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81 Cards in this Set
- Front
- Back
Hypotonicity
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Abnormal decrease in muscle tone. lesion to LMN |
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Hypertonicity |
Abnormal increase in muscle tone/ resistance to active/passive movement. Lesion to UMN |
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Hyporflexia |
Lack of reflexes: decrease in deep tendon reflexes. Lesion to LMN |
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Hyperflexia |
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Spasticity |
Form of Hypertonicity: increase muscle tone. Difficulty actively/passively moving spastic muscle groups, either flexor or reflexors (one not both). Lesion UMN |
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Rigidity (two types) |
2. Cogwheel: little movement Form of hypertonicity. Difficulty actively &passively moving the muscle groups on both sides of joint. Lesion UMN: result of basal ganglia dysfunction. |
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Clonus |
Lesion UMN |
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Praxis ( 3 types) |
2. ideomotor planning I- store motor planning abilities- can access it. Ability to plan to access appropriate motor plan 3. Ideomotor planning II- implement appropriate motor plan, or put into action |
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Apraxia |
Lesion UMN. |
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Hemiballismus
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Involuntary hyperkinetic movement involving ipsalateral arm and leg. |
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Athetosis |
Constant writhing movement cause by injury to deeply situated structures caused by lack of oxygen. Damage to Basal Ganglia. Lesion UMN. Ex: cerebral palsy |
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Chorea |
Sudden involuntary jerky movements that involve the axial and proximal limb areas. Damage to Basal Ganglia. |
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Dystonia |
Repetitive muscle contractions result in abnormal postures. worsen over time |
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Tardive Dyskinesia
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Tremor (2 types)
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Tremor- involuntary oscillating movements of muscles. 1. Static (dynamic)- trying to use muscles 2. Resting- involuntary at rest. No lesion but damage to cerebellum |
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TICS
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Dopamine involvement; recovery from strep throat. No lesion areas |
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Ataxia |
Uncoordinated movements due to cerebellar lesions. |
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Dysmetria (two types) |
-inability to judge distance; perceive objects 1. hypermetria: overshoot 2. hypometria: understood |
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Adiachokinesia
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neocerrebellar lesions in posterior lobe. |
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Asthenia |
Neocerrebellar lesions in posterior lobe. |
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Fasciculations |
Presents itself when damage in the LMN |
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atrophy
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Sign of severe LMN damage |
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reflexes
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Damage in LMN presents as hyporeflexia. |
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Muscle tone
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Damage in LMN presents as decreased/flaccid. |
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Babinkski sign
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If absent; toes goes down; LMN damage |
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Upper Motor Neurons (innervation and origins)
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Innervation: NIL Functions: controls activity of LMN Examples: coritospinal tract |
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Lower Motor Neurons (innervation and origins)
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Innervation: skeletal muscles Functions: controls voluntary movement of skeletal muscles in face and periphery Examples: spinal nerve & motor cranial nerves. |
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Kinetic |
Movement |
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Tonic |
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Brady
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slow
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tachy |
fast |
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hypo
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low |
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hyper
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High
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Presentations of UMN lesions |
Lesions: neurons who cell bodies in brain & spinal cord; brain stem too. |
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Presentation of LMN Lesions |
Lesions: neurons who cell bodies are located outside spinal cord &brain |
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Stroke presentations
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Drooping face Visual disturbances Slurred speech Confusion |
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Stroke in basilar artery
