: coritospinal tract



Lower Motor Neurons (innervation and origins)


Location of cell body: lie in grey matter of spinal cord and brain stem


Innervation: skeletal muscles


Functions: controls voluntary movement of skeletal muscles in face and periphery


Examples: spinal nerve & motor cranial nerves.




Kinetic

Movement

Tonic


Tone



Brady
slow

tachy

fast
hypo

low
hyper
High


Presentations of UMN lesions


UMN: Part of CNS


Lesions: neurons who cell bodies in brain & spinal cord; brain stem too.


Presentation of LMN Lesions


Part of PNS


Lesions: neurons who cell bodies are located outside spinal cord &brain



Stroke presentations


Gait disturbances


Drooping face


Visual disturbances


Slurred speech


Confusion

Stroke in basilar artery
Problems with automatic base functions: balance; proprioception, breathing, swallowing

multi-infarct dimentia

Mini strokes in small articles/ capillaries in the brain.

Carotid artery


Right & left.


Anterior supply




Basilar artery


Posterior supply.


Form by right & left vertebral artery joining


Circle of Willis


Posterior communicating artery.


Where most strokes occur. For SLP.


TIA


Transient Ischemic Attack:


Mini stroke


Completely resolve


Symptoms go away


Might not realize occuring


Major arteries


1. Right internal carotid artery


2. Left internal carotid artery


3. Right vertebral artery


4. Left Vertebral artery


3&4 Join to form basilar artery.




Cerebrum houses these arteries


Middle cerebral artery


Anterior cerebral artery


Posterior cerebral artery


Cerebellum houses these arteries


Superior cerebellar artery


Anterior inferior cerebellar artery


Posterior inferior cerebellar artery

Brain stem houses these arteries


pontine branches


Anterior spinal artery




Thrombotic stroke


Profound stroke:


Occurs secondary to clot. Plaque development occluded in vessels and walls.


embolic stroke

Stroke due to plaque becoming dislodged and moves in body/ brain/heart.
Hemorrhagic stroke


Blown up ruptured vessel/ hemorrhage in brain. Usually fatal.




Lacuner infarct


Tiny small clots, subcortical along brain stem. Cognitive function weakness.


Occurs because occlusion of smaller branches of arteries.

LEFT
MCACVA

Left middle cerebral artery- Cerebral Vascular Accident


Right hemiplegia (weakness; motor function)


Right hemiparesthesia (sensation)


Paralysis


Aphasia: compromised language: receptive & expressive.


Lability or depression

RIGHT


MCACVA

Right middle cerebral artery; cerebral vascular accident
Left hemiplegia


Left hemiparesthesia


Aphasia (inconsistent error) Adults: groping & motor speech planning compromise


Affect Flat or Euphoria



PCA Occlusion or PCACVA


Posterior cerebral artery occlusion:


Memory loss


Visual compromise


Perceptual compromise


Visual field cut




ACA occlusion or ACAVCA


Anterior cerebral artery occlusion:


Cognitive change (^) involvement in frontal lobe:


judgement, sequencing, planning


Aphasia


Depression


Emotional liability/euphoria


contralateral hemiplegia


Contralateral hemiparethesia

Spinal Cord Injury: lesion between C1 & C5




UMN signs are present in both upper and lower limbs (hyper/spastic)



Spinal Cord Injury: lesion between C6 & T2


UMN signs are present in lower limbs (spastic)


LMN signs are present in upper limbs (flaccid)

Spinal Cord Injury: lesion between T3 & L3


UMN signs are present in lower limbs.


Upper limbs are normal.




Spinal Cord Injury: Lesion between L4 & S2


LMN signs are present in lower limbs.


Upper limbs are normal.

