Digestive System Lecture 7- Dental Disease In Dogs And Cats

Front 1

oral defence mechanism - chewing

Back 1

mechanical effect

Front 2

oral defence mechanism- salive

Back 2

protective lubricant

plays a role in the formation of plaque and calculus

influences the nature of oral flora and the chemical composition of teeth

Front 3

oral defence mechanism- cell mediated immunity

Back 3

lymphocytes produce antibodies and kill virus infected cells

may cause inflammation

Front 4

plaque

Back 4

mass of bacteria firmly adherent to a pellicle on the enamel

Front 5

pellicle

Back 5

derives from saliva and is a mucopolysaccharide

Front 6

calculus

Back 6

mineralised plaque which provides suitable surface for plaque to hide and survive

Front 7

plaque deposition

Back 7

first step: deposition of a pellicle
second step: pellicle actively being involved in bacterial colonization
third step: involves maturation of plaque bacteria and chage in the species predominating

Front 8

pellicle formation

Back 8

1. bathing of rooth surfaces in saliva
2. selective absorption of glycoproteins
3. loss of solubility of these proteins by surface denaturation and acid precipitation
4. enzymes from bacteria alter these glycoproteins

Front 9

central components of periodontal disease

Back 9

1. caused by bacteria
2. invading bacteria disrupt the junctional epithilium producing pockets and changes in connectives tissues, eventually causing loss of alveolar tooth bone

Front 10

gingivitis

Back 10

reversible inflammation of the gingiva associated with plaque build up

Front 11

peridontitis

Back 11

irreversable loss of connective tissue attachment with pocket formation.
preceded by gingivitis, it increases in severity and preveleance with age

Front 12

plaque control

Back 12

a state of equilibrium should exist between the host and the plque biofilm and this prevents the growth of pathogenic bacteria

once attached removal of plaque only possible by mechanical means or chemical means

Front 13

tooth resportion

Back 13

lesions are characterised by a cervical defect of enamel, dentine and cementum.

caused by resportion of enamel, dentine and cementum by odontoblast cells

Front 14

clinical appearance of tooth resoption

Back 14

most commonly seen in the mesial premolar teeth, secondly in other distal teeth

inflammation of the gingiva

cats- seen more on buccal surface than on the lingual/palatal surface

Front 15

clinical signs of tooth resportion

Back 15

painful
anorexia
ptyalism
lethargy
depression
halitosis
dysphagia
jaw spasm when probing with dental instruments

Front 16

scaling

Back 16

supra and subgingival removal of plaque and calculus from tooth surfaces

Front 17

root planing

Back 17

residual calculus and bacteria toxin affected cementum is removed from roots

Front 18

closed curettage

Back 18

process by which pocket epithilium is removed without a surgical flap

Front 19

open curettage

Back 19

process by which pockets epithilium is removed under direct vision by surgically raising a flap

reccomanded is pocket is >5mm deep

Front 20

periodontal probing

Back 20

with a blunt probe, measures attachment loss and pocket depth.

provides a practical way of assessing periodental health

Front 21

dental prophylaxis

Back 21

dental treatment limited to patients with no established periodentitis, therefore it is a treatment to prevent the establishment of periodontitis