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122 Cards in this Set
- Front
- Back
BP procedure
always record ? numbers and round up to ? feet? cuff should circle at least? inflate cuff to ? release at rate of ? |
even numbers and round up to nearest 2mmHg
flat 2/3 of arm (arm at heart level, supported) 20mmhg above estimates SBP 2-5mmHg/secs |
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SBP is indirect measure of
DBP is indirect measure of |
Cardiac output
TPR-total peripheral resistance |
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Pulse pressure equation
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SBP-DBP (big minus small)
During exercise, SBP increases while DBP will either decrease or remain the same |
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DIRECT measure of Mean arterial pressure equation
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MAP=CO x TPR
(CO=HRxSV) (TPR=total peripheral resistance) |
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INDIRECT MAP measurement equation if HR is above or below 100 BPM
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HR<100bpm
• MAP = (1/3(SBP-DBP))+DBP HR > 100 bpm • MAP= (1/2(SBP-DBP))+DBP |
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Cardiac output = (2 equations)
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CO = HR x SV = VO2/ A-VO2 Diff
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rate pressure product
(aka rate pulse pressure equaton) |
HR x SBP/100 (heat beats=systolicbp)
The indirect measurement of myocardial oxygen consumption |
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be sure to start at ? when counting HR
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ZERO at first beat then one t next
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fick equation
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VO2 (maximal oxygen consumption) = CO x (a-v) O2 difference
(VO2 increases with exercise; (a-v) O2 also increases with exercise) |
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Changes in CO with body position to maintain CONSTANT CO.... standing?? and supine??? effect on HR and SV??
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(CO = HR x SV)
Supine/resting = highest SV, and lowest HR (Blood located thoracicly!) Standing = blood flow to the whole body so HR increases and SV decreases |
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Ankle brachial index used to test ?
taken at? ratio of? what # is normal? if abi drops with exercise then? |
severity of PVD (peripheral vascular disease)
posterior tibial artery or dorsalis pedis artery ankle SBP/Brachial SBP 1-1.1=normal they have PVD |
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Hemoglobin (men and women #)
Hematocrit (men and women #) fasting glucose # kidney tests (name) livertest (name) |
M 13.7-17 F 11.5-15.5
M 40-52 F 36-48 60-99 BUN- blood urea nitrogen,creatine bilirubin,SGOT- serum glutamic oxaloacetic transaminase, SGPT - serum glutamic - pyruvic transamimase |
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Sources of Injury for arteries
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o Dyslipidemia (abnormal cholesterol levels)
o Hypertension (HIGH BP) o Immune responses o Smoking o Vasoconstrictor substances o Viral/ bacterial infection o Homocysteine (byproduct of red meat) o High levels of glucose o Loss of estrogen |
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CAD Risk Factors (non-modifiable)
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o Advanced age (INC # free radicals)
o Male gender o Family history of CVD/MI/Revascularization -Male 1st degree relative < 55 years -Female 1st degree relative < 65 years |
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CAD Risk Factors (modifiable – can change with behavior/lifestyle choices)
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o Hypertension (HIGH BP)
o Dyslipidemia (abnormal lipid levels) o HDL < 40 mg/dL o Obesity o Impaired fasting glucose (100-125 mg/dL) o Smoking o Physical inactivity/Sedentary lifestyle |
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Emerging Risk Factors (no-conclusive evidence/correlation)***
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o Triglycerides
o Lipoprotein (a) o Lipoprotein remnants o HDL subspecies o Apolipoproteins o Total cholesterol to HDL ratio o Homocysteine o Inflammatory markers (C reactive proteins) o Thrombogenic and hemostatic factors |
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Classification of Total Serum Cholesterol Levels
3 |
Total Cholesterol
< 200 mg/dL Desirable 200-239 mg/dL Borderline High > 240 mg/dL High |
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• Classification of Serum Low (LDL) and High (HDL) Cholesterol***
5 1 |
LDL Cholesterol