Name the 5 sections of Spine


Cervical: C1-C7


Thoracic: T1-T12


Lumbar: L1-L5


Sacral: S1-S5


Coccygeal




PICCVA


Posterior Inferior Cerebellar Cerebral vascular accident : occlusion in cerebellum:


Balance


Vertigo


Nausea


nystagmus


Hyper tonic


Hyper flexia








Aiccva


Anterior inferior cerebellar CVA: occlusion in cerebellar:


dysmetria- inability to judge distance


adiodokinesia


Brain stem Occlusions


Breathing


Swallowing


Balance


Arousal/alertness






Endorphins


Neurotransmitter


1. Function: diminishes sensation of pain


2. Deficient: depression, body aches, confrontational


3. Excessive: feel less pain, less negative effects of stress


4. Disorders: chronic fatigue syndrome, runner's high, endorphin deficiency disorder


Serotonin (5-HT)


1.funtion: implicated in sleep, emotional control, pain regulation & carbohydrate feeding behaviors.


2. Deficient: depression, suicidal, aggression, anger, violence, increase carb. Craving


3. Excessive: high fever, irregular heartbeat, unconsciousness, seizures


4. Disorders: Circadian rhythms, depression, OCD, Appetite & eating disorders


Substance P

1. Function: mood regulation, anxiety, respiratory rhythm, pain perception.


2. Deficient: person may feel less pain? Need more research


3. Excessive: interfere with sleep, wired/anxious feeling


4. Disorders: fibromyalgia, PTSD

Dopamine


1. Function: affects motor system; cognition, motivation and reward.


2. Deficient: depression, low drive, motivation, flat, bored, over sleep, gaining weight.


3. Excessive: Parkinson's, Schizophrenia


4. Disorders: Bipolar, battered women syndrome, eating disorders, impulse control. Decline in memory, attention. Paranoia


Glutamine

1. Function: most common neurotransmitter in CNS. Role in learning and memory. Taste of Umami in foods


2. Deficient: insomnia, mental exhaustion, lack of concentration.


3. Excessive: epileptic seizures, excitotoxicity, ADHD, restlessness


4. Disorders: ALS, TBI, Alzheimer


Norepinephrine


1. Function: major role in wakefulness and arousal


2. Deficient: ADHD, Depression


3. Excessive: fear, anxiety panic


Disorders: ADHD

Gamma Aminobutyric Acid (GABA)


1. Function: major inhibitory neurotransmitter of brain


2. Deficient: anxiety disorders, insomnia, epilepsy


3. Excessive: memory loss


4. Disorders: anxiety/panic attacks .

Spastic Dysarthria Secondary to Stroke


-type of speech disorder resulting from bilateral damage. UMN damage both pyramidal /extrapyramidal tracts. Stroke is common cause


-characteristics: articulation, probation, resonance, prosody


-presents: drooling, dysphagia, pseubulbar affect


-movements: slow, spasticity, abnormal reflexes, weakness


Meditation


Effects on brain: increases activation in: anterior consulate cortex, prefrontal cortex, anxiety relief.


On Amygdala: increased emotional stability and response to stress.


Changes in grey matter: increased processing. increase matter in left hippocampus; areas in learning, memory, emotion


Changes in cortical thickness: think in prefrontal cortex & anterior insular: areas in attention, self awareness


Autism


-a mental condition from early childhood. Characterized by difficulty in communicating and forming relationships with other people/ abstract concepts.


Symptoms: nonverbal communication skills lacking. Failure to create friendships, lack on interest, no empathy, delayed talking. No pragmatics. Preoccupation with objects. Stereotyped behaviors (rocking/hand flapping)


Treatment: EI. SLP: play skills. Risperidone/aripiprazole.


Brain: sensory overload, lack facial recognition, less activation in front of brain. Connections from superior temporal sulcus (sts) to Amygdala are activated.. pathways incomplete. Heavier brains

BDD (body dysmorphic disorder)

-a condition marked by excessive pre-occupation with an imaginary or minor defect in a facial feature or localized part of body.


