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46 Cards in this Set

  • Front
  • Back
Atrial enlargement: large initial component means?
Right atrial enlargement.
Atrial enlargement: large terminal component means?
Left atrial enlargement.
Atrial enlargement: size of p-wave indicative of atrial enlargement?
> 2.5 mm.
Infarction: what is the pathophysiology of Wellens syndrome?
Stenosis of left anterior descending coronary artery.
Wellens syndrome: findings on EKG?
T-wave inversion in V2-V3.
Infarction: what is the pathophysiology of Brugada syndrome?
Dysfunctional cardiac Na+ channels.
Brugada syndrome: findings on EKG?
RBBB with ST elevation in V1-V3.
ST segment depression: can be indicative of an infarction in which layer of the heart?
Subendocardium (non-Q wave infarction).
ST segment depression: can occur with what drug toxicity?
Digitalis (Salvador Dali's mustache on EKG).
Anterior hemiblock: causes what axis deviation?
Left axis deviation.
Anterior hemiblock: findings on EKG?
Lead I: Q-wave
Lead III: wide/deep S wave
(Q1S3)
Anterior hemiblock: this location of infarction can also cause left axis deviation (careful when reading).
Inferior infarction.
RBBB + left axis deviation + acute anterior MI: most likely diagnosis?
Anterior hemiblock (left anterior descending supplies RBBB).
Posterior hemiblock: causes what axis deviation?
Right axis deviation.
Posterior hemiblock: what are the findings on EKG?
Lead I: wide/deep S wave
Lead III: Q-wave
(S1Q3)
Posterior hemiblock: when associated with RBBB, can progress to what fatal sequelae?
AV blocks.
Intermittent Mobitz: pathophysiology of this pattern?
Two permanently blocked fascicles + one intermittently blocked fascicle (occasionally dropped QRS with regular, punctual p-wave).
Long QT syndrome: QT criteria on EKG?
QT interval longer than 1/2 of the cardiac cycle.
COPD: appearance of the QRS complexes?
Low amplitude.
COPD: which axis deviation?
Right axis deviation.
COPD: pathophysiology of right axis deviation?
RV works against resistance --> RVH --> right atrial deviation.
COPD: what type of rhythm can be seen with this disease?
Multifocal atrial tachycardia (MAT).
COPD: low voltage also seen with what conditions?
Hypothyroidism and chronic constrictive pericarditis.
PE: what are the three EKG findings?
Lead I: large S-wave
Lead III: Large Q-wave
Lead III: T-wave inversion
(S1Q3T3)

Also lead II: ST depression.
PE: which axis deviation can be seen?
Right axis deviation.
PE: T-wave inversion can be seen in what horizontal leads?
V1-V4.
PE: may cause what block?
RBBB (transient or incomplete).
Hyperkalemia: what are the EKG findings?
P-wave: wide and flat
QRS: wide (ventricular depolarization takes longer)
T-wave: peaked
Hypokalemia: what are the EKG findings?
T-wave: flat/inverted
U-wave: prominent in extreme cases

Think of a tent filling up with K+ in hyper- vs. hypokalemia.
Hypokalemia: can initiate which deadly rhythm?
Torsades de Pointes.
Hypokalemia: can enhance toxic effects of what drug?
Digitalis.
Hypercalcemia: what is the EKG finding?
Shortened QT interval.
Hypocalcemia: what is the EKG finding?
Prolonged QT interval.
Digitalis: which foci are exceptionally sensitive to digitalis?
Supraventricular (esp. atrial) foci.
Digitalis: earliest warning signs for digitalis toxicity on EKG?
Premature atrial beats (PABs).
Digitalis: what electrolyte should you worry about before using digitalis?
Potassium (hypokalemia potentiates toxicity of digitalis).
Quinidine: findings on EKG?
P-wave: wide, notched
QRS: widened
ST segment: depressed
QT: prolonged
U-waves: present
Quinidine: what fatal rhythm can result from quinidine toxicity?
Torsades de Pointes.
Pacemaker location: LBBB + RAD on EKG.
Below pulmonic valves (lead I is negative).
Pacemaker location: LBBB + normal axis on EKG.
Mid-inflow tract of right ventricle (lead I/aVF is positive).
Pacemaker location: LBBB + LAD on EKG.
Apex of right ventricle (lead aVF is negative).
External pacemakers: appearance on EKG?
Pacemaker spike: wide with flat end (requires impulse of longer duration than intracardiac pacemakers).
Heart transplant patients: appearance on EKG?
P-waves: two (patient/donor)

Patient's p-wave not conducted past suture line whereas donor p-wave will have QRS complexes following p-wave.
Early repolarization: seen in what population?
Young male athletes.
Early repolarization: appearance on EKG?
ST segment: elevated (1/2 mm) in V5-V6
Rotation: rightward (horizontal plane)
Hyperacute T-waves: most likely diagnosis?
Very acute myocardial infarction.