< 100 mg/dL Optimal 100 - 129 mg/dL Near optimal 130 - 159 mg/dL Borderline high 160 - 189 mg/dL High > 190 mg/dL Very high HDL Cholesterol < 40 mg/dL Low |
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Classification of Fasting Serum Triglyceride Levels***
4 |
Triglyceride
< 150 mg/dL Normal 150 - 199 mg/dL Borderline high 200 - 499 mg/dL High > 500 mg/dL Very high |
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Blood Pressure***
4 |
Systolic Diastolic
Optimal <120 and <80 Prehypertension 120-139 or 80-89 HYPERTENSION Stage 1 140-159 or 90-99 Stage 2 > 160 or > 100 |
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Treatment for Hypertension***
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o Reduce dietary fat
o Increase physical activity Decrease BP as well as risk for CVD o Lose weight if overweight o Limit alcohol intake to no more than 1 oz. a day o Reduce sodium intake (<2.3 grams Na or <6 grams NaCl) o maintain electrolyte balance o Stop smoking o Reduce saturated fat and cholesterol |
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• Smoking***
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o Causes vasoconstriction
o Increases platelet aggregation o Promotes oxidation of LDL o Decreases lung function |
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• Additional Actions of Smoking***
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o Promotes oxidation of LDL-c
o Increases sympathetic nerve activity and catecholamine release resulting in: Increase BP by 5-10 mmHg Increase HR by 10-15 bpm Increase myocardial contractility Constriction of coronary arteries |
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Type I Diabetes
called? Caused by? They need? |
o Insulin dependent diabetes mellitus (IDDM)
Used to be called juvenile onset o An autoimmune disease affecting the beta cells of the islet of Langerhans of the pancreas, in which the body doesn’t produce insulin o These patients require insulin injections to stay alive o Comprises 5-10% of diabetic patients |
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Type II Diabetes
type of disease? prevalence? |
o Non-insulin dependent diabetes mellitus (NIDDM)
Used to be called adult onset o A metabolic disorder in which the body can’t make enough insulin (insufficiency) or can’t properly use it (insensitivity) o 90-95% of diabetic patients |
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Gestational Diabetes
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o Happens during pregnancy (men can’t get it)
o Associated with a 40-60% chance of developing type II diabetes within the next 5-10 years |
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Diabetes Diagnostic Criteria (KNOW #s or else!)
o Glycosylated hemoglobin (A1c) value of ??% or higher o Confirmed fasting blood glucose of > ??? mg/dL o Confirmed blood glucose of > ??? mg/dL in response to an oral glucose tolerance test (OGTT) using ??g of glucose o Confirmed non-fasting blood glucose of > ???mg/dL in the presence of symptoms of ? |
o Glycosylated hemoglobin (A1c) value of 6.5% or higher
o Confirmed fasting blood glucose of > 126 mg/dL o Confirmed blood glucose of > 200 mg/dL in response to an oral glucose tolerance test (OGTT) using 75 g of glucose o Confirmed non-fasting blood glucose of > 200 mg/dL in the presence of symptoms of diabetes (polyuria, polydipsia, unexplained weight loss) |
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---Pre-Diabetes: Impaired Glucose Tolerance and Impaired Fasting Glucose
o HbA1c of ??-??% o IGT is defined as blood glucose of ???-??? mg/dL in response to an OGTT o IFG is defined as fasting blood glucose of ???-??? mg/dL*** A major risk factor for ? Present in ? million Americans Especially prevalent in ? |
o HbA1c of 5.7-6.4%
o IGT is defined as blood glucose of 140-199 mg/dL in response to an OGTT o IFG is defined as fasting blood glucose of 100-125 mg/dL*** A major risk factor for NIDDM Present in 15-20 million Americans Especially prevalent in elderly |
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Risk Factor Management
focus should be? what is almost as important as glycemic control? what is an importation component in management? |
o Management of identifiable risk factors should be the focus in treatment of diabetes
o Recent case studies suggest control of hypertension is as important as glycemic control o Regular physical activity is an important component in management of diabetes |
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Obesity
ACSM/NIH BMI criteria: • Normal • Overweight -----Obesity o Class I o Class II o Extreme |
• Normal 18.5 - 24.9 kg/m2
• Overweight 25.0 - 29.9 kg/m2 ------ Obesity o Class I 30.0 - 34.9 kg/m2 o Class II 35.0 - 39.9 kg/m2 o Extreme > 40 kg/m2 |
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BMI equation?
