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33 Cards in this Set

  • Front
  • Back

aggravating factors for acne

dirt/oil, weather extremes, endocrine disorders, drugs, sugar

cause of acne

plugged sebaceous glands within hair follicles


more secretions than can exit follicle

signs and symptoms of acne

white heads, black heads, pustules, cysts, redness/inflammation, crusty, scar

treatment for acne

topical

oral antibiotics


surgical (chemical peel, microdermabrasion)


lessen sun exposure


home care



complications of acne

cyst, abscess formation, scarring/keloids, side effects medications, psychological/social side effects

contact dermatitis causes and risk factors

history of allergies


varies form person


may involve repeated use

types of contact dermatitis

contact with irritants


hypersensitivity/allergic reactions


acid/alkali

signs and symptoms of contact dermatitis

pruritis, redness, inflammation, tenderness, localized swelling, rash, pustules, vesicles, crusting, scaly, raw

melanocytic nevus

congenitally pigmented lesions

melanocytic nevus types

junctional


intradermal


compound

junctional melanocytic nevus

melanocytes at dermis/epidermis junction, most likely to become malignant

intradermal melanocytic nevus

melanocyte in connective tissue

compound melanocytic nevus

combined junctional and intradermal

cold sores

recurrent herpes labialis

cause of cold sores

HSV

cod sores cure

none


antiviral meds can help


heal within 2 weeks

aphthous ulcer

nonviral origin


intraoral


heals within a week


painful

basal cell carcinoma

cancer

causes/risk factors of basal cell carcinoma

regular exposure to sunligh, UV, light skin/hair, 75% of skin cancers, remains local, invades surrounding tissue




"locally invasive" rarely metastasize

signs and symptoms of basal cell carcinoma

ulcerated, bleeds easily, does not heal well, pearly, waxy, flat, sl raised, visible blood vessels, scar lesion

prevention of basal cell carcinoma

minimize sun exposure

squamous cell carcinoma causes/risk factors

changes in existing lesion, ulceration, lack of healing, areas exposed to sunlight, UV, dray, chemical pollution, genetic disposition, age

signs and symptoms of squamous cell carcinoma

firm, reddened, scaly, crusty, nodular, flat, variable

skin changes of squamous cell carcinoma

color, size, texture, appearance, pain, inflammation, bleeding, itching, asymmetrical, irregular diffuse borders, multiple colors

prevention of squamous cell carcinoma

minimize skin exposure

causes and risk factors of malignant melanoma

acute, intense, intermittent, blistering, sunburn, may develop near existing lesions, healthy skin, unexposed skin, race, gender (male), age, UV (UVA/UVB together induce melanoma, suppress immune system of the skin)

examinations/tests for malignant melanoma

CVC, X-rays, CT, MRI, biopsy, total body exam

ABCD system for malignant melanoma

Asymmetry


Border irregularity


Color


Diameter

superficial spreading melanoma

SSM


70% of cutaneous melanomas/young


arises from long standing nevus


any body surface

lentigo maligna melanoma

LMM


10-15%/ elderly


most common melanoma in hawaii


invasion form of lentigo malign a


face, ears, arms, upper trunk

acral lentiginous melanoma

ALM


black, brown discoloration on hands/feet/palms


most common in african am. and asians

nodular melanoma

NM


10-15%/ elderly


trunk/legs/arm/scalp


symmetrical/uniform


dark, brown, black amelonotic


short radial growth period, high risk lesion, invasive by time of detection

treatment for malignant melanoma

surgy (w or w/o lymph node removal


chemotherapy


radiation


biological therapy




(precision medicine)