Cva/stroke Quiz By Jeff

Front 1

ischemic stroke:

Back 1

-decreased CBF (cerebral blood flow)

-can lead to clots

Front 2

causes:

-clots

Back 2

-thrombotic (clot in place)

-Embolus- clot which came from somewhere else inbody

-decreased elasticity of artery

-CHF>decreased CO>decreased CBF

-decreased blood volume- drowning

-hypotensive

Front 3

Hemorrhagic stroke

causes:

Back 3

HTN

aneurysm

AVN (arterio-venous malformation)

Front 4

at risk for stroke:

Back 4

-atherosclerosis- increase cholesterol

-^LDL or hypercholesterolemia (gretar than 240),borderline:130-159 to high:160-189 and very high: 190

-^triglycerides- fasting 150

-decreased HDL: below 40 for males and 50 for females

-increased age

-DM

-HTN

Front 5

R cva

Back 5

L hemiplegic, L homonymous hemianopsia, decreased spatial/perceptual tasks, quick and impulsive, overestimate abilities, unawareof deficits, slow down- steps “whole pic”, does not respond to visual-spatial cues especially in cluttered areas.

Hint 5

Spqousvs

Front 6

L cva:

Back 6

R hemiplegic, R homonymous hemianopsia, aphasias, aproxias/disorganized, cautious/anxious, hesitant, likes more feedback, realistic of existing problem

Hint 6

Aaadchfr

Front 7

both

Back 7

homonymous hemianopsias

emotional abnormalities

cognitive defects

Front 8

lethargic

Back 8

-arousal w/stimuli

-falls asleep when not stim

-decreased awarness

-loss of train of thought

Front 9

stupor (semicoma)

Back 9

-arousal only w/strong noxious stimulus

-returns to unconscious state when stim stop

-unable to interact w/ clinician

Front 10

Obtunded

Back 10

-difficult to arouse

-needs constant stim to maintain consciousness

-confused when awake

-may be unproductive w/ clinician

Front 11

comatose (coma)

Back 11

-unarousable

-deep unconsciousness

Front 12

indirect complications to CVA's

Back 12

-seizures, B+B dysfunction, pressure ulcer, cardiopul dysfunction, DVT/PE, osteoporosis/fx

Front 13

synergy patterns

UE: flexion

**strongest component

Back 13

-scap ret/elevation or hyperex

-shld abd, ER

-elbow flex**

-FA sup

-wrist/finger flexed

Front 14

UE: extension

Back 14

-scap prot

-shld add**, IR

-elbow ext

-FA pron**

-wrist/finger flex

Front 15

LE: flexion

Back 15

-hip flex**, abd, er

-knee flex

-DF, INV

-toe DF

Front 16

LE: extension

Back 16

-hip ext, add**, ir

-knee ext**

-PF**, inv

-toe PF

Front 17

Common postural deviations

Pelvis:

Back 17

-assymm WB (more wb on unaffected side)

-fear of wgt shifting to affected side

-PPT>sacral sitting

-in standing>unilateral retraction/elev of affected side

Front 18

trunk

Back 18

-flattened L-curve, Increase T-curve, fwd head

-lateral flexion

Front 19

shld

Back 19

-unequal height/affected shld depressed

-subluxation of HH, downward scap rot

-a cap instability may be present (winging)

Front 20

Head/neck

Back 20

prot

lat flex (rotate away from affected side)

Front 21

Affected UE

Back 21

-flex, add, IR, elbow flex, FA pron, wrist/finger flex, limb is non wb

Front 22

unaffected UE

Back 22

-stronger UE used for postural support

Front 23

LE in sitting

Back 23

affected LE-hip abd, er, w/hip and knee flex (flex synergy pattern)

Front 24

LE in standing

Back 24

affected LE- hip/knee ext, add, ir, PF, unequal wb

Front 25

synergies

Back 25

functionally linked mvmnts that are constrained by the CNS to act coop. to produce an intended motor action

Front 26

deteriorating stroke

Back 26

neurological status cont to decrease after admission to hospital

Front 27

agnosia

Back 27

the loss of the ability to recognize objects, faces, voices, or places.

Front 28

apraxia

Back 28

An impairment of voluntary learned movement that is characterized by an inability to perform purposeful movements not accounted for by inadequate strength loss of coordination, impaired sensation, attention deficits, or lack of comprehension

Front 29

associated reactions

Back 29

An involuntary movement of a body part associated with the resisted movement of another body part.

Front 30

ipsilateral pushing (pushers syndrome)

Back 30

active pushing w/stronger extremities twds hemiparetic side w/lat posture imbal

Front 31

TIA

Back 31

temp interruption of blood supply to the brain not more than 24 hrs> no damage to brain

Front 32

dysarthria

Back 32

often is characterized by slurred or slow speech that can be difficult to understand

Front 33

ideational apraxia

Back 33

Inability to perform a purposeful motor act either automatically or on command

Front 34

ideomotor apraxia

Back 34

Inability to perform a motor act on command althoughpatient understands the concept of the task and is ableto carry out habitual tasks automatically

Front 35

ataxia

Back 35

-the loss of full control of bodily movements

-lack of muscle control during voluntary movements

Front 36

hemiparesis

Back 36

weakness of one side

Front 37

hemiplegia

Back 37

paralysis of one side

Front 38

aphasia

Back 38

loss of ability to understand or express speech, caused by brain damage

Front 39

non-fluent aphasia

Back 39

flow of speech is slow and hesitant, vocab limited, speech production is lost but comprehension still intact.

Front 40

fluent aphasia

Back 40

the ability to grasp the meaning of spoken words and sentences is impaired, while the ease of producing connected speech is not very affected.

Front 41

hyperreflexia

Back 41

overactive or over-responsive reflexes.

Front 42

motor impersistence

Back 42

is the inability to maintain postures or positions (such as keeping eyes closed, protruding the tongue, maintaining conjugate gaze steadily in a fixed direction, or making a prolonged “ah” sound) without repeated prompts.

Front 43

homonymous hemianopsia

Back 43

is the loss of half of the field of view on the same side in both eyes