inches to cm? pounds to kg? |
BMI=Kg/M^2
times inches by 2.54 divided lbs by 2.2 |
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Obesity Definitions - Waist circumferences:
men > ?? inches women > ?? inches |
men > 40 inches
women > 35 inches |
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• Health Risks Associated with Abdominal Obesity
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o Glucose intolerance and insulin resistance
o NIDDM o Impaired fibrinolysis and increased coagulation (blood clotting) o Dyslipidemia o Hypertension o Macro vascular disease |
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• New Strategies in Treatment of Obesity
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o Normalization of body weight and body fat may not be realistic goals
o Small body weight and fat losses may produce significant improvements in risk factors o Emphasis should be on metabolic health through reduction of CAD risk factors o Improved cardiorespiratory fitness is also beneficial; it may independently reduce risk of CAD o Increase physical activity, reduce dietary fat (<30% of total calories) o Begin with small, gradual weight loss (1-2 lbs per week) initial goal of 5-10% weight loss o Focus on that goal for 6 months before attempting further loss |
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-Metabolic Syndrome
6 factors and numbers? need how many? |
o Abdominal obesity (M > 40 in/102 cm; W > 35 in/88cm)
o Triglycerides (> 150 mg/dL = Borderline High) o HDL cholesterol (men < 40 mg/dL, women < 50 mg/dL) o BP (>130/85 mmHg = Pre-hypertensive) o Fasting glucose (> 100 mg/dL = Pre-diabetes) o Prothrombotic (fibrinogen > 350 mg/dL) and pro-inflammatory (C-reactive protein > 3.0 mg/L) **This is not diabetes (metabolic disease) Need 3/6 |
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=Selected Blood Variables***
o Hemoglobin (g/dL): men ???-??; women ???-??? o Hematocrit (% RBC) men ??-??; women ??-?? o Normal Fasting glucose: ??-??mg/dL o Kidney tests: ? o Liver tests: ? |
o Hemoglobin (g/dL): men 13.5-17; women 11.5-15.5
o Hematocrit (% RBC) men 40-52; women 36-48 o Normal Fasting glucose: 60-99 mg/dL o Kidney tests: Blood Urea Nitrogen (BUN) Test; Creatinine o Liver tests: bilirubin; SGOT - Serum Glutamic-Oxaloacetic Transaminase; SGPT - Serum Glutamic-Pyruvic Transaminase |
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• Pre-participation Health Screening
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o Identification and exclusion of individuals with contraindications/exclusions
o Identification of high risk individuals o Identification of persons with disease o Identification of special needs |
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PAR-Q (Physical Activity Readiness Questionnaire)***
what is it? has only what questions? what to see physician? |
o Minimal standard for entry into low-moderate intensity exercise program
o Yes/no questions o If yes is answered to any of the questions the participant needs to see a physician |
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• Informed Consent
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o Purpose and explanation of procedures or test
o Risks and discomfort o Responsibilities of participants o Benefits to be expected –be fasted, dressed properly o Inquiries o Use of medical records (HIPPA) o Freedom of consent o Place to sign |
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• Medical History
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o Medical diagnosis
o Previous findings on physical examination o History of symptoms, allergies o Recent illnesses and surgical procedures o Orthopedic problems o Medications o Other habits o Exercise history o Work history o Family history |
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CVD risk factors how many?
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o There are 8 positive risk factors (bad), and 1 negative risk factor (good)
• Risk factors, very important: o There are 8 total but the least amount you can have is -1 o Don’t just say “dyslipidemia” must explain why: low HDL, high HDL, on BP meds |
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What are the risk factors?
