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15 Cards in this Set

  • Front
  • Back
stimulants
-amphetamine
-cocaine
-methamphetamine
-methylphenidate
-nictoine
hallucinogens and other compounds
-LSD
-Mescaline
-Phenycyclidine and analogs
-Psilocybin
-amphetamine varients
-marijuana
-hash
-THC
-anabolic steroids
Opiods and morphine derivatives
-codeine
-heroin
-methadone
-morphine
-opium
-oxycodone
depressants
-ETOH
-Barbituates
-benzodiazepines
-methqualone
Opioid Overdose
Heroin most common, cut with various agents, 20-30% pure


S/S
-euphoria
-arousable somnolence (nodding)
-nausea
-pinpoint pupils
-coma
-seizures

TX
-Narcan reverses big 3 (respiratory depression, coma, miosis)
Alchoholism
3 contributing factors
1) personality
2) environment
3) addictive nature of the drug

average time to go through 4 stages is 10 years
ETOH metabolism
80-90% of ingested ETOH is absorbed within 30 minutes (20% percent in the stomach, 80% small intestines)

3-5% is excreted unchanged via the lungs and kidneys, the rest is metabolized in the liver to carbon dioxide and water
ETOH neurological disorders
-short term memory deficit
-problems with coordination
-difficulty with concentration and abstraction
Nutritional Deficiency
ETOH decreases appetite and provides empty calories

S/S
-altered immunity
-anorexia
-cardiac dysrhythmias
-irritablity and disorientation
-muscle cramps
-paresthesias
-poor wound healing
-seizures
-tremor and ataxia
ETOH fluid and electrolyte imbalances
urinary output increases over fluid intake due to ETOH blocking ADH hormone.

dehydration and electrolyte imbalances result
ETOH GI disorders

GI hemorrhage
GI Hemorrhage
4 causes
1) gastritis
2) ulcer formation
3) esophageal tear (mallory-weiss syndrome)
4) variceal hemorrhage

most problems due to vomiting

TX
-large bore IV
-fluids
ETOH GI disorders

Cirrhosis
bands of fibrosis (scar tissue) develop and break up the normal structure of the liver, leading to portal hypertension

complications include
-ascities
-splenomegaly
-bleeding esophageal and gastric varices
-hepatic encepalopathy (build up of waste products normally filtered by the liver)
ETOH GI disorders

Acute or Chronic Pancreatitis
origin unknown related to
-activation of pancreatic proenzymes
-obstruction of pancreatic ducts
-stimulation of enzymatic secretion

chronic
-malabsorption (due to gorked enzymes)
-electrolyte imba
-diabetes mellitus
-hemorrhagic pancretitis
-sepsis
-pancreatic abscess
ETOH cardiac and skeletal muscle myopathy
results from direct toxic effect of ETOH and its metabolites.

-intracellular edema
-formation of lipid droplets
-excessive cellular glycogen
-deranged sarcoplasmic reticula and mitochondria
-weakness
-muscle wasting

Heart Muscle
-decreased force of contraction
-dsyrhythmias
-CHF
Immune Suppresion
ETOH supressues bone marrow production of white blood cells

RBCs and platelets production is decreased

impaired macrophage mobilization and mucociliary function in lungs, aspiration and pnuemonia common