Symptoms: ritualistic behavior (to manage anxiety)


Camouflaging problem: frequent requests for assurance about appearance.


Treatment: CBT, antidepressant meds, selective serotonin reuptake inhibitors (SSRI)


Brain: Lower than normal levels of serotonin. disorder in visual processing (frontostriatal and limbic system)


Schizophrenia

-chronic, severe, and disabling disorder in the brain. psychotic disorder


Causes- genes, environment, brain chemistry, structure


Environment: prenatal exposure to viruses, malnutrition.


Brain chemistry & structure: neurotransmitters: dopamine/ glutamate/ larger ventricles, less grey matter, less/more activity in areas of Brian.


Symptoms: hallucinations, delusions, thought disorders, movement disorders, flat affect, lack of pleasure, lack of ability to being and sustain planned activities, little spoken language.


Cognitive symptoms: poor executive functioning, poor attention, poor working memory,


Treatment: antipsychotic meds, CBT, co-occurring substance abuse.


Selective Mutism

-social anxiety disorder where a whiled who can speak does not speak in certain social situations. Diagnosed before age 5.


Brain: lack of serotonin (dopamine/ norepinephrine). Treated with drugs (Prozac) speech therapy (ritual sound approach)


Cerebral Palsy- Hypoxic Ischemic Encephalopathy (HIE)

-brain injury caused by oxygen deprivation to brain; during pregnancy; during delivery; after delivery. Premature babies at risk.


Symptoms: meconium-stained amniotic fluid, low heart rate, poor muscle tone, weak breathing, bluish/pale skin, excessive acid in blood.


Cause: developmental delays, epilepsy, cognitive issues, motor skill delays; mild/severe.


Treatment: mechanical ventilation, cooling baby brain; hyperbaric chambers, oxygen treatment.


SLP: swallowing, dietary needs, cognitive therapy, advocate, family/team center approach



Alzheimer's disease


-progressive brain disorder that damages and eventually destroys brain cells (beta-amyloid proteins clump and form plaques that destroy cells in brain tissue.


-lead to memory loss, changing in thinking, orientation, speech.


Brain: shrinkage and of larger ventricle spaces due to dying cells. No single test/ cure.

TBI in foot ball


CTE: degenerative disease of the brain and associated with repeated head traumas ; concussions. Not limited to athletes.


Brain: repeated trauma triggers progressive degeneration of brain tissue, including build up of abnormal protein called tau. Atrophy of hippocampus, Amygdala & thalamus.


Symptoms: cognitive impairment, memory loss, emotional instability, depression, aggression. ALS, Parkinson's dementia. Headaches, confusion, nausea/vomiting, blurred vision, fatigue


PTSD


-anxiety disorder that some people develop after seeing or living through traumatic event.


Symptoms: flashbacks, bad dreams, emotional numbness, guilt/ worry, anger.


Brain: hippocampus- damage to brain area involved in memory forming, organizing storing memory. Amygdala: startle response: parasympathetic and sympathetic nervous system reponse and hypothalamic-pituitary-adrenal response to stress. This signals other parts of the brain when danger to send output to hypothalamus. Always in a state of fear alarm, making memories feel like experiencing them in present time.


MS


Multiple sclerosis- autoimmune disease, body's own immune system mistakenly attack normal tissue.


CNS affected, spinal cord and optic nerve.


Symptoms: tingling, numbness, loss of balance, weakness, blurred/double vision, slurred speech, paralysis.


Neurological compromise: vestibular dysfunction, fatigue/pain, spasticity, motor coordination loss, breathing/swallowing difficulties, bladder/bowel issues, mood disorders


Post Concussion Syndrome


-neuropsychological problem that persists for a period of time after a head injury


Symptoms: presence of past TBI, headache, riskiness, fatigue, irritability, insomnia, memory difficulty


Increased risk: history of concussions, being a female, younger age, cognitive dysfunction, affective disorders


Causes: PCS secondary to brain injury/concussion