Ec Reproductive Health

Front 1

Hirshprungs disease absecne of what

Back 1

autonomic parasympathetic ganglion cells

Front 2

HIsrchsprungs is common is what sex

Back 2

male

Front 3

Newborn failure to pass meconimum first 24 hours what DX

Back 3

Hirshprunghs Disease

Front 4

S/S of HIrsprungs

Back 4

failure to pass stook. bile stained vomit, refusal to eat, abdomen distended

Front 5

Hirsphrugns in infancy and childhoold what will you see

Back 5

infancy constipation with explosive diahrria, Childhool Constipation with ribbon like stgools which smell foul, admomen distention, poor nourished child, anemic

Front 6

NX interventions for Hirshprings pre op

Back 6

rehydration,informed conscent, NPO, andno rectal tmep

Front 7

post op Nrsg Interevengtion for Hirshpringus

Back 7

Monitor site, bowel sounds, reheydrate, NG tube, parned education regarding colostomy

Front 8

Hirshprungs Diseas may have temporarily what

Back 8

colostomt

Front 9

Plyoric stenosis rare in who

Back 9

rare in black and asians

Front 10

Pylosris strenosis more common in what sex

Back 10

male

Front 11

s/s pyloric stenosis

Back 11

asymptomatic first 2 weeks of life, 2-4 wks regurgitation,6 week complete obstruction, projectile vomiting after feeding

Front 12

failur to gian olive shaped mass right of unbilicus

Back 12

pyloris stenosis

Front 13

Nursing intervention pre op Pyloris stenosis

Back 13

rehydration

Front 14

Nsg Interventions Post op plyoric stenosos

Back 14

slow feedeing upright position left side if needed

Front 15

cystic fibrosis

Back 15

inherite when both parents are carrier

Front 16

cystic fibrosos what race

Back 16

white both men and woman men, do better

Front 17

cystic fibrosis

Back 17

pulmonary involvement

Front 18

cystic firbrosis sweat increased or decreased

Back 18

increased

Front 19

cystic fibrosis what type of lung sounds

Back 19

non productive cough, wheezing, dyspnea, thick and sticky sputum

Front 20

PKU foods to avoid

Back 20

animal proteins ,meat, fish, eggs, milk, some vegtable proteings such as nuts, legume nutrasweet, aspertaime

Front 21

tay sach disease what must both parents be to have child with disease

Back 21

both parents must be carriers

Front 22

tay sachs dx 1 in how many children will have dx

Back 22

4

Front 23

tay sachs how many out of 4 will be carrier

Back 23

2

Front 24

tay sachs life expectancy

Back 24

5 yrs no cure

Front 25

down syndrom look of children

Back 25

occiptal flat, slanted eyes flat bridge of nose, small mouth, protuding tongue

Front 26

out of 4 pregnancys wiht sickle cell how many will be normal and how many will have trait

Back 26

one will be normal one will have trait if both parents are carriers

Front 27

spina bifida

Back 27

external saccular protrussion of any point along the spinal colum caused by failure of the nerual tube closure

Front 28

surgery goal for spina bifida

Back 28

not to reprair faulty spinal cord but to preserve existing function

Front 29

spina bifida keep sac

Back 29

moist and warm goal is to maintain sac no diaper until after repair and healing

Front 30

post op spina bifida

Back 30

monitor ICP neuro assessment, head circumfrance, infection

Front 31

CHF classic signs

Back 31

tachypena, tachycardia, dyspnea at rest, INCRASED LIVER SIZE VOLUME OVERLOAD, PRESSURE OVERLAOD, SEVERE ANEMAI, SEVERE SESISP, HYPERTHRYROIDISM , ELECTROLYTES IMBALANCE,

Front 32

CHF S/S

Back 32

EDEMA PHERIPHAL

Front 33

TX CHF

Back 33

CONSERVE ENERGY, UP RIGH AND ON SIDE TO SLEEP, PROTUGUESE SLING , FEEDING TAKE UP TO ONE HOUR, TREAT INFECTIONS, MAINTAINE ROOM TEMP NOT TO HOT NOT TO COLD

Front 34

WHY GIVE DIG

Back 34

TO IMPROVE MYOCARDIA CONTRACTILITY, INPROVE OUT PUT AND INCREASE RENAL FUNCTION

Front 35

KYPOKALEMIA INCREASE RISK OF WHAT TOXICITY

Back 35

DIG

Front 36

S/S DIG TOX

Back 36

MUSLCLE SPASMS, TWITCHING

Front 37

ACEINHIBITORS PRODUCE

Back 37

AFTER LOAD AND DECREASESWORK ON HEART TO IMPROVE CARDIAC FUNCTION

Front 38

CAPTON AND VASOTEC USED FOR

Back 38

CHF

Front 39

HEMOPHILLIA A CLASSIC FACTOR

Back 39

8 DEFECT

Front 40

HEMOPHILLIA B CHRISTMAS DISEASE

Back 40

FACTOR 9 DEFECT

Front 41

HEMOPHELLIA WHO IS CARRIER

Back 41

MOTHER AND PASSES TO SON

Front 42

WHAT IS MOST COMMON POSITION FOR TERATOLOGY OF FALLOT

Back 42

KNEE CHEST

Front 43

FACTOR 8

Back 43

HUMAFAC OR KOATE

Front 44

FACTORY 9

Back 44

PROLOX OR KONYNE

Front 45

TRACHEALESPHOGUIAL FISTULA

Back 45

COUGH, CYNOSIS , CHOCKING (3'S)

Front 46

HIP DYSPLAISS WHAT WILL YOU SEE

Back 46

HIP WRINKLNG

Front 47

atropine

Back 47

block vague stimulus resulting in increased heart rate

Front 48

calan (vermapamil)

Back 48

channel blocker will decrease heart rate

Front 49

quinidine

Back 49

an antidysrythmic used for tachyarrythmias

Front 50

hodgikins diseas

Back 50

neoplastic disease that is characterized by prgression enlargement of lymph nodes, spleen, and liver bhy prgressive anemail

Front 51

hirschsprungs dx

Back 51

involves and absence in the autonomic ganglia of the smooth muscle in a section of the colon

Front 52

myelomeningocele

Back 52

sac like protrusion outside the bodyand contains CSF adn mengies

Front 53

meningocele

Back 53

sac containing CSF and may containmengies but not neural tissue

Front 54

cyctole

Back 54

hernia of bladder

Front 55

RIGHT SIDED HEART FAILURE

Back 55

PULMONARY HTN, PULMONARY EMBOLISIM, CHD ( VENTRICAULR SEPTAL DEFECT, PATENT DUCTUS ARTERIOUSUS

Front 56

LEFT HEART FAILURE

Back 56

MYCOCARDIDTISIS, AMLYDISIS