Not criteria just names. |
Age
Family history Smoking Sedentary Obesity Hypertension Dyslipidemia Pre diabetes HDl (only negative risk factor) |
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Risk Factors for CVD
o Age men > ??years; women > ??years |
o Age men > 45 years; women > 55 years
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o Family history (MI, revascularization, death)
Before ??for father or 1st degree male relative Before ??for mother or 1st degree female relative |
Family history (MI, revascularization, death)
Before 55 for father or 1st degree male relative Before 65 for mother or 1st degree female relative |
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?or those who quit within ?months or exposure to ?tobacco
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o Smoking or those who quit within six months or exposure to environmental tobacco (2nd hand smoke)
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Sedentary lifestyle
?min of exercise for ? days per week for ? months |
not participating in at least 30 minutes of moderate intensity exercise (40-60% VO2) on at least 3 days per week for at least 3 months
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Obesity
Waist girth or BMI trumps? |
(BMI > 30 kg/m2 or waist girth men > 40 inches/102 cm; women > 35 inches/88 cm)
**WAIST GIRTH TRUMPS BMI! |
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o Hypertension (not ?):
> ???/?? mmHg or on ? Confirmed on ?+ occasions |
o Hypertension (not prehypertension):
> 140/90 mmHg or on BP medication Confirmed on 2+ occasions |
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o Dyslipidemia:
> ??? mg/dL LDL, on ? medication, or HDL < ?? mg/dL If total cholesterol is only given, use > ??? mg/dL |
Dyslipidemia
> 130 mg/dL LDL, on lipid lowering medication, or HDL < 40 mg/dL If total cholesterol is only given, use > 200 mg/dL |
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o Pre-diabetes (NOT ?):
impaired fasting glucose > ??? mg/dL but less than ???mg/dL; or impaired oral glucose tolerance > ???mg/dL but less than ??? mg/dL confirmed on ? occasions |
Pre-diabetes (NOT diabetes):
impaired fasting glucose > 100 mg/dL but less than 126 mg/dL; or impaired oral glucose tolerance > 140 mg/dL but less than 200 mg/dL confirmed on 2 occasions |
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high serum HDL levels >?
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Negative risk factor if high serum HDL levels (> 60)
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• Other Important Risk Factors
2 |
o Stress
o Type A personality- hostility |
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Major Signs or Symptoms for CV or Pulmonary Disease
- If someone has any then? - 9 total !! KNOW !! |
(angina)
(dyspnea) Dizziness or syncope Orthopnea Ankle edema Palpitation or tachycardia (over 100bpm at rest) Intermitted claudication (PVD) Known heart murmur Unusual fatigue or shortness of breath |
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ACSM RISK
low moderate high |
o Low Risk
Asymptomatic one (or less) CVD risk factor o Moderate Risk Asymptomatic 2 (or greater) CVD risk factors o High risk Individual with signs/symptoms listed or have known cardiovascular, pulmonary, or metabolic disease (diabetes). (must be eval by Dr. prior) |
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ACSM Recommendations for Current Medical Examination and Exercise Testing Prior to Participation
-3 ways (METS,MPH,%VO2max) |
Moderate exercise is 3-6 METS
-1MET=3.5mL/Kg/min 3-4 MPH Less than 60% VO2 max (40-60%) |
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ACSM Recommendations for Current Medical Examination and Exercise Testing Prior to Participation
- recommended at moderate exercise and ? risk -Recommended at vigorous exercise and ? risk |
Moderate exercise and HIGH risk
Vigorous exercise and MODERATE and HIGH risk |
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ACSM Recommendations for Physician’s Supervision of Exercise Tests
-recommended at sub max if ? risk -recommended at max if ? risk |
Sub max if HIGH risk
MAX if MOD OR HIGH risk |
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• Order of Tests
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o Fill out paperwork informed consents, health history, PAR-Q, etc.
o Blood pressure o Blood work o Body composition- height, weight, % fat o Pulmonary function o Aerobic measurements o Strength measurements o Flexibility |
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• When HR is below 100 ????? increases HR***
• When HR is 100+ ???? causes HR to increase!*** |
• When HR is below 100, parasympathetic activity (vagal tone) decreases, which increases HR***
• When HR is 100+, an increase in sympathetic nervous system causes HR to increase!*** |
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How SV increases during exercise?