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Problems with automatic base functions: balance; proprioception, breathing, swallowing
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multi-infarct dimentia |
Mini strokes in small articles/ capillaries in the brain. |
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Carotid artery |
Anterior supply |
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Basilar artery |
Form by right & left vertebral artery joining |
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Circle of Willis |
Where most strokes occur. For SLP. |
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TIA |
Mini stroke Completely resolve Symptoms go away Might not realize occuring |
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Major arteries |
2. Left internal carotid artery 3. Right vertebral artery 4. Left Vertebral artery 3&4 Join to form basilar artery. |
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Cerebrum houses these arteries |
Anterior cerebral artery Posterior cerebral artery |
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Cerebellum houses these arteries |
Anterior inferior cerebellar artery Posterior inferior cerebellar artery |
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Brain stem houses these arteries
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Anterior spinal artery |
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Thrombotic stroke |
Occurs secondary to clot. Plaque development occluded in vessels and walls. |
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embolic stroke |
Stroke due to plaque becoming dislodged and moves in body/ brain/heart. |
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Hemorrhagic stroke
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Lacuner infarct |
Occurs because occlusion of smaller branches of arteries. |
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LEFT
MCACVA |
Left middle cerebral artery- Cerebral Vascular Accident Right hemiplegia (weakness; motor function) Right hemiparesthesia (sensation) Paralysis Aphasia: compromised language: receptive & expressive. Lability or depression |
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RIGHT MCACVA |
Right middle cerebral artery; cerebral vascular accident Left hemiparesthesia Aphasia (inconsistent error) Adults: groping & motor speech planning compromise Affect Flat or Euphoria |
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PCA Occlusion or PCACVA
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Memory loss Visual compromise Perceptual compromise Visual field cut |
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ACA occlusion or ACAVCA |
Cognitive change (^) involvement in frontal lobe: judgement, sequencing, planning Aphasia Depression Emotional liability/euphoria contralateral hemiplegia Contralateral hemiparethesia |
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Spinal Cord Injury: lesion between C1 & C5 |
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Spinal Cord Injury: lesion between C6 & T2
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LMN signs are present in upper limbs (flaccid) |
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Spinal Cord Injury: lesion between T3 & L3
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Upper limbs are normal. |
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Spinal Cord Injury: Lesion between L4 & S2 |
Upper limbs are normal. |
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Name the 5 sections of Spine
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Thoracic: T1-T12 Lumbar: L1-L5 Sacral: S1-S5 Coccygeal |
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PICCVA |
Balance Vertigo Nausea nystagmus Hyper tonic Hyper flexia |
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Aiccva |
dysmetria- inability to judge distance adiodokinesia |
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Brain stem Occlusions |
Swallowing Balance Arousal/alertness |
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Endorphins |
1. Function: diminishes sensation of pain 2. Deficient: depression, body aches, confrontational 3. Excessive: feel less pain, less negative effects of stress 4. Disorders: chronic fatigue syndrome, runner's high, endorphin deficiency disorder |
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Serotonin (5-HT) |
2. Deficient: depression, suicidal, aggression, anger, violence, increase carb. Craving 3. Excessive: high fever, irregular heartbeat, unconsciousness, seizures 4. Disorders: Circadian rhythms, depression, OCD, Appetite & eating disorders |
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Substance P |
1. Function: mood regulation, anxiety, respiratory rhythm, pain perception. 2. Deficient: person may feel less pain? Need more research 3. Excessive: interfere with sleep, wired/anxious feeling 4. Disorders: fibromyalgia, PTSD |
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Dopamine |
2. Deficient: depression, low drive, motivation, flat, bored, over sleep, gaining weight. 3. Excessive: Parkinson's, Schizophrenia 4. Disorders: Bipolar, battered women syndrome, eating disorders, impulse control. Decline in memory, attention. Paranoia |
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Glutamine
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1. Function: most common neurotransmitter in CNS. Role in learning and memory. Taste of Umami in foods 2. Deficient: insomnia, mental exhaustion, lack of concentration. 3. Excessive: epileptic seizures, excitotoxicity, ADHD, restlessness 4. Disorders: ALS, TBI, Alzheimer |
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Norepinephrine |
2. Deficient: ADHD, Depression 3. Excessive: fear, anxiety panic Disorders: ADHD |
