Emergency

Front 1

Anticholinergic Side effects

Back 1

Mad as a hatter(delirum, agitation, hallucinations)

Red as a beet(skin flushing)

Dry as a bone(anhidrosis)

Blind as a bat(midriasis)

Full as a flask(urinary retention) decreased BMs

Increased pulse, BP

Front 2

Differential for cholinergic toxicity vs amphetamines/aspirin/serotonin syndrome

Back 2

Mental status changes

Tachycardia

Hyperexia

on all

cholinergic toxicity has also dry skin, anidrosis, urinary retention

Front 3

Beta blocker overdose

Back 3

Cardiac: hypotension, sinus bradycardia, heart block
Non cardiac:Mental status changescoma, deliriumSeizuresResp depressionHypoglycemiahyperkalemia

Front 4

Destrometrophan toxicity

Back 4

abrupt change in behavior associated with findings of sympathetic stimulation (tachycardia, mydriasis, diaphoresis), gait disturbance, and euphoria or hallucinations.

If co-ingested with monoamine oxidase inhibitors or other serotonergic agents, the serotonin syndrome may be produced

Front 5

Basillar Skull Fractures

Back 5

hemotympanum, cerebrospinal fluid (CSF) otorrhea or rhinorrhea, periorbital ecchymosis (“raccoon eyes”), and postauricular ecchymoses (Battle sign).

Front 6

MC complication of inhalants

Back 6

Recurrent inhalant use causes damage to multiple organ systems, the most significant of which is the central nervous system.

Front 7

Brain death criteria

Back 7

Pediatric brain death criteria have been established and updated as recently as 2012. There has to be a known and irreversible cause of coma, absence of hypotension, hypothermia, metabolic disturbances, and sedating effects of medication. Two brain death examinations by different examiners, done 12 to 24 hours apart, must confirm the absence of brainstem reflexes including the reflex to breathe.

Front 8

Iron toxicity stages

Back 8

Stage 1 (0-6 hours after ingestion)

Stage 2 (6-12 hours after ingestion)

Stage 3 (12-24 hours after ingestion)

Stage 4 (within 48 hours after ingestion)

Stage 5 (2-8 weeks after ingestion)

Front 9

Superior Vena Cava Syndrome

Back 9

Swelling of the face, neck, or upper chestFacial plethora or cyanosisProminent neck and upper chest veinsCoughDyspneaOrthopneaHoarse voice or stridorChest painWheezingPleural effusionsPericardial effusion

Front 10

scorpion (C sculputalus) poisoning presentation

Back 10

Dysautonomias (most commonly tachycardia and hypertension), hyperthermia, and rhabdomyolysis from muscular hyperactivity, gastrointestinal disturbances, pancreatitis, and eventual multisystem organ failure may arise from C sculpturatus envenomation

Front 11

Digoxin toxicity

Back 11

digoxin typically present with vomiting, bradycardia with atrioventricular heart block, hyperkalemia, and mental status changes.

Front 12

Lithium toxicity SE

Back 12

gastrointestinal upset (nausea, vomiting, and diarrhea), dehydration, tremor, weakness, hyperreflexia, slurred speech, visual disturbances, mental status changes, and even seizures with severe toxicity

Front 13

Calcium channel blockers SE

Back 13

bradycardia and hypotension. Neurologic and respiratory system derangements can occur from calcium channel blocker overdose, but these generally arise secondary to cardiovascular toxicity and shock.

Front 14

Phosphenytoin Toxicity signs

Back 14

Acute ataxia, including truncal ataxia, limb dysmetria, and lateral nystagmus