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-Myocardial contractility
-INC Pre-Load/Frank Starling mechanism= optimal alignment of actin/myosin -Decrease in afterload (pressure in aorta that the heart has to overcome) ‡ increase SV*** |
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CO ? with exercise
VO2 ? with exercise SBP ? with exercise DBP ? or stays the same with exercise MAP ? with exercise PNS ? with exercise SNS ? with exercise ~? bpm during exercise Plateaus ~?% VO2 |
CO increases with exercise ( = HR x SV)
VO2 increase with exercise ( = CO x (a-vO2) difference) Rate pulse pressure (RPP) increases with exercise ( = HR x SBP/100) SBP increases with exercise DBP decreases or stays the same with exercise ( = TPR) MAP increases with exercise ( = TPR x CO) PNS decreases with exercise ( HR < 100) SNS increases with exercise (HR > 100) ~110 bpm during exercise Plateaus ~40-50% VO2 |
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Absolute Submaximal Exercise
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Absolute Submaximal Exercise = same workload in trained vs. untrained states***
• Test an individual, train them for 6 weeks, give them the SAME exact test after training (should be easier for them to complete) |
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o Same Relative Submaximal Exercise
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o Same Relative Submaximal Exercise = trained individuals have larger workloads to reach the same VO2 max percentage as untrained individuals***
• Work out feels the same for both individuals but the trained individual will be tested with more weight or a longer distance because they are more physically fit (want them to be expending just as much as an untrained person) |
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-REST
HR PNS SNS CO VO2 SV SBP DBP RPP |
HR ↓
PNS ↑ SNS ↓ CO - VO2 - SV ↑ SBP - DBP - RPP ↓ |
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-Absolute
HR PNS SNS CO VO2 SV SBP DBP RPP |
HR ↓
PNS ↑ SNS ↓ CO - VO2 - SV ↑ SBP - DBP - RPP ↓ |
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-REST
HR PNS SNS CO VO2 SV SBP DBP RPP |
HR (220 – age)
PNS- SNS- CO↑ VO2↑ SV↑ SBP- DBP- RPP- |
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-REST
HR PNS SNS CO VO2 SV SBP DBP RPP |
HR -
PNS- SNS- CO↑ VO2↑ SV↑ SBP- DBP- RPP- |
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heart blood flow
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1. Blood that has circulated through the body, deoxygenated, enters through the inferior and superior vena cava into the right atrium of the heart.
2. The right atrium contracts and pumps the blood through the tricuspid valve and into the right ventricle. 3. The right ventricle then pumps blood through pulmonary valve into the pulmonary artery*** into the lungs. **Pulmonary artery does not carry oxygenated blood 4. In the lungs, capillaries absorb carbon dioxide from the blood and replace it with oxygen. 5. Oxygenated blood then flows through the pulmonary vein and into the left atrium. 6. Oxygenated blood then pumps through the mitral valve and into the left ventricle. 7. The left side of the heart contracts the strongest to send blood out the left ventricle and through the aortic arch on its way to all parts of the body. At this point, there are a few options for the blood flow…blood can be pumped → • Through the carotid artery and into the brain. • Through the auxiliary arteries and into the arms. • Through the aorta and into the torso and legs. 8. Blood will then move through the arteries, then through capillaries, and then return through the veins. 9. Deoxygenated blood (blood without oxygen) will then return to the heart. 10. The cycle repeats. |
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heart depolarization path
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SA Node→ via intra-atrial septum into Right and Left Atria (Bachman Bundle)→ AV Node→ Bundle of His→ L/R Bundle Branches (Left bundle branch fascicles – septal, anterior, posterior) → Purkinje Fibers→ Myocardial cells
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P wave
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P wave = depolarization of R and L atria
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PR segment
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• Little time lag (PR segment) after P wave between atrial and ventricle depolarization (tenth of a second)
**Time lag important for the atria to be able to empty fully and allow ventricles to fill with blood! |
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QRS complex
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QRS complex = depolarization of ventricles (repolarization of atria)
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T wave
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T wave = repolarization of ventricles
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o EKG paper speed is
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o EKG paper speed is 25 mm/sec (horizontal = time (sec); vertical = milli-volts (mV))
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• Each little box horizontally
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• Each little box horizontally = 0.04 seconds
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• Each big box horizontally
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• Each big box horizontally = 5 little boxes= 0.2 seconds
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• Each little box vertically
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• Each little box vertically = 0.1 mV
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• PR interval
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• PR interval = beginning of P wave to beginning of QRS complex (start of atrial depolarization → start of ventricular depolarization)
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• PR segment
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• PR segment = end of P wave, beginning of QRS complex (end of atrial depolarization → start of ventricular depolarization)
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• ST segment
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• ST segment = end of QRS complex, beginning of T wave (end of ventricular depolarization → start of ventricular repolarization)
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• QT interval
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• QT interval = beginning of QRS complex to end of T wave (start of ventricular depolarization → end of ventricular repolarization)
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• RR interval
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• RR interval = QRS complex to QRS complex (HR!)