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Gamma Aminobutyric Acid (GABA)
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2. Deficient: anxiety disorders, insomnia, epilepsy 3. Excessive: memory loss 4. Disorders: anxiety/panic attacks . |
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Spastic Dysarthria Secondary to Stroke
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-characteristics: articulation, probation, resonance, prosody -presents: drooling, dysphagia, pseubulbar affect -movements: slow, spasticity, abnormal reflexes, weakness |
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Meditation |
On Amygdala: increased emotional stability and response to stress. Changes in grey matter: increased processing. increase matter in left hippocampus; areas in learning, memory, emotion Changes in cortical thickness: think in prefrontal cortex & anterior insular: areas in attention, self awareness |
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Autism |
Symptoms: nonverbal communication skills lacking. Failure to create friendships, lack on interest, no empathy, delayed talking. No pragmatics. Preoccupation with objects. Stereotyped behaviors (rocking/hand flapping) Treatment: EI. SLP: play skills. Risperidone/aripiprazole. Brain: sensory overload, lack facial recognition, less activation in front of brain. Connections from superior temporal sulcus (sts) to Amygdala are activated.. pathways incomplete. Heavier brains |
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BDD (body dysmorphic disorder)
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-a condition marked by excessive pre-occupation with an imaginary or minor defect in a facial feature or localized part of body. Symptoms: ritualistic behavior (to manage anxiety) Camouflaging problem: frequent requests for assurance about appearance. Treatment: CBT, antidepressant meds, selective serotonin reuptake inhibitors (SSRI) Brain: Lower than normal levels of serotonin. disorder in visual processing (frontostriatal and limbic system) |
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Schizophrenia |
-chronic, severe, and disabling disorder in the brain. psychotic disorder Causes- genes, environment, brain chemistry, structure Environment: prenatal exposure to viruses, malnutrition. Brain chemistry & structure: neurotransmitters: dopamine/ glutamate/ larger ventricles, less grey matter, less/more activity in areas of Brian. Symptoms: hallucinations, delusions, thought disorders, movement disorders, flat affect, lack of pleasure, lack of ability to being and sustain planned activities, little spoken language. Cognitive symptoms: poor executive functioning, poor attention, poor working memory, Treatment: antipsychotic meds, CBT, co-occurring substance abuse. |
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Selective Mutism |
-social anxiety disorder where a whiled who can speak does not speak in certain social situations. Diagnosed before age 5. Brain: lack of serotonin (dopamine/ norepinephrine). Treated with drugs (Prozac) speech therapy (ritual sound approach) |
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Cerebral Palsy- Hypoxic Ischemic Encephalopathy (HIE) |
-brain injury caused by oxygen deprivation to brain; during pregnancy; during delivery; after delivery. Premature babies at risk. Symptoms: meconium-stained amniotic fluid, low heart rate, poor muscle tone, weak breathing, bluish/pale skin, excessive acid in blood. Cause: developmental delays, epilepsy, cognitive issues, motor skill delays; mild/severe. Treatment: mechanical ventilation, cooling baby brain; hyperbaric chambers, oxygen treatment. SLP: swallowing, dietary needs, cognitive therapy, advocate, family/team center approach |
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Alzheimer's disease |
-lead to memory loss, changing in thinking, orientation, speech. Brain: shrinkage and of larger ventricle spaces due to dying cells. No single test/ cure. |
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TBI in foot ball
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Brain: repeated trauma triggers progressive degeneration of brain tissue, including build up of abnormal protein called tau. Atrophy of hippocampus, Amygdala & thalamus. Symptoms: cognitive impairment, memory loss, emotional instability, depression, aggression. ALS, Parkinson's dementia. Headaches, confusion, nausea/vomiting, blurred vision, fatigue |
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PTSD |
Symptoms: flashbacks, bad dreams, emotional numbness, guilt/ worry, anger. Brain: hippocampus- damage to brain area involved in memory forming, organizing storing memory. Amygdala: startle response: parasympathetic and sympathetic nervous system reponse and hypothalamic-pituitary-adrenal response to stress. This signals other parts of the brain when danger to send output to hypothalamus. Always in a state of fear alarm, making memories feel like experiencing them in present time. |
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MS |
CNS affected, spinal cord and optic nerve. Symptoms: tingling, numbness, loss of balance, weakness, blurred/double vision, slurred speech, paralysis. Neurological compromise: vestibular dysfunction, fatigue/pain, spasticity, motor coordination loss, breathing/swallowing difficulties, bladder/bowel issues, mood disorders |
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Post Concussion Syndrome |
Symptoms: presence of past TBI, headache, riskiness, fatigue, irritability, insomnia, memory difficulty Increased risk: history of concussions, being a female, younger age, cognitive dysfunction, affective disorders Causes: PCS secondary to brain injury/concussion |