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28 Cards in this Set
- Front
- Back
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what is disassociation of sensory loss
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epi without proto
or proto without epi seen in spinal cord |
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guillian barre
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1. effects large, spare small
2. => epi = large, so affected. proto not affected 3. large contain motor and sensory , therefore lost effects - large myelinated 4. effect schwann cells, therefore PNS |
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leprosy
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1- affects small non myelinated
2. effects proto not epi |
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syrengiomyelia has three forms name them
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- less severe
- more severe - most severe |
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what do u see in less severe syrengomyelia
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- bilateral proto loss ( pain and temp loss)
- cape districbution across the arms, hands and top of back - lower cervical, and upper thoracic |
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why is the proto effected in less severe syrygeomye
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because they cross in the lower spinal cord
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what does more severe form of syryn involve
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- LMN since cavity opens into ventral grey
- bilateral loss of pin (fast pain ) and temperature, bilateral flacidity and paralysis and etc |
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most severe form
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- UMN below level of cavitation-
- bialt loss of pain an dtemp and bilat flaccid paralysis (bilateral LMN signs ) - bilateral spastic paralysis (UMN signs) below - Ipsilateral horners - 1. superior cervical ganglion 2. lateral grey T1, T2 3. DIT- descending ipsilateral tract- T1 |
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what causes tabes dorsalis
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tertiary syphilis
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what is the result of tabes dorsalis
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- demyline dorsal column (bilateral loss)
- sensory loss, epi loss - blood supply: post spinal provides to ipsi dorsal column , occlusion can give an ipsi rhomberg sign . ( fall to one side ) |
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what is the pathological effect of rhomberg sign
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babinski (no negative - etiehr normal or rhomberg
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what are the three systems that send info to our feet together
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visual, concious proprioception and vestibular
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what is rhhomberg sign
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- pathological sign like
- babinski - no negative - rhomberg |
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what are the three things that need to be sent to our brain inputs for us to stand erect on our two feet
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- visual , vestibular and conceious proprioception
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how will you test for rhomberg sign
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- patient closes eyes
- therefore visual and concious proprioception (epi) absent - patient falls if positive phomberb - does not test cerebellar problems |
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when will he have cerebella problem
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puts feet gether and falls when eyes open = cerebellar probelem
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where does the demyelination occur in the diabetic peripheral neuropathy
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- demylination in the stocking and glove distribution
- so lose sensory and motor in stocking and hand - lose motor and sensory - ie both epi and proto ie bilateral not dissociated sensory loss - feet flapping on the ground |
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no loss of pain and temperature but loss of proprioception
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dissociate sensory loss
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peripheral leiosn
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dorsal column leision
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slow pain is called by what fibers
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C fiebrss
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what is the NT in slow pain
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P substance
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subtantia gelatinosa is the
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nucleus proprius
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proprioception is
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nomrmal and goes to the nucleus proprius
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waht kinda relay is the slow pain
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bilateral and diffuse relay
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what is the pathway of the spinothalamicinput
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relays multiple times in reticulo formation
C fibers > subtantia gelatinosa> nucleus proprius (bilateral) > brain stem> reticular formation (no idrect thalamic input) |
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what are the two types of relays in the slow pain
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- anterior nucleus
- dorsal nucleus |
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anterior nucleus relay
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goes to singulate gyrus
leision = dont feel pain anymore , bilateral |
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medial nucleus dorsal relays
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prefrontal lobe
- patient feels pain but doesnt care- no results |