**U Wave → Some people have it right after the T wave, not close to the P wave |
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• Calculating HR from EKG***→ COUNT BOXES BETWEEN RR INTERVALS!!
o Small box method = ? / number of small boxes o Large box method = ? / number of large boxes |
• Calculating HR from EKG***
→ COUNT BOXES BETWEEN RR INTERVALS!! o Small box method = 1500 / number of small boxes o Large box method = 300 / number of large boxes o |
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six sec method
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Six second method = 10 × number of beats between the hash marks
• In 6 seconds (2 sets of hash marks) you measure number of beats (starting with 0!), then multiply that number by 10 to get beats per minute • MAKE SURE YOU ALWAYS GET THE MOST BEATS IN THE 6 SECOND INTERVAL YOU PICK |
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• Every 1 second there are big boxes
• Every 3 seconds there’s a (15 big boxes) • 30 big boxes in ? seconds |
• Every 1 second there are 5 big boxes
• Every 3 seconds there’s a hash mark (15 big boxes) • 30 big boxes in 6 seconds |
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R wave
Q wave S wave |
o The first positive deflection in the complex is always the R wave
• Label the Q and S wave based on the R wave o 1st negative deflection BEFORE positive deflection (before an R wave) = Q wave o 1st negative deflection AFTER positive deflection (after R wave) = S wave |
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QS wave
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o ONLY one deflection going down is called a QS wave (\/) memorize***
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RSR'
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o Positive deflection, negative, then positive again = RSR’ (/\/\)
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? electrodes
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10
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? exetrmity leads
what do they measure? what are they? |
o 6 extremity (limb) leads: measure the frontal plane***
• I, II, III, augmented voltage foot, augmented voltage R arm, augmented voltage L arm (aVR, aVL, aVF) • Bipolar extremity leads: I, II, III*** • Unipolar extremity leads: aVR, aVL, aVF*** o “Ground” electrode is opposite aVF on the lower right side of body |
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? chest/ precordial leads: measure ?
names? |
6 chest/ precordial leads: measure in the horizontal plane (cross section picture of the heart)
• V1, V2, V3, V4, V5, and V6 • These are also unipolar leads (so there are a total of 9 unipolar leads) |
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• Lead I =
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• Lead I = LA - RA (aVL - aVR)
one l |
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lead II
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• Lead II = LL - RA (aVF - aVR)***
two ls |
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Lead III
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• Lead III = LL - LA (aVF - aVL)
three ls |
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• Lead aVr
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→ Right Delta Fossa
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Lead aVl
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→ Left Delta Fossa
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Lead aVf
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Left iliac crest/off lower rib
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GROUND
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Right iliac crest/off lower rib
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V1
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• Lead V1 fourth intercostal space to right of sternum
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V2
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• Lead V2 fourth intercostal space left of the sternum
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V3
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• Lead V3 midway between leads V2 and V4
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V4
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• Lead V4 mid-clavicular line, 5th intercostal space
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V5
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• Lead V5 anterior axillary line same level as V4
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V6
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• Lead V6 mid-axillary line, same level as V4
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Transition zones
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(R WAVE PROGRESSION → R wave = S wave) in precordial leads (V1 → V6)
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Axis deviation
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(found in extremity leads)
Left (upper L) Right (lower R) Extreme (upper R) Normal (lower L) |
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Movement in heart
