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51 Cards in this Set

  • Front
  • Back
posterior pituitary gland
acts mostly as storage, releases oxytocin and ADH
anterior pituitary and hormones
controlled by hypothalamus,
TSH
prolactin
Adrenocorticotropic ATCH
gonadotropins: LH FSM
GH
Melanoctye SH
TSH
thyroid stimulating,
normal activity of thyroid glands,
thyroid released thyroxin
ATCH
adrenocorticotropic hormone, stimulates adrenal to release glucocorticoids (stress)
gonadotropins: FSH, LH
levels rise during puberty
FSH: stimulates gamete production
LH: ovulation, testorerone
prolactin
females: milk production
males: testosterone
GH
growth hormone, stimulates mitosis and increase in size, targets bone cartilage and muscle
gigantism/acromegaly
pituitary dwarfism
hyper secretion in childhood,proportionally
hyper secretion in adult, large forehead elongated jaw/nose

drawfism: hypo in child
MSH
melanocyte stimulating
-makes melanin (skin pigment)
active during fetal childhood and pregnancy
Posterior pituitary, ADH
anti-directic
concentrations urine
diabetes insipidus
hyposecretion, large urine outputs, massive thirst, dehydration
SIADH
hypersecretion, decrease urine, fluid retention, hypertension, weight gain
oxytocin
females: contractions (positive feedback)
males: ejaculation
thyroid gland
biggest gland,two lobes
thyroid follicle: thyroxin
colloid
parafollicular:
thyroxin
triiodothryonine
thyroid hormones
thyroxin: 4 iodines, produce by follicles
triiodothryonine: target cells convert T4 to T3, targets everything except brain and gland itself
effect BMR, body temp.
myexdema
hypothyroid, musuc swelling
adult
weight gain low bar
endemic goiter
lack of iodine in diet
cretinism
hyposecretion in child, short disproportionate and mental deficits
graves disease
hyperthyroid in adult
antibodies minic TSH, always releasing
-high bar, irregular heartbeat, treated with removal
or radioactive
calcitonin
lowers blood calcium levels, targets skeleton
inhibits osteoclats (chew bone)
stimulates osteoblasts (deposits)
parathyroid gland
increases blood calcium,
hyperparathyroid
bone demineralization, calcium buildup
hypoparathyroid
trauma event caused, prolonged muscle contractions, loss of sensation
adrenal cortex + 3 types
does not store hormones, produced and released as needed
-mineralocorticoids
-Glucocorticoids
mineralocorticoids
regulates extracellular electrolytes, Na, K
aldosterone is most common
aldosterone
conserves sodium, removes K
effect lasts 20 minutes
aldosteronism
hyper secretion
usually due to tumour
excessive thirst, high blood pressure, weight gain, fluid retention
Glucocorticoids
energy metabolism, helps resist stress
normal: active in unfed state, helps maintain glucose
stress: spikes, maintains glucose
cortisol
bursts, highest in morning
forms glucose from fats and protein to provide glucose to brain
Cushing’s Disease
hyper secretion, depresses bone formation, depresses immune system, constant stress
signs: swollen moon face, tendency to bruise
addisons disease
hyposecretion of glucocorticoids and mineralocorticoids, results in weightloss , decreased sodium and glucose, increase potassium, severe dehydration
gonadocorticoids
stimulates onset of puberty in both sexes
androgen
testosterone, converted at target tissues to potent form, male sex hormone, female muslce formation
androgential syndrome
hypersecretion, masculinization, effects females and prepubescent males
adrenal medulla:
release of hormones controlled by sympathetic NS, low level of continual release
secrets stress hormones epinephrine and norepinephrine
catecholamines
epinephrine: heart and metabolic activities, major product, used for allergic reactions
nor: vaso constrictor, help with blood pressure, stops sending blood to non important functions
catecholamine imbalances
not required for life
hypersecretion: increased BMR HR, hypertension, sweating
pineal gland hormone
melatonin: rises and falls during day, response to darkness
functions: timing of puberty, setting circadian rhythm, role in body temp appetite and sleep
SAD:
seasonal affective disorder, milk depression from darkness
pancreas: acinar cells
exo/endocrine functions, secrets to GI, external to body
acinar cells produce juice that help digestion
pancreatic islets:clusters of cells within pancreas that produce hormones
alpha cells:
clusters of cells within pancreas that produce hormones
alpha: glucagon
beta cells:
secret insulin
insulin
lowers blood glucose, secreted as proinsulin the enzyme creates it to insult so that it doesnt act IN the pancreas
cells need glucose transporter that is activated by insulin
stimulates glycogen formation
glucagon
released glucose in blood
targets liver some muscle
fight or flight response
stimulates glycogen breakdown
diabetes mellitus
types 1/2
too high blood glucose
1: defective insulin production due to damage, childhood, glucose is not being taken in
2: adult,change in insulin receptors cuz of decrease in sensitivity
signs and symptoms
poluria
polydipsia
polyphagia
polyuria: excess urination, kidneys excreting peeing glucose
polydipsia: dehydration from peeing
polyphagia: excessive hunger since cells are starving since they cant take in glucose
damage to other tissues
blindness, obesity, kidney damage,heart problems
hypoglycemia
low blood sugar, usually due to excess insulin
anxiety, nervous, weak
decrease brain activity, treated with sugar
ovaries
estrogen: mature repo strucutres,menstral cycle, secondary characterisitics
progesterone: secreted by corpus luteum, helps uterine lining for implantation, highest before period, maintains pregnancy
testis
testosterone: maturation of rep organs, secondary charc
produced by intersistial cells
inhibin: inhibits FSH production when test levels are high
other hormone producing organs
placenta: sustains fetus (estrogen,progesterone)
intestines: controls activty of digestive tract
kidneys: erythropoetin, increase RBC production
heart: atrial peptide
thymus