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o Movement in heart
• SA node→ AV node = right to left movement through right atria (away from V1) • Then left to right movement through intraventricular septum (toward V1) • Then RIGHT to LEFT movement from AV node to R and L ventricles • Right to left atrial depolarization is moving away from V1 (toward left ventricle) → will have a large S wave (NEG deflection) • V1 = small R, big S*** • V6 = small Q, big R*** |
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o Normal R wave progression
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o Normal R wave progression is when the R wave slowly gets bigger and S wave gets smaller and smaller until it becomes a Q wave
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• Axis
o Normal: o Right axis deviated: o Left axis deviated: o Extreme axis deviated: • 0 – 90 = • -90 – 0 = • 180 – 90 = |
• Axis
o Normal: 0 to 90 degrees o Right axis deviated: 91 to 180 degrees*** o Left axis deviated: -1 to -90 degrees o Extreme axis deviated: -179 to -91 degrees • 0 – 90 = normal • -90 – 0 = left axis • 180 – 90 = right axis |
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look at leads ? for axid dev
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leads I and aVF
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o Lead I + Lead III =
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o Lead I + Lead III = Lead II (bipolar)***
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o (-) aVR + aVL + aVF =
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o (-) aVR + aVL + aVF = 0 (unipolar)***
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o P wave:
• + in ? • - in ? |
o P wave:
• + in lead II • - in aVR |
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o T wave:
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• Usually follows QRS complex
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o Enlargement:
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o Enlargement: dilation of chamber, caused by volume overload
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o Hypertrophy:
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o Hypertrophy: increase in muscle mass, caused by pressure overload
( sometimes a good thing because it’s in response to exercise, increased CO) |
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o With enlargement or hypertrophy will cause changes in EKG:
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• Increase in duration of waves
• Increase in amplitude of waves • Axis deviation |
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• Right atrial enlargement:
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• Right atrial enlargement: P pulmonale- severe lung disease or tricuspid valve
• Increase in amplitude (> 2.5 mm) of the first portion of the P wave • No change in duration |
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• Left atrial enlargement:
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• Left atrial enlargement: P mitral- mitral valve disease
• Occasionally increased amplitude of second half of P wave • Will definitely see an increase in duration for back half of P wave (> .12 seconds) |
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• Right ventricular hypertrophy:
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• Right ventricular hypertrophy: pulmonary disease*** and congenital heart disease
• Right axis deviation- QRS complex is more negative in lead I • Normal progression in precordial leads is disrupted → “S wave progression” o V1, R wave is larger than S wave o V6, S wave is larger than R wave o If you don’t have normal R wave progression you won’t have transition zone! • More electrically dominant than L ventricle |
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• Left ventricular hypertrophy:
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systemic hypertension and valvular disease
• Will not ask for criteria exactly, but precordial leads: o 1. R wave amplitude in lead V5 or 6 plus the S wave amplitude in lead V1 or 2 greater than 35 mm • Most important (according to book) o 2. R wave amplitude in lead V5 greater than 26 mm*** • Panton likes this one o 3. R wave amplitude in V6 greater than 18 mm o 4. R wave amplitude in lead V6 exceeds the R wave amplitude in V5 • In limb leads: o 1. R wave amplitude in lead aVL exceeds 13 mm • Most important (according to book) o 2. R wave amplitude in lead aVF exceeds 21 mm o 3. R wave amplitude in lead I exceeds 14 mm o 4. R wave amplitude in lead I plus the S wave amplitude in lead III exceeds 25 mm • Basically, you’ll see really high amplitudes in R and S waves of leads overlying left and right ventricles |
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Strain
caused by? will get? What leads tell right or left? |
o With severe ventricular hypertrophy you may see strain
• This is caused by subendocardial ischemia which affects both ventricular depolarization and repolarization • Will get:*** • ST segment depression • T wave inversion • Most evident in leads with tall R waves • Right ventricular strain o V1 and V2 • Left ventricular strain o Leads I, aVL, V5, V6 |